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The microglial NLRP3 inflammasome is activated by amyotrophic lateral sclerosis proteins

Glia, 2020-02, Vol.68 (2), p.407-421

2020

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Prion-Like Propagation of Protein Misfolding and Aggregation in Amyotrophic Lateral Sclerosis

Frontiers in molecular neuroscience, 2019-11, Vol.12, p.262-262

2019

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Extracellular chaperones and proteostasis

Annual review of biochemistry, 2013-01, Vol.82 (1), p.295-322

2013

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Intercellular propagated misfolding of wild-type Cu/Zn superoxide dismutase occurs via exosome-dependent and -independent mechanisms

Proceedings of the National Academy of Sciences - PNAS, 2014-03, Vol.111 (9), p.3620-3625

2014

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The heat shock response in neurons and astroglia and its role in neurodegenerative diseases

Molecular neurodegeneration, 2017-09, Vol.12 (1), p.65-20, Article 65

2017

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Nucleo-cytoplasmic transport of TDP-43 studied in real time: impaired microglia function leads to axonal spreading of TDP-43 in degenerating motor neurons

Acta neuropathologica, 2018-09, Vol.136 (3), p.445-459

2018

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Tryptophan residue 32 in human Cu-Zn superoxide dismutase modulates prion-like propagation and strain selection

PloS one, 2020-01, Vol.15 (1), p.e0227655

2020

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The cysteine-reactive small molecule ebselen facilitates effective SOD1 maturation

Nature communications, 2018-04, Vol.9 (1), p.1693-9, Article 1693

2018

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Strategies to promote the maturation of ALS-associated SOD1 mutants: small molecules return to the fold

Neural regeneration research, 2019-09, Vol.14 (9), p.1511-1512

2019

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P2X7 Receptor Activation Induces Reactive Oxygen Species Formation and Cell Death in Murine EOC13 Microglia

Mediators of Inflammation, 2013-01, Vol.2013 (2013), p.1-18

2013

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SOD1A4V aggregation alters ubiquitin homeostasis in a cell model of ALS

Journal of cell science, 2018-06, Vol.131 (11), p.jcs209122-jcs209122

2018

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Mutant Cu/Zn Superoxide Dismutase (A4V) Turnover Is Altered in Cells Containing Inclusions

Frontiers in molecular neuroscience, 2021-11, Vol.14, p.771911-771911

2021

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High-content analysis of proteostasis capacity in cellular models of amyotrophic lateral sclerosis (ALS)

Scientific reports, 2024-06, Vol.14 (1), p.13844-11

2024

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ANS Binding Reveals Common Features of Cytotoxic Amyloid Species

ACS chemical biology, 2010-08, Vol.5 (8), p.735-740

2010

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Disease Mechanisms in ALS: Misfolded SOD1 Transferred Through Exosome-Dependent and Exosome-Independent Pathways

Cellular and molecular neurobiology, 2016-04, Vol.36 (3), p.377-381

2016

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Misfolding at the synapse: A role in amyotrophic lateral sclerosis pathogenesis?

Frontiers in molecular neuroscience, 2022-09, Vol.15, p.997661-997661

2022

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Extracellular aggregated Cu/Zn superoxide dismutase activates microglia to give a cytotoxic phenotype

Glia, 2013-03, Vol.61 (3), p.409-419

2013

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Clusterin facilitates in vivo clearance of extracellular misfolded proteins

Cellular and molecular life sciences : CMLS, 2011-12, Vol.68 (23), p.3919-3931

2011

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TDP-43 is a ubiquitylation substrate of the SCFcyclin F complex

Neurobiology of disease, 2022-06, Vol.167, p.105673-105673, Article 105673

2022

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Pathogenic mutation in the ALS/FTD gene, CCNF, causes elevated Lys48-linked ubiquitylation and defective autophagy

Cellular and molecular life sciences : CMLS, 2018-01, Vol.75 (2), p.335-354

2018

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Improving the Delivery of SOD1 Antisense Oligonucleotides to Motor Neurons Using Calcium Phosphate-Lipid Nanoparticles

Frontiers in neuroscience, 2017-08, Vol.11, p.476-476

2017

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Disabled in academia: to be or not to be, that is the question

Trends in neurosciences (Regular ed.), 2021-07, Vol.44 (7), p.507-509

2021

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Ubiquitin homeostasis disruption, a common cause of proteostasis collapse in amyotrophic lateral sclerosis?

Neural regeneration research, 2022-10, Vol.17 (10), p.2218-2220

2022

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Structural Characterization of Clusterin-Chaperone Client Protein Complexes

The Journal of biological chemistry, 2009-08, Vol.284 (33), p.21920-21927

2009

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SerpinB2 (PAI-2) Modulates Proteostasis via Binding Misfolded Proteins and Promotion of Cytoprotective Inclusion Formation

PloS one, 2015-06, Vol.10 (6), p.e0130136-e0130136

2015

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The microglial NLRP3 inflammasome is activated by amyotrophic lateral sclerosis proteins

Prion-Like Propagation of Protein Misfolding and Aggregation in Amyotrophic Lateral Sclerosis

Extracellular chaperones and proteostasis

Intercellular propagated misfolding of wild-type Cu/Zn superoxide dismutase occurs via exosome-dependent and -independent mechanisms

The heat shock response in neurons and astroglia and its role in neurodegenerative diseases

Nucleo-cytoplasmic transport of TDP-43 studied in real time: impaired microglia function leads to axonal spreading of TDP-43 in degenerating motor neurons

Tryptophan residue 32 in human Cu-Zn superoxide dismutase modulates prion-like propagation and strain selection

The cysteine-reactive small molecule ebselen facilitates effective SOD1 maturation

Strategies to promote the maturation of ALS-associated SOD1 mutants: small molecules return to the fold

P2X7 Receptor Activation Induces Reactive Oxygen Species Formation and Cell Death in Murine EOC13 Microglia

SOD1A4V aggregation alters ubiquitin homeostasis in a cell model of ALS

Mutant Cu/Zn Superoxide Dismutase (A4V) Turnover Is Altered in Cells Containing Inclusions

High-content analysis of proteostasis capacity in cellular models of amyotrophic lateral sclerosis (ALS)

ANS Binding Reveals Common Features of Cytotoxic Amyloid Species

Disease Mechanisms in ALS: Misfolded SOD1 Transferred Through Exosome-Dependent and Exosome-Independent Pathways

Misfolding at the synapse: A role in amyotrophic lateral sclerosis pathogenesis?

Extracellular aggregated Cu/Zn superoxide dismutase activates microglia to give a cytotoxic phenotype

Clusterin facilitates in vivo clearance of extracellular misfolded proteins

TDP-43 is a ubiquitylation substrate of the SCFcyclin F complex

Pathogenic mutation in the ALS/FTD gene, CCNF, causes elevated Lys48-linked ubiquitylation and defective autophagy

Improving the Delivery of SOD1 Antisense Oligonucleotides to Motor Neurons Using Calcium Phosphate-Lipid Nanoparticles

Disabled in academia: to be or not to be, that is the question

Ubiquitin homeostasis disruption, a common cause of proteostasis collapse in amyotrophic lateral sclerosis?

Structural Characterization of Clusterin-Chaperone Client Protein Complexes

SerpinB2 (PAI-2) Modulates Proteostasis via Binding Misfolded Proteins and Promotion of Cytoprotective Inclusion Formation

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