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Wt1 functions in ovarian follicle development by regulating granulosa cell differentiation

Human molecular genetics, 2014-01, Vol.23 (2), p.333-341

2014

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Reprogramming of Sertoli cells to fetal-like Leydig cells by Wt1 ablation

Proceedings of the National Academy of Sciences - PNAS, 2015-03, Vol.112 (13), p.4003-4008

2015

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Children's Oncology Group's 2013 blueprint for research: Renal tumors

Pediatric blood & cancer, 2013-06, Vol.60 (6), p.994-1000

2013

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The Wilms tumor gene, Wt1, is critical for mouse spermatogenesis via regulation of sertoli cell polarity and is associated with non-obstructive azoospermia in humans

PLoS genetics, 2013-08, Vol.9 (8), p.e1003645-e1003645

2013

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Wilm's tumor 1 promotes memory flexibility

Nature communications, 2019-08, Vol.10 (1), p.3756-18, Article 3756

2019

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Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors

Translational oncology, 2014-08, Vol.7 (4), p.484-492

2014

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Wilms’ Tumor Blastemal Stem Cells Dedifferentiate to Propagate the Tumor Bulk

Stem cell reports, 2014-07, Vol.3 (1), p.24-33

2014

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Race Disparities in Peptide Profiles of North American and Kenyan Wilms Tumor Specimens

Journal of the American College of Surgeons, 2014-04, Vol.218 (4), p.707-720

2014

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Treatment With Nephrectomy Only for Small, Stage I/Favorable Histology Wilms’ Tumor: A Report From the National Wilms’ Tumor Study Group

Journal of clinical oncology, 2001-09, Vol.19 (17), p.3719-3724

2001

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Generation of a Wt1 conditional deletion, nuclear red fluorescent protein reporter allele in the mouse

Differentiation (London), 2024-07, Vol.138, p.100791, Article 100791

2024

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Wt1 directs the lineage specification of sertoli and granulosa cells by repressing Sf1 expression

Development (Cambridge), 2017-01, Vol.144 (1), p.44-53

2017

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β-Catenin Activation in a Novel Liver Progenitor Cell Type Is Sufficient to Cause Hepatocellular Carcinoma and Hepatoblastoma

Cancer research (Chicago, Ill.), 2014-08, Vol.74 (16), p.4515-4525

2014

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Wt1 negatively regulates beta-catenin signaling during testis development

Development (Cambridge), 2008-05, Vol.135 (10), p.1875-1885

2008

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Wilms tumor 1 (WT1) regulates KRAS-driven oncogenesis and senescence in mouse and human models

The Journal of clinical investigation, 2010-11, Vol.120 (11), p.3940-3952

2010

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Left-sided cryptorchidism in mice with Wilms' tumour 1 gene deletion in gubernaculum testis

The Journal of pathology, 2013-05, Vol.230 (1), p.39-47

2013

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Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumor using whole genome sequencing

Journal of medical genetics, 2015-11, Vol.53 (6), p.385-388

2015

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Abstract 5047: Impact of kidney progenitor cell differentiation status in Wilms tumorigenesis

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.5047-5047

2013

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Inheritance of Unequal Numbers of the Genes Encoding the Human Neutrophil Defensins HP-1 and HP-3 (∗)

The Journal of biological chemistry, 1995-12, Vol.270 (51), p.30371-30376

1995

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Knock-in of the Wt1 R394W mutation causes MDS and cooperates with Flt3/ITD to drive aggressive myeloid neoplasms in mice

Oncotarget, 2018-10, Vol.9 (82), p.35313-35326

2018

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Genetic changes associated with relapse in favorable histology Wilms tumor: A Children’s Oncology Group AREN03B2 study

Cell reports. Medicine, 2022-06, Vol.3 (6), p.100644, Article 100644

2022

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A Wilms Tumor 1 (WT1) Mutation Causes Myelodysplastic Syndrome in a Knock-in Mouse Model, and a Mixed Myelodysplastic/Myeloproliferative Neoplam in Double Knock-in Mice with WT1 and FLT3/ITD Mutations

Blood, 2015-12, Vol.126 (23), p.312-312

2015

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The Wt1+/R394W Mouse Displays Glomerulosclerosis and Early-Onset Renal Failure Characteristic of Human Denys-Drash Syndrome

Molecular and Cellular Biology, 2004-11, Vol.24 (22), p.9899-9910

2004

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Reprogramming of Sertoli cells to fetal-like Leydig cells byWt1ablation

Proceedings of the National Academy of Sciences - PNAS, 2015-03, Vol.112 (13), p.4003-4008

2015

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Wt1 functions in ovarian follicle development by regulating granulosa cell differentiation

Reprogramming of Sertoli cells to fetal-like Leydig cells by Wt1 ablation

Children's Oncology Group's 2013 blueprint for research: Renal tumors

The Wilms tumor gene, Wt1, is critical for mouse spermatogenesis via regulation of sertoli cell polarity and is associated with non-obstructive azoospermia in humans

Wilm's tumor 1 promotes memory flexibility

Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors

Wilms’ Tumor Blastemal Stem Cells Dedifferentiate to Propagate the Tumor Bulk

Race Disparities in Peptide Profiles of North American and Kenyan Wilms Tumor Specimens

Treatment With Nephrectomy Only for Small, Stage I/Favorable Histology Wilms’ Tumor: A Report From the National Wilms’ Tumor Study Group

Generation of a Wt1 conditional deletion, nuclear red fluorescent protein reporter allele in the mouse

Wt1 directs the lineage specification of sertoli and granulosa cells by repressing Sf1 expression

β-Catenin Activation in a Novel Liver Progenitor Cell Type Is Sufficient to Cause Hepatocellular Carcinoma and Hepatoblastoma

Wt1 negatively regulates beta-catenin signaling during testis development

Wilms tumor 1 (WT1) regulates KRAS-driven oncogenesis and senescence in mouse and human models

Left-sided cryptorchidism in mice with Wilms' tumour 1 gene deletion in gubernaculum testis

Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumor using whole genome sequencing

Abstract 5047: Impact of kidney progenitor cell differentiation status in Wilms tumorigenesis

Hallmark discoveries in the biology of Wilms tumour

Inheritance of Unequal Numbers of the Genes Encoding the Human Neutrophil Defensins HP-1 and HP-3 (∗)

Knock-in of the Wt1 R394W mutation causes MDS and cooperates with Flt3/ITD to drive aggressive myeloid neoplasms in mice

Genetic changes associated with relapse in favorable histology Wilms tumor: A Children’s Oncology Group AREN03B2 study

A Wilms Tumor 1 (WT1) Mutation Causes Myelodysplastic Syndrome in a Knock-in Mouse Model, and a Mixed Myelodysplastic/Myeloproliferative Neoplam in Double Knock-in Mice with WT1 and FLT3/ITD Mutations

The Wt1+/R394W Mouse Displays Glomerulosclerosis and Early-Onset Renal Failure Characteristic of Human Denys-Drash Syndrome

Reprogramming of Sertoli cells to fetal-like Leydig cells byWt1ablation

Treatment of Wilms tumor relapsing after initial treatment with vincristine, actinomycin D, and doxorubicin. A report from the National Wilms Tumor Study Group

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