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Dysregulation of mitochondria-lysosome contacts by GBA1 dysfunction in dopaminergic neuronal models of Parkinson's disease

Nature communications, 2021-03, Vol.12 (1), p.1807-1807, Article 1807

2021

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Mitochondria-lysosome contacts regulate mitochondrial fission via RAB7 GTP hydrolysis

Nature (London), 2018-02, Vol.554 (7692), p.382-386

2018

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Mitochondria-lysosome contacts regulate mitochondrial Ca2+ dynamics via lysosomal TRPML1

Proceedings of the National Academy of Sciences - PNAS, 2020-08, Vol.117 (32), p.19266-19275

2020

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Zn2+ dyshomeostasis caused by loss of ATP13A2/PARK9 leads to lysosomal dysfunction and alpha-synuclein accumulation

Human molecular genetics, 2014-06, Vol.23 (11), p.2791-2801

2014

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Synaptic, Mitochondrial, and Lysosomal Dysfunction in Parkinson’s Disease

Trends in neurosciences (Regular ed.), 2019-02, Vol.42 (2), p.140-149

2019

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Regulation and Function of Mitochondria–Lysosome Membrane Contact Sites in Cellular Homeostasis

Trends in cell biology, 2019-06, Vol.29 (6), p.500-513

2019

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Parkin Modulates Endosomal Organization and Function of the Endo-Lysosomal Pathway

The Journal of neuroscience, 2016-02, Vol.36 (8), p.2425-2437

2016

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α-Synuclein–induced lysosomal dysfunction occurs through disruptions in protein trafficking in human midbrain synucleinopathy models

Proceedings of the National Academy of Sciences - PNAS, 2016-02, Vol.113 (7), p.1931-1936

2016

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Lysosomal Regulation of Inter-mitochondrial Contact Fate and Motility in Charcot-Marie-Tooth Type 2

Developmental cell, 2019-08, Vol.50 (3), p.339-354.e4

2019

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LRRK2 kinase activity regulates lysosomal glucocerebrosidase in neurons derived from Parkinson's disease patients

Nature communications, 2019-12, Vol.10 (1), p.5570-9, Article 5570

2019

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Mutant Huntingtin Is Secreted via a Late Endosomal/Lysosomal Unconventional Secretory Pathway

The Journal of neuroscience, 2017-09, Vol.37 (37), p.9000-9012

2017

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Mitochondrial Parkin recruitment is impaired in neurons derived from mutant PINK1 induced pluripotent stem cells

The Journal of neuroscience, 2011-04, Vol.31 (16), p.5970-5976

2011

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Dopamine oxidation mediates mitochondrial and lysosomal dysfunction in Parkinson’s disease

Science (American Association for the Advancement of Science), 2017-09, Vol.357 (6357), p.1255-1261

2017

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Functional Impairment in Miro Degradation and Mitophagy Is a Shared Feature in Familial and Sporadic Parkinson’s Disease

Cell stem cell, 2016-12, Vol.19 (6), p.709-724

2016

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Progranulin mutations result in impaired processing of prosaposin and reduced glucocerebrosidase activity

Human molecular genetics, 2020-03, Vol.29 (5), p.716-726

2020

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Emerging links between pediatric lysosomal storage diseases and adult parkinsonism

Movement disorders, 2019-05, Vol.34 (5), p.614-624

2019

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Progranulin-mediated deficiency of cathepsin D results in FTD and NCL-like phenotypes in neurons derived from FTD patients

Human molecular genetics, 2017-12, Vol.26 (24), p.4861-4872

2017

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The role of dopamine in the pathogenesis of GBA1-linked Parkinson's disease

Neurobiology of disease, 2019-12, Vol.132, p.104545-104545, Article 104545

2019

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Dopamine metabolism by a monoamine oxidase mitochondrial shuttle activates the electron transport chain

Nature neuroscience, 2020-01, Vol.23 (1), p.15-20

2020

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Identification of ASCL1 as a determinant for human iPSC-derived dopaminergic neurons

Scientific reports, 2021-11, Vol.11 (1), p.22257-22257, Article 22257

2021

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Protocol to investigate Parkinson’s patient-derived dopaminergic neurons by live-cell microscopy and oxidized dopamine assays

STAR protocols, 2024-03, Vol.5 (1), p.102889-102889, Article 102889

2024

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Human iPSC-Based Modeling of Late-Onset Disease via Progerin-Induced Aging

Cell stem cell, 2013-12, Vol.13 (6), p.691-705

2013

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Rapamycin reverses cellular phenotypes and enhances mutant protein clearance in Hutchinson-Gilford progeria syndrome cells

Science translational medicine, 2011-06, Vol.3 (89), p.89ra58

2011

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Sirt1 mediates neuroprotection from mutant huntingtin by activation of the TORC1 and CREB transcriptional pathway

Nature medicine, 2012-01, Vol.18 (1), p.159-165

2012

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Lysosomal dysfunction in neurodegeneration: The role of ATP13A2/PARK9

Autophagy, 2012-06, Vol.8 (6), p.987-988

2012

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Dysregulation of mitochondria-lysosome contacts by GBA1 dysfunction in dopaminergic neuronal models of Parkinson's disease

Mitochondria-lysosome contacts regulate mitochondrial fission via RAB7 GTP hydrolysis

Mitochondria-lysosome contacts regulate mitochondrial Ca2+ dynamics via lysosomal TRPML1

Zn2+ dyshomeostasis caused by loss of ATP13A2/PARK9 leads to lysosomal dysfunction and alpha-synuclein accumulation

Synaptic, Mitochondrial, and Lysosomal Dysfunction in Parkinson’s Disease

Regulation and Function of Mitochondria–Lysosome Membrane Contact Sites in Cellular Homeostasis

Parkin Modulates Endosomal Organization and Function of the Endo-Lysosomal Pathway

α-Synuclein–induced lysosomal dysfunction occurs through disruptions in protein trafficking in human midbrain synucleinopathy models

Lysosomal Regulation of Inter-mitochondrial Contact Fate and Motility in Charcot-Marie-Tooth Type 2

LRRK2 kinase activity regulates lysosomal glucocerebrosidase in neurons derived from Parkinson's disease patients

Mutant Huntingtin Is Secreted via a Late Endosomal/Lysosomal Unconventional Secretory Pathway

Mitochondrial Parkin recruitment is impaired in neurons derived from mutant PINK1 induced pluripotent stem cells

Dopamine oxidation mediates mitochondrial and lysosomal dysfunction in Parkinson’s disease

Functional Impairment in Miro Degradation and Mitophagy Is a Shared Feature in Familial and Sporadic Parkinson’s Disease

Progranulin mutations result in impaired processing of prosaposin and reduced glucocerebrosidase activity

Emerging links between pediatric lysosomal storage diseases and adult parkinsonism

Progranulin-mediated deficiency of cathepsin D results in FTD and NCL-like phenotypes in neurons derived from FTD patients

The role of dopamine in the pathogenesis of GBA1-linked Parkinson's disease

Dopamine metabolism by a monoamine oxidase mitochondrial shuttle activates the electron transport chain

Identification of ASCL1 as a determinant for human iPSC-derived dopaminergic neurons

Protocol to investigate Parkinson’s patient-derived dopaminergic neurons by live-cell microscopy and oxidized dopamine assays

Human iPSC-Based Modeling of Late-Onset Disease via Progerin-Induced Aging

Rapamycin reverses cellular phenotypes and enhances mutant protein clearance in Hutchinson-Gilford progeria syndrome cells

Sirt1 mediates neuroprotection from mutant huntingtin by activation of the TORC1 and CREB transcriptional pathway

Lysosomal dysfunction in neurodegeneration: The role of ATP13A2/PARK9

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