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Calcineurin Broadly Regulates the Initiation of Skeletal Muscle-Specific Gene Expression by Binding Target Promoters and Facilitating the Interaction of the SWI/SNF Chromatin Remodeling Enzyme

Molecular and cellular biology, 2019-10, Vol.39 (19)

2019

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Differential Contributions of mSWI/SNF Chromatin Remodeler Sub-Families to Myoblast Differentiation

International journal of molecular sciences, 2023-07, Vol.24 (14), p.11256

2023

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Hyperglycemia Increases Interstitial Cells of Cajal via MAPK1 and MAPK3 Signaling to ETV1 and KIT, Leading to Rapid Gastric Emptying

Gastroenterology (New York, N.Y. 1943), 2017-08, Vol.153 (2), p.521-535.e20

2017

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Differential requirements for different subfamilies of the mammalian SWI/SNF chromatin remodeling enzymes in myoblast cell cycle progression and expression of the Pax7 regulator

Biochimica et biophysica acta. Gene regulatory mechanisms, 2022-02, Vol.1865 (2), p.194801-194801, Article 194801

2022

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PRMT5 links lipid metabolism to contractile function of skeletal muscles

EMBO reports, 2023-08, Vol.24 (8), p.e57306-n/a

2023

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Hypoxia-Inducible Factor 1α Stabilization Restores Epigenetic Control of Nitric Oxide Synthase 1 Expression and Reverses Gastroparesis in Female Diabetic Mice

Gastroenterology (New York, N.Y. 1943), 2023-12, Vol.165 (6), p.1458-1474

2023

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Protein Arginine Methyltransferase PRMT5 Regulates Fatty Acid Metabolism and Lipid Droplet Biogenesis in White Adipose Tissues

Advanced science, 2020-12, Vol.7 (23), p.2002602-n/a

2020

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Hedgehog pathway dysregulation contributes to the pathogenesis of human gastrointestinal stromal tumors via GLI-mediated activation of KIT expression

Oncotarget, 2016-11, Vol.7 (48), p.78226-78241

2016

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FAM96A is a novel pro‐apoptotic tumor suppressor in gastrointestinal stromal tumors

International journal of cancer, 2015-09, Vol.137 (6), p.1318-1329

2015

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423 Genome-Wide Epigenetic Determinants of Interstitial Cell of Cajal (ICC) Dysfunction

Gastroenterology (New York, N.Y. 1943), 2014-05, Vol.146 (5), p.S-91-S-91

2014

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382 The Polycomb Histone Lysine Methyltransferase Ezh2 Reversibly Regulates the Fate of Interstitial Cells of Cajal (ICC) In Vivo

Gastroenterology (New York, N.Y. 1943), 2015-04, Vol.148 (4), p.S-79-S-79

2015

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339 Hypoxia-Inducible Factor 1 Transactivates Neuronal Nitric Oxide Synthase and Is a Potential Therapeutic Target in Gastroparesis

Gastroenterology (New York, N.Y. 1943), 2016-04, Vol.150 (4), p.S80-S80

2016

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131 Glucose Oxidative Metabolism Regulates Interstitial Cell of Cajal Populations by Stimulating KIT Expression via ERK MAPK-Dependent ETV1 Stabilization

Gastroenterology (New York, N.Y. 1943), 2016-04, Vol.150 (4), p.S31-S31

2016

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Hypoxia-Inducible Factor 1 Alpha (HIF1A) Stimulates Neuronal Nitric Oxide Synthase (NOS1) Transcription by Binding to Multiple Enhancers

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S565-S566

2017

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Wnt-induced, TRP53-mediated Cell Cycle Arrest of Precursors Underlies Interstitial Cell of Cajal Depletion During Aging

Cellular and molecular gastroenterology and hepatology, 2021-01, Vol.11 (1), p.117-145

2021

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Sa1677 The Polycomb Histone Lysine Methyltransferase EZH2 Controls Interstitial Cell of Cajal Populations by Regulating Stem Cell Differentiation and Self-Renewal

Gastroenterology (New York, N.Y. 1943), 2016-04, Vol.150 (4), p.S343-S344

2016

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Succinate Accumulation Epigenetically Represses Kit Expression, Reduces Interstitial Cells of Cajal (ICC) and Delays Gastric Emptying of Solids

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S129-S130

2017

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113 Restoration of Kit Expression by Pharmacological Inhibition of Polycomb Group (PcG)-Mediated Epigenetic Repression in Interstitial Cells of Cajal (ICC) Subjected to Chronic Kit Ligand (Kitl) Withdrawal

Gastroenterology (New York, N.Y. 1943), 2013-05, Vol.144 (5), p.S-27-S-27

2013

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240 Epigenetic Regulation of Kit Expression in Murine KitLow Interstitial Cell of Cajal Stem Cells (ICC-SC) and KitLow Gastrointestinal Stromal Tumor (GIST) Cells by Polycomb Group (PcG) Repressive Complexes (PRC)

Gastroenterology (New York, N.Y. 1943), 2013-05, Vol.144 (5), p.S-52-S-52

2013

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743 - Physiological Effects of Regulation of ANO1 Transcription by Gli in Mouse Interstitial Cells of Cajal

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-155-S-155

2018

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Su1829 - Hypoxia-Inducible Factor 1 Alpha (HIF1A) Stimulates Neuronal Nitric Oxide Synthase (NOS1) Transcription by Binding to Multiple Enhancers

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S565-S566

2017

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724 - Epigenomic Landscape, Spatial Conformation and Function of Regulatory Elements of the PDGFRA-Kit Locus in Interstitial Cells of Cajal (ICC) and Gastrointestinal Stromal Tumor (GIST) Cells

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-149-S-149

2018

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593 - Succinate Accumulation Epigenetically Represses Kit Expression, Reduces Interstitial Cells of Cajal (ICC) and Delays Gastric Emptying of Solids

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S129-S130

2017

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Wnt-induced, TRP53-mediated Cell Cycle Arrest of Precursors Underlies Interstitial Cell of Cajal Depletion During AgingSummary

Cellular and molecular gastroenterology and hepatology, 2021-01, Vol.11 (1), p.117-145

2021

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FAM96A is a novel pro-apoptotic tumor suppressor in gastrointestinal stromal tumors: FAM96A is a novel proapoptotic tumor suppressor in GIST

International journal of cancer, 2015-09, Vol.137 (6), p.1318-1329

2015

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Calcineurin Broadly Regulates the Initiation of Skeletal Muscle-Specific Gene Expression by Binding Target Promoters and Facilitating the Interaction of the SWI/SNF Chromatin Remodeling Enzyme

Differential Contributions of mSWI/SNF Chromatin Remodeler Sub-Families to Myoblast Differentiation

Hyperglycemia Increases Interstitial Cells of Cajal via MAPK1 and MAPK3 Signaling to ETV1 and KIT, Leading to Rapid Gastric Emptying

Differential requirements for different subfamilies of the mammalian SWI/SNF chromatin remodeling enzymes in myoblast cell cycle progression and expression of the Pax7 regulator

PRMT5 links lipid metabolism to contractile function of skeletal muscles

Hypoxia-Inducible Factor 1α Stabilization Restores Epigenetic Control of Nitric Oxide Synthase 1 Expression and Reverses Gastroparesis in Female Diabetic Mice

Protein Arginine Methyltransferase PRMT5 Regulates Fatty Acid Metabolism and Lipid Droplet Biogenesis in White Adipose Tissues

Hedgehog pathway dysregulation contributes to the pathogenesis of human gastrointestinal stromal tumors via GLI-mediated activation of KIT expression

FAM96A is a novel pro‐apoptotic tumor suppressor in gastrointestinal stromal tumors

423 Genome-Wide Epigenetic Determinants of Interstitial Cell of Cajal (ICC) Dysfunction

382 The Polycomb Histone Lysine Methyltransferase Ezh2 Reversibly Regulates the Fate of Interstitial Cells of Cajal (ICC) In Vivo

339 Hypoxia-Inducible Factor 1 Transactivates Neuronal Nitric Oxide Synthase and Is a Potential Therapeutic Target in Gastroparesis

131 Glucose Oxidative Metabolism Regulates Interstitial Cell of Cajal Populations by Stimulating KIT Expression via ERK MAPK-Dependent ETV1 Stabilization

Hypoxia-Inducible Factor 1 Alpha (HIF1A) Stimulates Neuronal Nitric Oxide Synthase (NOS1) Transcription by Binding to Multiple Enhancers

Wnt-induced, TRP53-mediated Cell Cycle Arrest of Precursors Underlies Interstitial Cell of Cajal Depletion During Aging

Sa1677 The Polycomb Histone Lysine Methyltransferase EZH2 Controls Interstitial Cell of Cajal Populations by Regulating Stem Cell Differentiation and Self-Renewal

Succinate Accumulation Epigenetically Represses Kit Expression, Reduces Interstitial Cells of Cajal (ICC) and Delays Gastric Emptying of Solids

113 Restoration of Kit Expression by Pharmacological Inhibition of Polycomb Group (PcG)-Mediated Epigenetic Repression in Interstitial Cells of Cajal (ICC) Subjected to Chronic Kit Ligand (Kitl) Withdrawal

240 Epigenetic Regulation of Kit Expression in Murine KitLow Interstitial Cell of Cajal Stem Cells (ICC-SC) and KitLow Gastrointestinal Stromal Tumor (GIST) Cells by Polycomb Group (PcG) Repressive Complexes (PRC)

743 - Physiological Effects of Regulation of ANO1 Transcription by Gli in Mouse Interstitial Cells of Cajal

Su1829 - Hypoxia-Inducible Factor 1 Alpha (HIF1A) Stimulates Neuronal Nitric Oxide Synthase (NOS1) Transcription by Binding to Multiple Enhancers

724 - Epigenomic Landscape, Spatial Conformation and Function of Regulatory Elements of the PDGFRA-Kit Locus in Interstitial Cells of Cajal (ICC) and Gastrointestinal Stromal Tumor (GIST) Cells

593 - Succinate Accumulation Epigenetically Represses Kit Expression, Reduces Interstitial Cells of Cajal (ICC) and Delays Gastric Emptying of Solids

Wnt-induced, TRP53-mediated Cell Cycle Arrest of Precursors Underlies Interstitial Cell of Cajal Depletion During AgingSummary

FAM96A is a novel pro-apoptotic tumor suppressor in gastrointestinal stromal tumors: FAM96A is a novel proapoptotic tumor suppressor in GIST

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