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Wilms' tumours: about tumour suppressor genes, an oncogene and a chameleon gene

Nature reviews. Cancer, 2011-02, Vol.11 (2), p.111

Huff, Vicki

2011

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Wt1 functions in ovarian follicle development by regulating granulosa cell differentiation

Human molecular genetics, 2014-01, Vol.23 (2), p.333-341

2014

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Reprogramming of Sertoli cells to fetal-like Leydig cells by Wt1 ablation

Proceedings of the National Academy of Sciences - PNAS, 2015-03, Vol.112 (13), p.4003-4008

2015

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Wilms tumor genetics: Mutations in WT1, WTX, and CTNNB1 account for only about one-third of tumors

Genes chromosomes & cancer, 2008-06, Vol.47 (6), p.461-470

2008

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Children's Oncology Group's 2013 blueprint for research: Renal tumors

Pediatric blood & cancer, 2013-06, Vol.60 (6), p.994-1000

2013

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The Wilms tumor gene, Wt1, is critical for mouse spermatogenesis via regulation of sertoli cell polarity and is associated with non-obstructive azoospermia in humans

PLoS genetics, 2013-08, Vol.9 (8), p.e1003645-e1003645

2013

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The Wilms Tumor Gene, Wt1, Is Required for Sox9 Expression and Maintenance of Tubular Architecture in the Developing Testis

Proceedings of the National Academy of Sciences - PNAS, 2006-08, Vol.103 (32), p.11987-11992

2006

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Clinically Relevant Subsets Identified by Gene Expression Patterns Support a Revised Ontogenic Model of Wilms Tumor: A Children's Oncology Group Study

Neoplasia (New York, N.Y.), 2012-08, Vol.14 (8), p.742-IN21

2012

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Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with β-Catenin Activation or Wt1 Ablation and Igf2 Upregulation

Neoplasia (New York, N.Y.), 2016-02, Vol.18 (2), p.71-81

2016

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Wilm's tumor 1 promotes memory flexibility

Nature communications, 2019-08, Vol.10 (1), p.3756-18, Article 3756

2019

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Rare variant detection using family-based sequencing analysis

Proceedings of the National Academy of Sciences - PNAS, 2013-03, Vol.110 (10), p.3985-3990

2013

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Ubiquitin specific protease 18 (Usp18) is a WT1 transcriptional target

Experimental cell research, 2013-03, Vol.319 (5), p.612-622

2013

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Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors

Translational oncology, 2014-08, Vol.7 (4), p.484-492

2014

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Wilms’ Tumor Blastemal Stem Cells Dedifferentiate to Propagate the Tumor Bulk

Stem cell reports, 2014-07, Vol.3 (1), p.24-33

2014

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Race Disparities in Peptide Profiles of North American and Kenyan Wilms Tumor Specimens

Journal of the American College of Surgeons, 2014-04, Vol.218 (4), p.707-720

2014

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Familial Wilms tumor

American journal of medical genetics. Part C, Seminars in medical genetics, 2004-08, Vol.129C (1), p.29-34

2004

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Twenty-four new cases of WT1 germline mutations and review of the literature: Genotype/phenotype correlations for Wilms tumor development

American journal of medical genetics, 2004-06, Vol.127A (3), p.249-257

2004

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Generation of a Wt1 conditional deletion, nuclear red fluorescent protein reporter allele in the mouse

Differentiation (London), 2024-07, Vol.138, p.100791, Article 100791

2024

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Wt1 directs the lineage specification of sertoli and granulosa cells by repressing Sf1 expression

Development (Cambridge), 2017-01, Vol.144 (1), p.44-53

2017

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β-Catenin Activation in a Novel Liver Progenitor Cell Type Is Sufficient to Cause Hepatocellular Carcinoma and Hepatoblastoma

Cancer research (Chicago, Ill.), 2014-08, Vol.74 (16), p.4515-4525

2014

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Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumour

Journal of medical genetics, 2016-06, Vol.53 (6), p.385-388

2016

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A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28

PloS one, 2018-12, Vol.13 (12), p.e0208936

2018

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Wt1 negatively regulates beta-catenin signaling during testis development

Development (Cambridge), 2008-05, Vol.135 (10), p.1875-1885

2008

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Wilms' tumours: about tumour suppressor genes, an oncogene and a chameleon gene

Wt1 functions in ovarian follicle development by regulating granulosa cell differentiation

Reprogramming of Sertoli cells to fetal-like Leydig cells by Wt1 ablation

Wilms tumor genetics: Mutations in WT1, WTX, and CTNNB1 account for only about one-third of tumors

Children's Oncology Group's 2013 blueprint for research: Renal tumors

The Wilms tumor gene, Wt1, is critical for mouse spermatogenesis via regulation of sertoli cell polarity and is associated with non-obstructive azoospermia in humans

The Wilms Tumor Gene, Wt1, Is Required for Sox9 Expression and Maintenance of Tubular Architecture in the Developing Testis

Clinically Relevant Subsets Identified by Gene Expression Patterns Support a Revised Ontogenic Model of Wilms Tumor: A Children's Oncology Group Study

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with β-Catenin Activation or Wt1 Ablation and Igf2 Upregulation

Wilm's tumor 1 promotes memory flexibility

Rare variant detection using family-based sequencing analysis

Ubiquitin specific protease 18 (Usp18) is a WT1 transcriptional target

Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors

Wilms’ Tumor Blastemal Stem Cells Dedifferentiate to Propagate the Tumor Bulk

Race Disparities in Peptide Profiles of North American and Kenyan Wilms Tumor Specimens

Familial Wilms tumor

Twenty-four new cases of WT1 germline mutations and review of the literature: Genotype/phenotype correlations for Wilms tumor development

Generation of a Wt1 conditional deletion, nuclear red fluorescent protein reporter allele in the mouse

Wt1 directs the lineage specification of sertoli and granulosa cells by repressing Sf1 expression

A Children's Oncology Group and TARGET initiative exploring the genetic landscape of Wilms tumor

β-Catenin Activation in a Novel Liver Progenitor Cell Type Is Sufficient to Cause Hepatocellular Carcinoma and Hepatoblastoma

Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumour

A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28

Recurrent DGCR8, DROSHA, and SIX Homeodomain Mutations in Favorable Histology Wilms Tumors

Wt1 negatively regulates beta-catenin signaling during testis development

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