UB Paderborn / Katalog / Suche / in CDI

1 – 25 von 40

Sortiert nach

Danger-Associated Molecular Patterns (DAMPs): Molecular Triggers for Sterile Inflammation in the Liver

International journal of molecular sciences, 2018-10, Vol.19 (10), p.3104

Mihm, Sabine

2018

Volltextzugriff (PDF)

COVID-19: Possible Impact of the Genetic Background in IFNL Genes on Disease Outcomes

Journal of innate immunity, 2020-05, Vol.12 (3), p.273-274

Mihm, Sabine

2020

Volltextzugriff (PDF)

Hepatic IFNL4 Gene Activation in Hepatocellular Carcinoma Patients with Regard to Etiology

International journal of molecular sciences, 2021-08, Vol.22 (15), p.7803

2021

Volltextzugriff (PDF)

Diet high in fructose leads to an overexpression of lipocalin-2 in rat fatty liver

World journal of gastroenterology : WJG, 2014-02, Vol.20 (7), p.1807-1821

2014

Volltextzugriff (PDF)

Combination of alcohol and fructose exacerbates metabolic imbalance in terms of hepatic damage, dyslipidemia, and insulin resistance in rats

PloS one, 2014-08, Vol.9 (8), p.e104220

2014

Volltextzugriff (PDF)

Interleukin-29 uses a type 1 interferon-like program to promote antiviral responses in human hepatocytes

Hepatology (Baltimore, Md.), 2006-10, Vol.44 (4), p.896-906

2006

Volltextzugriff (PDF)

Interferon-lambda (IFNL) germline variations and their significance for HCC and PDAC progression: an analysis of The Cancer Genome Atlas (TCGA) data

BMC cancer, 2020-11, Vol.20 (1), p.1131-1131, Article 1131

2020

Volltextzugriff (PDF)

Interferon-lambda data

BMC cancer, 2020-11, Vol.20 (1)

2020

Volltextzugriff (PDF)

Interferon-lambda

BMC cancer, 2020-11, Vol.20 (1)

2020

Volltextzugriff (PDF)

Activation of Type I and Type III Interferons in Chronic Hepatitis C

Journal of innate immunity, 2015-01, Vol.7 (3), p.251-259

Mihm, Sabine

2015

Volltextzugriff (PDF)

Enhanced Expression of Interferon-Regulated Genes in the Liver of Patients with Chronic Hepatitis C Virus Infection: Detection by Suppression-Subtractive Hybridization

Journal of Virology, 2001-02, Vol.75 (3), p.1332-1338

2001

Volltextzugriff (PDF)

Hepatitis C Virus, Diabetes and Steatosis: Clinical Evidence in Favor of a Linkage and Role of Genotypes

Digestive diseases (Basel), 2010-01, Vol.28 (1), p.280-284

Mihm, Sabine

2010

Volltextzugriff (PDF)

Relating serum GGT to ALT activity enhances its predictability on treatment outcome in chronic hepatitis C

Hepatology (Baltimore, Md.), 2014-03, Vol.59 (3), p.1213-1214

2014

Volltextzugriff (PDF)

Interleukin‐29 uses a type 1 interferon‐like program to promote antiviral responses in human hepatocytes

Hepatology (Baltimore, Md.), 2006-10, Vol.44 (4), p.896-906

2006

Volltextzugriff (PDF)

Interferon-[lambda].sub.4 (IFNL4) Transcript Expression in Human Liver Tissue Samples

PloS one, 2013-12, Vol.8 (12), p.e84026

2013

Volltextzugriff (PDF)

Interferon-[lambda].sub.4

PloS one, 2013-12, Vol.8 (12), p.e84026

2013

Volltextzugriff (PDF)

Hepatic interferon-[lambda]3 (IFNL3) gene expression reveals not to be attenuated in non-Favorable IFNL3 rs4803217 or IFNL4 rs368234815 minor allele carriers in chronic hepatitis C

PloS one, 2015-11, Vol.10 (11)

2015

Volltextzugriff (PDF)

Does the interferon‐lambda rather than the interferon‐alpha pathway determine the outcome of hepatitis C virus infection?

Hepatology (Baltimore, Md.), 2014-10, Vol.60 (4), p.1437-1439

2014

Volltextzugriff (PDF)

Ferritin L is the Sole Serum Ferritin Constituent and a Positive Hepatic Acute-Phase Protein

Shock (Augusta, Ga.), 2013-06, Vol.39 (6), p.520-526

2013

Volltextzugriff (PDF)

Host response to hepatitis C virus

Hepatology (Baltimore, Md.), 2005-03, Vol.41 (3), p.679-679

Mihm, Sabine

2005

Volltextzugriff (PDF)

Interferon-α therapy does not modulate hepatic expression of classical type I interferon inducible genes

Journal of medical virology, 2008-11, Vol.80 (11), p.1912-1918

2008

Volltextzugriff (PDF)

Modulation of transcription factor NFχB activity by intracellular glutathione levels and by variations of the extracellular cysteine supply

The FASEB journal, 1995-02, Vol.9 (2), p.246-252

1995

Volltextzugriff (PDF)

Ultra-Rapid Virological Response, Young Age, Low gamma -GT/ALT-Ratio, and Absence of Steatosis Identify a Subgroup of HCV Genotype 3 Patients Who Achieve SVR with IFN- alpha sub(2a) Monotherapy

Digestive diseases and sciences, 2011-11, Vol.56 (11), p.3296-3304

2011

Volltextzugriff (PDF)

Ultra-Rapid Virological Response, Young Age, Low [gamma]-GT/ALT-Ratio, and Absence of Steatosis Identify a Subgroup of HCV Genotype 3 Patients Who Achieve SVR with IFN-[alpha].sub.2a Monotherapy

Digestive diseases and sciences, 2011-11, Vol.56 (11)

2011

Volltextzugriff (PDF)

Ultra-Rapid Virological Response, Young Age, Low [gamma]-GT/ALT-Ratio, and Absence of Steatosis Identify a Subgroup of HCV Genotype 3 Patients Who Achieve SVR with IFN-[alpha]^sub 2a^ Monotherapy

Digestive diseases and sciences, 2011-11, Vol.56 (11), p.3296

2011

Volltextzugriff (PDF)

1 – 25 von 40

Suche als RSS-Feed

Aktive Filter

Zeitschrift / SerieMethods In Enzymology

ThemaAdult

ThemaInfectious Diseases

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(38)
Mit Volltextzugriff
(38)

Publikationsform

Artikel
(39)
Buchkapitel
(1)

Sprache

Englisch
(40)
Japanisch
(4)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Zeitschrift / Serie

Hepatology
(5)
Plos One
(5)
Digestive Diseases And Sciences
(4)
Bmc Cancer
(3)
Cellular Immunology
(3)
Journal Of Medical Virology
(3)
International Journal Of Molecular Sciences
(2)
Journal Of Innate Immunity
(2)
The Faseb Journal
(2)
Aids
(1)

5 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Danger-Associated Molecular Patterns (DAMPs): Molecular Triggers for Sterile Inflammation in the Liver

COVID-19: Possible Impact of the Genetic Background in IFNL Genes on Disease Outcomes

Hepatic IFNL4 Gene Activation in Hepatocellular Carcinoma Patients with Regard to Etiology

Diet high in fructose leads to an overexpression of lipocalin-2 in rat fatty liver

Combination of alcohol and fructose exacerbates metabolic imbalance in terms of hepatic damage, dyslipidemia, and insulin resistance in rats

Interleukin-29 uses a type 1 interferon-like program to promote antiviral responses in human hepatocytes

Interferon-lambda (IFNL) germline variations and their significance for HCC and PDAC progression: an analysis of The Cancer Genome Atlas (TCGA) data

Interferon-lambda data

Interferon-lambda

Activation of Type I and Type III Interferons in Chronic Hepatitis C

Enhanced Expression of Interferon-Regulated Genes in the Liver of Patients with Chronic Hepatitis C Virus Infection: Detection by Suppression-Subtractive Hybridization

Hepatitis C Virus, Diabetes and Steatosis: Clinical Evidence in Favor of a Linkage and Role of Genotypes

Relating serum GGT to ALT activity enhances its predictability on treatment outcome in chronic hepatitis C

Interleukin‐29 uses a type 1 interferon‐like program to promote antiviral responses in human hepatocytes

Interferon-[lambda].sub.4 (IFNL4) Transcript Expression in Human Liver Tissue Samples

Interferon-[lambda].sub.4

Hepatic interferon-[lambda]3 (IFNL3) gene expression reveals not to be attenuated in non-Favorable IFNL3 rs4803217 or IFNL4 rs368234815 minor allele carriers in chronic hepatitis C

Does the interferon‐lambda rather than the interferon‐alpha pathway determine the outcome of hepatitis C virus infection?

Ferritin L is the Sole Serum Ferritin Constituent and a Positive Hepatic Acute-Phase Protein

Host response to hepatitis C virus

Interferon-α therapy does not modulate hepatic expression of classical type I interferon inducible genes

Modulation of transcription factor NFχB activity by intracellular glutathione levels and by variations of the extracellular cysteine supply

Ultra-Rapid Virological Response, Young Age, Low gamma -GT/ALT-Ratio, and Absence of Steatosis Identify a Subgroup of HCV Genotype 3 Patients Who Achieve SVR with IFN- alpha sub(2a) Monotherapy

Ultra-Rapid Virological Response, Young Age, Low [gamma]-GT/ALT-Ratio, and Absence of Steatosis Identify a Subgroup of HCV Genotype 3 Patients Who Achieve SVR with IFN-[alpha].sub.2a Monotherapy

Ultra-Rapid Virological Response, Young Age, Low [gamma]-GT/ALT-Ratio, and Absence of Steatosis Identify a Subgroup of HCV Genotype 3 Patients Who Achieve SVR with IFN-[alpha]^sub 2a^ Monotherapy

Aktive Filter

Suchergebnisse filtern