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Update on Lynch syndrome genomics

Familial cancer, 2016-07, Vol.15 (3), p.385-393

Peltomaeki, Paeivi

2016

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Role of DNA Mismatch Repair Defects in the Pathogenesis of Human Cancer

Journal of clinical oncology, 2003-03, Vol.21 (6), p.1174-1179

Päivi Peltomäki

2003

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Mutations and epimutations in the origin of cancer

Experimental cell research, 2012-02, Vol.318 (4), p.299-310

Peltomaeki, Paeivi

2012

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Germline Mutation of RPS20 , Encoding a Ribosomal Protein, Causes Predisposition to Hereditary Nonpolyposis Colorectal Carcinoma Without DNA Mismatch Repair Deficiency

Gastroenterology (New York, N.Y. 1943), 2014-09, Vol.147 (3), p.595-598.e5

2014

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Ten Years After Mutation Testing for Lynch Syndrome: Cancer Incidence and Outcome in Mutation-Positive and Mutation-Negative Family Members

Journal of clinical oncology, 2009-10, Vol.27 (28), p.4793-4797

2009

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Oncogenic Potential of Bisphenol A and Common Environmental Contaminants in Human Mammary Epithelial Cells

International journal of molecular sciences, 2020-05, Vol.21 (10), p.3735

2020

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Desmoid tumor patients carry an elevated risk of familial adenomatous polyposis

Journal of surgical oncology, 2016-02, Vol.113 (2), p.209-212

2016

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Novel insights into tumorigenesis revealed by molecular analysis of Lynch syndrome cases with multiple colorectal tumors

Frontiers in oncology, 2024-04, Vol.14, p.1378392-1378392

2024

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Epigenetic signatures of familial cancer are characteristic of tumor type and family category

Cancer research (Chicago, Ill.), 2008-06, Vol.68 (12), p.4597-4605

2008

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Promoter-specific alterations of APC are a rare cause for mutation-negative familial adenomatous polyposis

Genes chromosomes & cancer, 2014-10, Vol.53 (10), p.857-864

2014

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Biallelic germline nonsense variant of MLH3 underlies polyposis predisposition

Genetics in medicine, 2019-08, Vol.21 (8), p.1868-1873

2019

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Methyltransferase expression and tumor suppressor gene methylation in sporadic and familial colorectal cancer

Genes chromosomes & cancer, 2015-12, Vol.54 (12), p.776-787

2015

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Converging endometrial and ovarian tumorigenesis in Lynch syndrome: Shared origin of synchronous carcinomas

Gynecologic oncology, 2018-07, Vol.150 (1), p.92-98

2018

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Altered methylation at microRNA-associated CpG islands in hereditary and sporadic carcinomas: a methylation-specific multiplex ligation-dependent probe amplification (MS-MLPA)-based approach

Molecular medicine (Cambridge, Mass.), 2011-07, Vol.17 (7-8), p.726-735

2011

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Detection of a major Lynch Syndrome-causing MLH1 founder variant in a large-scale genotyped cohort

Familial cancer, 2024-11, Vol.23 (4), p.647-652

2024

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Thyroid Carcinomas That Occur in Familial Adenomatous Polyposis Patients Recurrently Harbor Somatic Variants in APC , BRAF , and KTM2D

Thyroid (New York, N.Y.), 2020-03, Vol.30 (3), p.380-388

2020

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Mlh1 deficiency in normal mouse colon mucosa associates with chromosomally unstable colon cancer

Carcinogenesis (New York), 2018-05, Vol.39 (6), p.788-797

2018

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Controlled 15-year trial on screening for colorectal cancer in families with hereditary nonpolyposis colorectal cancer

Gastroenterology (New York, N.Y. 1943), 2000-05, Vol.118 (5), p.829-834

2000

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Mono- and biallelic germline variants of DNA glycosylase genes in colon adenomatous polyposis families from two continents

Frontiers in oncology, 2022-10, Vol.12, p.870863-870863

2022

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Sequencing of Lynch syndrome tumors reveals the importance of epigenetic alterations

Oncotarget, 2017-12, Vol.8 (64), p.108020-108030

2017

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Molecular Analysis of Endometrial Tumorigenesis: Importance of Complex Hyperplasia Regardless of Atypia

Clinical cancer research, 2009-09, Vol.15 (18), p.5772-5783

2009

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Distinct molecular profiles in Lynch syndrome‐associated and sporadic ovarian carcinomas

International journal of cancer, 2013-12, Vol.133 (11), p.2596-2608

2013

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DNA methylation changes and somatic mutations as tumorigenic events in Lynch syndrome-associated adenomas retaining mismatch repair protein expression

EBioMedicine, 2019-01, Vol.39, p.280-291

2019

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Mitotic abnormalities precede microsatellite instability in lynch syndrome-associated colorectal tumourigenesis

EBioMedicine, 2024-05, Vol.103, p.105111-105111, Article 105111

2024

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Body Weight, Physical Activity, and Risk of Cancer in Lynch Syndrome

Cancers, 2021-04, Vol.13 (8), p.1849

2021

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Update on Lynch syndrome genomics

Role of DNA Mismatch Repair Defects in the Pathogenesis of Human Cancer

Mutations and epimutations in the origin of cancer

Germline Mutation of RPS20 , Encoding a Ribosomal Protein, Causes Predisposition to Hereditary Nonpolyposis Colorectal Carcinoma Without DNA Mismatch Repair Deficiency

Ten Years After Mutation Testing for Lynch Syndrome: Cancer Incidence and Outcome in Mutation-Positive and Mutation-Negative Family Members

Oncogenic Potential of Bisphenol A and Common Environmental Contaminants in Human Mammary Epithelial Cells

Desmoid tumor patients carry an elevated risk of familial adenomatous polyposis

Novel insights into tumorigenesis revealed by molecular analysis of Lynch syndrome cases with multiple colorectal tumors

Epigenetic signatures of familial cancer are characteristic of tumor type and family category

Promoter-specific alterations of APC are a rare cause for mutation-negative familial adenomatous polyposis

Biallelic germline nonsense variant of MLH3 underlies polyposis predisposition

Methyltransferase expression and tumor suppressor gene methylation in sporadic and familial colorectal cancer

Converging endometrial and ovarian tumorigenesis in Lynch syndrome: Shared origin of synchronous carcinomas

Altered methylation at microRNA-associated CpG islands in hereditary and sporadic carcinomas: a methylation-specific multiplex ligation-dependent probe amplification (MS-MLPA)-based approach

Detection of a major Lynch Syndrome-causing MLH1 founder variant in a large-scale genotyped cohort

Thyroid Carcinomas That Occur in Familial Adenomatous Polyposis Patients Recurrently Harbor Somatic Variants in APC , BRAF , and KTM2D

Mlh1 deficiency in normal mouse colon mucosa associates with chromosomally unstable colon cancer

Controlled 15-year trial on screening for colorectal cancer in families with hereditary nonpolyposis colorectal cancer

Mono- and biallelic germline variants of DNA glycosylase genes in colon adenomatous polyposis families from two continents

Sequencing of Lynch syndrome tumors reveals the importance of epigenetic alterations

Molecular Analysis of Endometrial Tumorigenesis: Importance of Complex Hyperplasia Regardless of Atypia

Distinct molecular profiles in Lynch syndrome‐associated and sporadic ovarian carcinomas

DNA methylation changes and somatic mutations as tumorigenic events in Lynch syndrome-associated adenomas retaining mismatch repair protein expression

Mitotic abnormalities precede microsatellite instability in lynch syndrome-associated colorectal tumourigenesis

Body Weight, Physical Activity, and Risk of Cancer in Lynch Syndrome

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