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Wilms' tumours: about tumour suppressor genes, an oncogene and a chameleon gene

Nature reviews. Cancer, 2011-02, Vol.11 (2), p.111

Huff, Vicki

2011

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A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28 (KAP1), a gene critical in early renal development: A Children's Oncology Group study

PloS one, 2018-12, Vol.13 (12), p.e0208936-e0208936

2018

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The Wilms tumor gene, Wt1, is critical for mouse spermatogenesis via regulation of sertoli cell polarity and is associated with non-obstructive azoospermia in humans

PLoS genetics, 2013-08, Vol.9 (8), p.e1003645-e1003645

2013

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Loss of Heterozygosity for Chromosomes 1p and 16q Is an Adverse Prognostic Factor in Favorable-Histology Wilms Tumor: A Report From the National Wilms Tumor Study Group

Journal of clinical oncology, 2005-10, Vol.23 (29), p.7312-7321

2005

Volltextzugriff (PDF)

WT1 Mutation and 11P15 Loss of Heterozygosity Predict Relapse in Very Low-Risk Wilms Tumors Treated With Surgery Alone: A Children's Oncology Group Study

Journal of clinical oncology, 2011-02, Vol.29 (6), p.698-703

2011

Volltextzugriff (PDF)

Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors

Translational oncology, 2014-08, Vol.7 (4), p.484-492

2014

Volltextzugriff (PDF)

Wilms’ Tumor Blastemal Stem Cells Dedifferentiate to Propagate the Tumor Bulk

Stem cell reports, 2014-07, Vol.3 (1), p.24-33

2014

Volltextzugriff (PDF)

Race Disparities in Peptide Profiles of North American and Kenyan Wilms Tumor Specimens

Journal of the American College of Surgeons, 2014-04, Vol.218 (4), p.707-720

2014

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Treatment With Nephrectomy Only for Small, Stage I/Favorable Histology Wilms’ Tumor: A Report From the National Wilms’ Tumor Study Group

Journal of clinical oncology, 2001-09, Vol.19 (17), p.3719-3724

2001

Volltextzugriff (PDF)

Denys-Drash syndrome: Relating a clinical disorder to genetic alterations in the tumor suppressor gene WT1

The Journal of pediatrics, 1993-11, Vol.123 (5), p.673-678

1993

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Long-term Outcomes for Infants With Very Low Risk Wilms Tumor Treated With Surgery Alone in National Wilms Tumor Study-5

Annals of surgery, 2010-03, Vol.251 (3), p.555-558

2010

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Reply to B. Royer-Pokora et al

Journal of clinical oncology, 2011-06, Vol.29 (16), p.e487-e488

2011

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Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors12

Translational oncology, 2014-06, Vol.7 (4), p.484-492

2014

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Parental origin of WT1 mutations and mental retardation in WAGR syndrome

Nature genetics, 1994-09, Vol.8 (1), p.13-13

Huff, Vicki

1994

Volltextzugriff (PDF)

Loss of heterozygosity for chromosomes 1p and 16q is an adverse prognostic factor in favorable-histology wilms tumor : A report from the national wilms tumor study group: Molecular oncology: genomics

Journal of clinical oncology, 2005, Vol.23 (29), p.7312-7321

2005

Volltextzugriff (PDF)

Genetic mosaicism in normal tissues of Wilms' tumour patients

Nature genetics, 1993-02, Vol.3 (2), p.127-131

1993

Volltextzugriff (PDF)

Familial Renal Cell Carcinoma: Hereditary or Coincidental?

The Journal of urology, 1990-10, Vol.144 (4), p.849-851

1990

Volltextzugriff (PDF)

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Wilms' tumours: about tumour suppressor genes, an oncogene and a chameleon gene

A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28 (KAP1), a gene critical in early renal development: A Children's Oncology Group study

The Wilms tumor gene, Wt1, is critical for mouse spermatogenesis via regulation of sertoli cell polarity and is associated with non-obstructive azoospermia in humans

Loss of Heterozygosity for Chromosomes 1p and 16q Is an Adverse Prognostic Factor in Favorable-Histology Wilms Tumor: A Report From the National Wilms Tumor Study Group

WT1 Mutation and 11P15 Loss of Heterozygosity Predict Relapse in Very Low-Risk Wilms Tumors Treated With Surgery Alone: A Children's Oncology Group Study

Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors

Wilms’ Tumor Blastemal Stem Cells Dedifferentiate to Propagate the Tumor Bulk

Race Disparities in Peptide Profiles of North American and Kenyan Wilms Tumor Specimens

Treatment With Nephrectomy Only for Small, Stage I/Favorable Histology Wilms’ Tumor: A Report From the National Wilms’ Tumor Study Group

Denys-Drash syndrome: Relating a clinical disorder to genetic alterations in the tumor suppressor gene WT1

Long-term Outcomes for Infants With Very Low Risk Wilms Tumor Treated With Surgery Alone in National Wilms Tumor Study-5

Reply to B. Royer-Pokora et al

Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors12

Parental origin of WT1 mutations and mental retardation in WAGR syndrome

Loss of heterozygosity for chromosomes 1p and 16q is an adverse prognostic factor in favorable-histology wilms tumor : A report from the national wilms tumor study group: Molecular oncology: genomics

Genetic mosaicism in normal tissues of Wilms' tumour patients

Familial Renal Cell Carcinoma: Hereditary or Coincidental?

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