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Placental Growth Factor Regulates Cardiac Inflammation Through the Tissue Inhibitor of Metalloproteinases-3/Tumor Necrosis Factor-α―Converting Enzyme Axis: Crucial Role for Adaptive Cardiac Remodeling During Cardiac Pressure Overload

Circulation (New York, N.Y.), 2011-09, Vol.124 (12), p.1337-1350

2011

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The PTEN/PI3K pathway governs normal vascular development and tumor angiogenesis

Genes & development, 2005-09, Vol.19 (17), p.2054-2065

2005

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Pathogenic DDX3X Mutations Impair RNA Metabolism and Neurogenesis during Fetal Cortical Development

Neuron (Cambridge, Mass.), 2020-05, Vol.106 (3), p.404-420.e8

2020

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A PKM2 signature in the failing heart

Biochemical and biophysical research communications, 2015-04, Vol.459 (3), p.430-436

2015

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Embryonic Stem Cell‐Derived Mesenchymal Stem Cells (MSCs) Have a Superior Neuroprotective Capacity Over Fetal MSCs in the Hypoxic‐Ischemic Mouse Brain

Stem cells translational medicine, 2018-05, Vol.7 (5), p.439-449

2018

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Endothelial mineralocorticoid receptor contributes to systolic dysfunction induced by pressure overload without modulating cardiac hypertrophy or inflammation

Physiological reports, 2017-06, Vol.5 (12), p.e13313-n/a

2017

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Cardiac lymphatics are heterogeneous in origin and respond to injury

Nature (London), 2015-06, Vol.522 (7554), p.62-67

2015

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Maternal and fetal roles in bacterially induced preterm labor in the mouse

American journal of obstetrics and gynecology, 2016-03, Vol.214 (3), p.386.e1-386.e9

2016

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Impaired cardiac performance, protein synthesis, and mitochondrial function in tumor-bearing mice

PloS one, 2019-12, Vol.14 (12), p.e0226440-e0226440

2019

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Stem Cell-Derived Exosomes Prevent Aging-Induced Cardiac Dysfunction through a Novel Exosome/lncRNA MALAT1/NF-κB/TNF-α Signaling Pathway

Oxidative medicine and cellular longevity, 2019-01, Vol.2019, p.9739258-14

2019

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Genistein inhibits TNF-α-induced endothelial inflammation through the protein kinase pathway A and improves vascular inflammation in C57BL/6 mice

International journal of cardiology, 2013-10, Vol.168 (3), p.2637-2645

2013

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FADD: Essential for Embryo Development and Signaling from Some, but Not All, Inducers of Apoptosis

Science (American Association for the Advancement of Science), 1998-03, Vol.279 (5358), p.1954-1958

1998

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MiR-155 Knockout in Fibroblasts Improves Cardiac Remodeling by Targeting Tumor Protein p53-Inducible Nuclear Protein 1

Journal of cardiovascular pharmacology and therapeutics, 2016-07, Vol.21 (4), p.423-435

2016

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5TNF-α and IL-1β Neutralization Ameliorates Angiotensin II-Induced Cardiac Damage in Male Mice

Endocrinology (Philadelphia), 2014-07, Vol.155 (7), p.2677-2687

2014

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Alleviation of Inflammation and Oxidative Stress in Pressure Overload-Induced Cardiac Remodeling and Heart Failure via IL-6/STAT3 Inhibition by Raloxifene

Oxidative medicine and cellular longevity, 2021, Vol.2021, p.6699054-15

2021

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Isoquercitrin Attenuated Cardiac Dysfunction Via AMPKα‐Dependent Pathways in LPS‐Treated Mice

Molecular nutrition & food research, 2018-12, Vol.62 (24), p.e1800955-n/a

2018

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Decreased thioredoxin reductase 3 expression promotes nickel‐induced damage to cardiac tissue via activating oxidative stress‐induced apoptosis and inflammation

Environmental toxicology, 2023-02, Vol.38 (2), p.436-450

2023

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Inhibition of CTRP9, a novel and cardiac-abundantly expressed cell survival molecule, by TNFα-initiated oxidative signaling contributes to exacerbated cardiac injury in diabetic mice

Basic research in cardiology, 2013-01, Vol.108 (1), p.315-315, Article 315

2013

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N-myc downstream-regulated gene 4, up-regulated by tumor necrosis factor-α and nuclear factor kappa B, aggravates cardiac ischemia/reperfusion injury by inhibiting reperfusion injury salvage kinase pathway

Basic research in cardiology, 2016-03, Vol.111 (2), p.11-11, Article 11

2016

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Exposure to Stress Alters Cardiac Gene Expression and Exacerbates Myocardial Ischemic Injury in the Female Murine Heart

International journal of molecular sciences, 2023-07, Vol.24 (13), p.10994

2023

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C1q-TNF-related protein-3 attenuates pressure overload-induced cardiac hypertrophy by suppressing the p38/CREB pathway and p38-induced ER stress

Cell death & disease, 2019-07, Vol.10 (7), p.520-19, Article 520

2019

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C1q/Tumor Necrosis Factor-Related Protein 9 Protects against Acute Myocardial Injury through an Adiponectin Receptor I-AMPK-Dependent Mechanism

Molecular and cellular biology, 2015-06, Vol.35 (12), p.2173-2185

2015

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Mitochondrial permeability regulates cardiac endothelial cell necroptosis and cardiac allograft rejection

American journal of transplantation, 2019-03, Vol.19 (3), p.686-698

2019

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Bone marrow-derived human mesenchymal stem cells express cardiomyogenic proteins but do not exhibit functional cardiomyogenic differentiation potential

Stem cells and development, 2012-09, Vol.21 (13), p.2457

2012

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MyD88 mediated inflammatory signaling leads to CaMKII oxidation, cardiac hypertrophy and death after myocardial infarction

Journal of molecular and cellular cardiology, 2012-05, Vol.52 (5), p.1135-1144

2012

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Placental Growth Factor Regulates Cardiac Inflammation Through the Tissue Inhibitor of Metalloproteinases-3/Tumor Necrosis Factor-α―Converting Enzyme Axis: Crucial Role for Adaptive Cardiac Remodeling During Cardiac Pressure Overload

The PTEN/PI3K pathway governs normal vascular development and tumor angiogenesis

Pathogenic DDX3X Mutations Impair RNA Metabolism and Neurogenesis during Fetal Cortical Development

A PKM2 signature in the failing heart

Embryonic Stem Cell‐Derived Mesenchymal Stem Cells (MSCs) Have a Superior Neuroprotective Capacity Over Fetal MSCs in the Hypoxic‐Ischemic Mouse Brain

Endothelial mineralocorticoid receptor contributes to systolic dysfunction induced by pressure overload without modulating cardiac hypertrophy or inflammation

Cardiac lymphatics are heterogeneous in origin and respond to injury

Maternal and fetal roles in bacterially induced preterm labor in the mouse

Impaired cardiac performance, protein synthesis, and mitochondrial function in tumor-bearing mice

Stem Cell-Derived Exosomes Prevent Aging-Induced Cardiac Dysfunction through a Novel Exosome/lncRNA MALAT1/NF-κB/TNF-α Signaling Pathway

Genistein inhibits TNF-α-induced endothelial inflammation through the protein kinase pathway A and improves vascular inflammation in C57BL/6 mice

FADD: Essential for Embryo Development and Signaling from Some, but Not All, Inducers of Apoptosis

MiR-155 Knockout in Fibroblasts Improves Cardiac Remodeling by Targeting Tumor Protein p53-Inducible Nuclear Protein 1

5TNF-α and IL-1β Neutralization Ameliorates Angiotensin II-Induced Cardiac Damage in Male Mice

Alleviation of Inflammation and Oxidative Stress in Pressure Overload-Induced Cardiac Remodeling and Heart Failure via IL-6/STAT3 Inhibition by Raloxifene

Isoquercitrin Attenuated Cardiac Dysfunction Via AMPKα‐Dependent Pathways in LPS‐Treated Mice

Decreased thioredoxin reductase 3 expression promotes nickel‐induced damage to cardiac tissue via activating oxidative stress‐induced apoptosis and inflammation

Inhibition of CTRP9, a novel and cardiac-abundantly expressed cell survival molecule, by TNFα-initiated oxidative signaling contributes to exacerbated cardiac injury in diabetic mice

N-myc downstream-regulated gene 4, up-regulated by tumor necrosis factor-α and nuclear factor kappa B, aggravates cardiac ischemia/reperfusion injury by inhibiting reperfusion injury salvage kinase pathway

Exposure to Stress Alters Cardiac Gene Expression and Exacerbates Myocardial Ischemic Injury in the Female Murine Heart

C1q-TNF-related protein-3 attenuates pressure overload-induced cardiac hypertrophy by suppressing the p38/CREB pathway and p38-induced ER stress

C1q/Tumor Necrosis Factor-Related Protein 9 Protects against Acute Myocardial Injury through an Adiponectin Receptor I-AMPK-Dependent Mechanism

Mitochondrial permeability regulates cardiac endothelial cell necroptosis and cardiac allograft rejection

Bone marrow-derived human mesenchymal stem cells express cardiomyogenic proteins but do not exhibit functional cardiomyogenic differentiation potential

MyD88 mediated inflammatory signaling leads to CaMKII oxidation, cardiac hypertrophy and death after myocardial infarction

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