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High glucose repatterns human podocyte energy metabolism during differentiation and diabetic nephropathy

The FASEB journal, 2017-01, Vol.31 (1), p.294-307

2017

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Mitochondrial bioenergetics and structural network organization

Journal of cell science, 2007-03, Vol.120 (Pt 5), p.838-848

2007

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Rheb Regulates Mitophagy Induced by Mitochondrial Energetic Status

Cell metabolism, 2013-05, Vol.17 (5), p.719-730

2013

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Mitochondrial CB₁ receptors regulate neuronal energy metabolism

Nature neuroscience, 2012-03, Vol.15 (4), p.558-564

2012

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Mitochondrial morphology and cellular distribution are altered in SPG31 patients and are linked to DRP1 hyperphosphorylation

Human molecular genetics, 2017-02, Vol.26 (4), p.674-685

2017

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TRIM4; a novel mitochondrial interacting RING E3 ligase, sensitizes the cells to hydrogen peroxide (H2O2) induced cell death

Free radical biology & medicine, 2015-12, Vol.89, p.1036-1048

2015

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Ventral Tegmental Area Cannabinoid Type-1 Receptors Control Voluntary Exercise Performance

Biological psychiatry (1969), 2013-05, Vol.73 (9), p.895-903

2013

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Mitochondrial degradation and energy metabolism

Biochimica et biophysica acta, 2015-10, Vol.1853 (10), p.2812-2821

2015

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The E3 ubiquitin ligase FBXL6 controls the quality of newly synthesized mitochondrial ribosomal proteins

Cell reports (Cambridge), 2023-06, Vol.42 (6), p.112579-112579, Article 112579

2023

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Complete loss of function of the ubiquitin ligase HERC2 causes a severe neurodevelopmental phenotype

European journal of human genetics : EJHG, 2016-01, Vol.25 (1), p.52-58

2016

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Ubiquitin-Dependent Degradation of Mitochondrial Proteins Regulates Energy Metabolism

Cell reports (Cambridge), 2018-06, Vol.23 (10), p.2852-2863

2018

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Ultrastructure of the mitochondrion and its bearing on function and bioenergetics

Antioxidants & redox signaling, 2008-08, Vol.10 (8), p.1313

2008

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Mitoplasticity: adaptation biology of the mitochondrion to the cellular redox state in physiology and carcinogenesis

Antioxidants & redox signaling, 2013-03, Vol.18 (7), p.808

2013

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Cannabinoid control of brain bioenergetics: Exploring the subcellular localization of the CB1 receptor

Molecular metabolism (Germany), 2014-07, Vol.3 (4), p.495-504

2014

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MitoBrain, Putting energy into the brain

Neurobiology of disease, 2016-06, Vol.90, p.1-2

2016

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Regulation of the mitochondrial functions by the Ubiquitin protéasome system: molecular bases and physiopathological relevance

Biochimica et biophysica acta. Bioenergetics, 2022-09, Vol.1863, p.148848, Article 148848

2022

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Bronchial smooth muscle remodeling involves calcium-dependent enhanced mitochondrial biogenesis in asthma

The Journal of experimental medicine, 2007-12, Vol.204 (13), p.3173-3181

2007

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IBRDC2, an IBR-type E3 ubiquitin ligase, is a regulatory factor for Bax and apoptosis activation

The EMBO journal, 2010-04, Vol.29 (8), p.1458-1471

2010

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The E3 ubiquitin ligase FBXL6 controls the quality of newly synthesized mitochondrial ribosomal proteins

Cell reports (Cambridge), 2023-06, Vol.42

2023

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Mitochondrial fusion and division: Regulation and role in cell viability

Seminars in cell & developmental biology, 2009-05, Vol.20 (3), p.365-374

2009

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Ubiquitin-dependent degradation of mitochondrial proteins regulates energy metabolism

Biochimica et biophysica acta. Bioenergetics, 2018-09, Vol.1859, p.e56-e57

2018

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Adaptative capacity of mitochondrial biogenesis and of mitochondrial dynamics in response to pathogenic respiratory chain dysfunction

Antioxidants & redox signaling, 2013-08, Vol.19 (4), p.350

2013

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Waves of gene regulation suppress and then restore oxidative phosphorylation in cancer cells

The international journal of biochemistry & cell biology, 2011-07, Vol.43 (7), p.950-968

2011

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Mobilization of Adenine Nucleotide Translocators as Molecular Bases of the Biochemical Threshold Effect Observed in Mitochondrial Diseases

The Journal of biological chemistry, 2004-05, Vol.279 (19), p.20411-20421

2004

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High glucose repatterns human podocyte energy metabolism during differentiation and diabetic nephropathy

Mitochondrial bioenergetics and structural network organization

A cannabinoid link between mitochondria and memory

Rheb Regulates Mitophagy Induced by Mitochondrial Energetic Status

Mitochondrial CB₁ receptors regulate neuronal energy metabolism

Mitochondrial morphology and cellular distribution are altered in SPG31 patients and are linked to DRP1 hyperphosphorylation

TRIM4; a novel mitochondrial interacting RING E3 ligase, sensitizes the cells to hydrogen peroxide (H2O2) induced cell death

Ventral Tegmental Area Cannabinoid Type-1 Receptors Control Voluntary Exercise Performance

Mitochondrial degradation and energy metabolism

The E3 ubiquitin ligase FBXL6 controls the quality of newly synthesized mitochondrial ribosomal proteins

Complete loss of function of the ubiquitin ligase HERC2 causes a severe neurodevelopmental phenotype

Ubiquitin-Dependent Degradation of Mitochondrial Proteins Regulates Energy Metabolism

Ultrastructure of the mitochondrion and its bearing on function and bioenergetics

Mitoplasticity: adaptation biology of the mitochondrion to the cellular redox state in physiology and carcinogenesis

Cannabinoid control of brain bioenergetics: Exploring the subcellular localization of the CB1 receptor

MitoBrain, Putting energy into the brain

Regulation of the mitochondrial functions by the Ubiquitin protéasome system: molecular bases and physiopathological relevance

Bronchial smooth muscle remodeling involves calcium-dependent enhanced mitochondrial biogenesis in asthma

IBRDC2, an IBR-type E3 ubiquitin ligase, is a regulatory factor for Bax and apoptosis activation

The E3 ubiquitin ligase FBXL6 controls the quality of newly synthesized mitochondrial ribosomal proteins

Mitochondrial fusion and division: Regulation and role in cell viability

Ubiquitin-dependent degradation of mitochondrial proteins regulates energy metabolism

Adaptative capacity of mitochondrial biogenesis and of mitochondrial dynamics in response to pathogenic respiratory chain dysfunction

Waves of gene regulation suppress and then restore oxidative phosphorylation in cancer cells

Mobilization of Adenine Nucleotide Translocators as Molecular Bases of the Biochemical Threshold Effect Observed in Mitochondrial Diseases

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