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Nucleus tractus solitarii is required for the development and maintenance of phrenic and sympathetic long-term facilitation after acute intermittent hypoxia

Frontiers in physiology, 2023-02, Vol.14, p.1120341-1120341

2023

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Gamma-Aminobutyric Acid Transporters in the Nucleus Tractus Solitarii Regulate Inhibitory and Excitatory Synaptic Currents That Influence Cardiorespiratory Function

Frontiers in physiology, 2022-01, Vol.12, p.821110-821110

2022

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Selective breeding for physical inactivity produces cognitive deficits via altered hippocampal mitochondrial and synaptic function

Frontiers in aging neuroscience, 2023-04, Vol.15, p.1147420-1147420

2023

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The role of astrocytes in the nucleus tractus solitarii in maintaining central control of autonomic function

American journal of physiology. Regulatory, integrative and comparative physiology, 2021-04, Vol.320 (4), p.R418-R424

2021

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Defective Carotid Body Function and Impaired Ventilatory Responses to Chronic Hypoxia in Mice Partially Deficient for Hypoxia-Inducible Factor 1α

Proceedings of the National Academy of Sciences - PNAS, 2002-01, Vol.99 (2), p.821-826

2002

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Tuning excitability of the hypothalamus via glutamate and potassium channel coupling

The Journal of physiology, 2017-07, Vol.595 (14), p.4583-4584

Kline, David D.

2017

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Cardiovascular deconditioning increases GABA signaling in the nucleus tractus solitarii

Journal of neurophysiology, 2022-07, Vol.128 (1), p.28-39

2022

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Exogenous brain-derived neurotrophic factor rescues synaptic dysfunction in Mecp2-null mice

The Journal of neuroscience, 2010-04, Vol.30 (15), p.5303-5310

2010

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Unilateral vagotomy alters astrocyte and microglial morphology in the nucleus tractus solitarii of the rat

American journal of physiology. Regulatory, integrative and comparative physiology, 2021-06, Vol.320 (6), p.R945-R959

2021

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Altered GABAergic neurotransmission at the nucleus tractus solitarii following Hindlimb Unloading

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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Hypoxia Augments TRPM3‐mediated Calcium Influx in Vagal Sensory Neurons

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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Unilateral Vagotomy Blunts Cardiorespiratory Responses to Vagal Afferent Stimulation and Nucleus Tractus Solitarii (NTS) Glutamate Microinjection

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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Oxytocin and corticotropin‐releasing hormone augment synaptic activity in the nTS of rats subjected to chronic intermittent hypoxia

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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TRPV1 channels contribute to spontaneous glutamate release in nucleus tractus solitarii following chronic intermittent hypoxia

Journal of neurophysiology, 2019-03, Vol.121 (3), p.881-892

2019

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Mechanisms Underlying Neuroplasticity in the Nucleus Tractus Solitarii Following Hindlimb Unloading in Rats

Neuroscience, 2020-11, Vol.449, p.214-227

2020

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Sustained Hypoxia Alters nTS Glutamatergic Signaling and Expression and Function of Excitatory Amino Acid Transporters

Neuroscience, 2020-03, Vol.430, p.131-140

2020

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Hypobaric intermittent hypoxia attenuates hypoxia-induced depressor response

PloS one, 2012-07, Vol.7 (7), p.e41656-e41656

2012

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Alpha adrenergic receptor signaling in the hypothalamic paraventricular nucleus is diminished by the chronic intermittent hypoxia model of sleep apnea

Experimental neurology, 2021-01, Vol.335, p.113517-113517, Article 113517

2021

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Hydrogen peroxide inhibits neurons in the paraventricular nucleus of the hypothalamus via potassium channel activation

American journal of physiology. Regulatory, integrative and comparative physiology, 2019-07, Vol.317 (1), p.R121-R133

2019

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Oxytocin and corticotropin‐releasing hormone exaggerate nucleus tractus solitarii neuronal and synaptic activity following chronic intermittent hypoxia

The Journal of physiology, 2024-07, Vol.602 (14), p.3375-3400

2024

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The PVN enhances cardiorespiratory responses to acute hypoxia via input to the nTS

American journal of physiology. Regulatory, integrative and comparative physiology, 2019-12, Vol.317 (6), p.R818-R833

2019

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Vagotomy blunts cardiorespiratory responses to vagal afferent stimulation via pre‐ and postsynaptic effects in the nucleus tractus solitarii

The Journal of physiology, 2024-03, Vol.602 (6), p.1147-1174

2024

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Exaggerated potassium current reduction by oxytocin in visceral sensory neurons following chronic intermittent hypoxia

Autonomic neuroscience, 2020-12, Vol.229, p.102735-102735, Article 102735

2020

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Paraventricular nucleus projections to the nucleus tractus solitarii are essential for full expression of hypoxia‐induced peripheral chemoreflex responses

The Journal of physiology, 2023-10, Vol.601 (19), p.4309-4336

2023

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Kv2 channels contribute to neuronal activity within the vagal afferent-nTS reflex arc

American Journal of Physiology: Cell Physiology, 2024-01, Vol.326 (1), p.C74

2024

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1 – 25 von 670

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Nucleus tractus solitarii is required for the development and maintenance of phrenic and sympathetic long-term facilitation after acute intermittent hypoxia

Gamma-Aminobutyric Acid Transporters in the Nucleus Tractus Solitarii Regulate Inhibitory and Excitatory Synaptic Currents That Influence Cardiorespiratory Function

Selective breeding for physical inactivity produces cognitive deficits via altered hippocampal mitochondrial and synaptic function

The role of astrocytes in the nucleus tractus solitarii in maintaining central control of autonomic function

Defective Carotid Body Function and Impaired Ventilatory Responses to Chronic Hypoxia in Mice Partially Deficient for Hypoxia-Inducible Factor 1α

Tuning excitability of the hypothalamus via glutamate and potassium channel coupling

Cardiovascular deconditioning increases GABA signaling in the nucleus tractus solitarii

Exogenous brain-derived neurotrophic factor rescues synaptic dysfunction in Mecp2-null mice

Unilateral vagotomy alters astrocyte and microglial morphology in the nucleus tractus solitarii of the rat

Altered GABAergic neurotransmission at the nucleus tractus solitarii following Hindlimb Unloading

Hypoxia Augments TRPM3‐mediated Calcium Influx in Vagal Sensory Neurons

Unilateral Vagotomy Blunts Cardiorespiratory Responses to Vagal Afferent Stimulation and Nucleus Tractus Solitarii (NTS) Glutamate Microinjection

Oxytocin and corticotropin‐releasing hormone augment synaptic activity in the nTS of rats subjected to chronic intermittent hypoxia

TRPV1 channels contribute to spontaneous glutamate release in nucleus tractus solitarii following chronic intermittent hypoxia

Mechanisms Underlying Neuroplasticity in the Nucleus Tractus Solitarii Following Hindlimb Unloading in Rats

Sustained Hypoxia Alters nTS Glutamatergic Signaling and Expression and Function of Excitatory Amino Acid Transporters

Hypobaric intermittent hypoxia attenuates hypoxia-induced depressor response

Alpha adrenergic receptor signaling in the hypothalamic paraventricular nucleus is diminished by the chronic intermittent hypoxia model of sleep apnea

Hydrogen peroxide inhibits neurons in the paraventricular nucleus of the hypothalamus via potassium channel activation

Oxytocin and corticotropin‐releasing hormone exaggerate nucleus tractus solitarii neuronal and synaptic activity following chronic intermittent hypoxia

The PVN enhances cardiorespiratory responses to acute hypoxia via input to the nTS

Vagotomy blunts cardiorespiratory responses to vagal afferent stimulation via pre‐ and postsynaptic effects in the nucleus tractus solitarii

Exaggerated potassium current reduction by oxytocin in visceral sensory neurons following chronic intermittent hypoxia

Paraventricular nucleus projections to the nucleus tractus solitarii are essential for full expression of hypoxia‐induced peripheral chemoreflex responses

Kv2 channels contribute to neuronal activity within the vagal afferent-nTS reflex arc

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