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Nicotinamide mononucleotide alters mitochondrial dynamics by SIRT3‐dependent mechanism in male mice

Journal of neuroscience research, 2019-08, Vol.97 (8), p.975-990

2019

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Multi-targeted Effect of Nicotinamide Mononucleotide on Brain Bioenergetic Metabolism

Neurochemical research, 2019-10, Vol.44 (10), p.2280-2287

2019

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Nicotinamide mononucleotide inhibits post-ischemic NAD+ degradation and dramatically ameliorates brain damage following global cerebral ischemia

Neurobiology of disease, 2016-11, Vol.95, p.102-110

2016

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Nicotinamide Mononucleotide Administration Prevents Experimental Diabetes-Induced Cognitive Impairment and Loss of Hippocampal Neurons

International journal of molecular sciences, 2020-05, Vol.21 (11), p.3756

2020

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Effect of nicotinamide mononucleotide on brain mitochondrial respiratory deficits in an Alzheimer's disease-relevant murine model

BMC neurology, 2015-03, Vol.15 (1), p.19-19, Article 19

2015

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Brain energy metabolism: A roadmap for future research

Journal of neurochemistry, 2024-05, Vol.168 (5), p.910-954

2024

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NICOTINAMIDE MONONUCLEOTIDE PREVENTS ISCHEMIA INDUCED MITOCHONDRIAL PROTEIN HYPERACETYLATION AND INCREASE IN FREE RADICALS PRODUCTION

IBRO neuroscience reports, 2023-10, Vol.15, p.S593-S593

2023

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Mitochondrial dynamics: cell-type and hippocampal region specific changes following global cerebral ischemia

Journal of bioenergetics and biomembranes, 2015-04, Vol.47 (1-2), p.13-31

2015

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Mechanisms of impaired mitochondrial energy metabolism in acute and chronic neurodegenerative disorders

Journal of neuroscience research, 2007-11, Vol.85 (15), p.3407-3415

2007

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NAD + Precursors Reverse Experimental Diabetic Neuropathy in Mice

International journal of molecular sciences, 2024-01, Vol.25 (2), p.1102

2024

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Role of NAD + -Modulated Mitochondrial Free Radical Generation in Mechanisms of Acute Brain Injury

Brain sciences, 2020-07, Vol.10 (7), p.449

2020

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Guidelines for Using Mouse Global Cerebral Ischemia Models

Translational stroke research, 2013-06, Vol.4 (3), p.343-350

2013

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Hyperoxic reperfusion after global cerebral ischemia promotes inflammation and long-term hippocampal neuronal death

Journal of neurotrauma, 2010-04, Vol.27 (4), p.753-762

2010

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NAD+ precursor modulates post-ischemic mitochondrial fragmentation and reactive oxygen species generation via SIRT3 dependent mechanisms

Experimental neurology, 2020-03, Vol.325, p.113144-113144, Article 113144

2020

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Genetic inhibition of PKCε attenuates neurodegeneration after global cerebral ischemia in male mice

Journal of neuroscience research, 2019-04, Vol.97 (4), p.444-455

2019

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Calcium‐induced precipitate formation in brain mitochondria: composition, calcium capacity, and retention

Journal of neurochemistry, 2007-08, Vol.102 (4), p.1346-1356

2007

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Astrocyte mitochondrial mechanisms of ischemic brain injury and neuroprotection

Neurochemical research, 2004-03, Vol.29 (3), p.601-608

2004

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Calcium in ischemic cell death

Stroke (1970), 1998-03, Vol.29 (3), p.705-718

1998

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Brain diabetic neurodegeneration segregates with low intrinsic aerobic capacity

Annals of clinical and translational neurology, 2014-08, Vol.1 (8), p.589-604

2014

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Neuron-Specific Conditional Expression of a Mitochondrially Targeted Fluorescent Protein in Mice

The Journal of neuroscience, 2006-12, Vol.26 (51), p.13123-13127

2006

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Isolation of mitochondria from the CNS

Current Protocols in Neuroscience, 2010-07, Vol.Chapter 7 (1), p.Unit 7.22

Kristian, Tibor

2010

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Interplay between NAD+ and acetyl‑CoA metabolism in ischemia-induced mitochondrial pathophysiology

Biochimica et biophysica acta. Molecular basis of disease, 2019-08, Vol.1865 (8), p.2060-2067

2019

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Mitochondrial NUDIX hydrolases: A metabolic link between NAD catabolism, GTP and mitochondrial dynamics

Neurochemistry international, 2017-10, Vol.109, p.193-201

2017

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The Protein Modification and Degradation Pathways after Brain Ischemia

Translational stroke research, 2018-06, Vol.9 (3), p.199-200

2018

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Significance of Mitochondrial Protein Post-translational Modifications in Pathophysiology of Brain Injury

Translational stroke research, 2018-06, Vol.9 (3), p.223-237

2018

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Nicotinamide mononucleotide alters mitochondrial dynamics by SIRT3‐dependent mechanism in male mice

Multi-targeted Effect of Nicotinamide Mononucleotide on Brain Bioenergetic Metabolism

Nicotinamide mononucleotide inhibits post-ischemic NAD+ degradation and dramatically ameliorates brain damage following global cerebral ischemia

Nicotinamide Mononucleotide Administration Prevents Experimental Diabetes-Induced Cognitive Impairment and Loss of Hippocampal Neurons

Effect of nicotinamide mononucleotide on brain mitochondrial respiratory deficits in an Alzheimer's disease-relevant murine model

Brain energy metabolism: A roadmap for future research

NICOTINAMIDE MONONUCLEOTIDE PREVENTS ISCHEMIA INDUCED MITOCHONDRIAL PROTEIN HYPERACETYLATION AND INCREASE IN FREE RADICALS PRODUCTION

Mitochondrial dynamics: cell-type and hippocampal region specific changes following global cerebral ischemia

Mechanisms of impaired mitochondrial energy metabolism in acute and chronic neurodegenerative disorders

NAD + Precursors Reverse Experimental Diabetic Neuropathy in Mice

Role of NAD + -Modulated Mitochondrial Free Radical Generation in Mechanisms of Acute Brain Injury

Guidelines for Using Mouse Global Cerebral Ischemia Models

Hyperoxic reperfusion after global cerebral ischemia promotes inflammation and long-term hippocampal neuronal death

NAD+ precursor modulates post-ischemic mitochondrial fragmentation and reactive oxygen species generation via SIRT3 dependent mechanisms

Genetic inhibition of PKCε attenuates neurodegeneration after global cerebral ischemia in male mice

Calcium‐induced precipitate formation in brain mitochondria: composition, calcium capacity, and retention

Astrocyte mitochondrial mechanisms of ischemic brain injury and neuroprotection

Calcium in ischemic cell death

Brain diabetic neurodegeneration segregates with low intrinsic aerobic capacity

Neuron-Specific Conditional Expression of a Mitochondrially Targeted Fluorescent Protein in Mice

Isolation of mitochondria from the CNS

Interplay between NAD+ and acetyl‑CoA metabolism in ischemia-induced mitochondrial pathophysiology

Mitochondrial NUDIX hydrolases: A metabolic link between NAD catabolism, GTP and mitochondrial dynamics

The Protein Modification and Degradation Pathways after Brain Ischemia

Significance of Mitochondrial Protein Post-translational Modifications in Pathophysiology of Brain Injury

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