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Selective Modulation of Coupled Ryanodine Receptors During Microdomain Activation of Calcium/Calmodulin-Dependent Kinase II in the Dyadic Cleft

Circulation research, 2013-11, Vol.113 (11), p.1242-1252

2013

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Calcium/calmodulin‐dependent kinase II and nitric oxide synthase 1‐dependent modulation of ryanodine receptors during β‐adrenergic stimulation is restricted to the dyadic cleft

The Journal of physiology, 2016-10, Vol.594 (20), p.5923-5939

2016

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Microdomain [Ca2+] near ryanodine receptors as reported by L‐type Ca2+ and Na+/Ca2+ exchange currents

The Journal of physiology, 2011-05, Vol.589 (10), p.2569-2583

2011

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Pharmacological Inhibition of Na/Ca Exchange Results in Increased Cellular Ca2 Load Attributable to the Predominance of Forward Mode Block

Circulation research, 2008-06, Vol.102 (11), p.1398-1405

2008

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T-tubules and ryanodine receptor microdomains: on the road to translation

Cardiovascular research, 2013-05, Vol.98 (2), p.159-161

2013

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Dyssynchrony of Ca2+ release from the sarcoplasmic reticulum as subcellular mechanism of cardiac contractile dysfunction

Journal of molecular and cellular cardiology, 2011-03, Vol.50 (3), p.390-400

2011

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Impaired calcium homeostasis is associated with sudden cardiac death and arrhythmias in a genetic equivalent mouse model of the human HRC-Ser96Ala variant

Cardiovascular research, 2017-09, Vol.113 (11), p.1403-1417

2017

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Calcium overload, spontaneous calcium release, and ventricular arrhythmias

Heart rhythm, 2006-08, Vol.3 (8), p.977-979

Sipido, Karin R.

2006

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Targeting Sarcoplasmic Reticulum Ca2+ Uptake to Improve Heart Failure: Hit or Miss

Circulation research, 2010-02, Vol.106 (2), p.230-233

2010

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Calcium release near l -type calcium channels promotes beat-to-beat variability in ventricular myocytes from the chronic AV block dog

Journal of molecular and cellular cardiology, 2015-12, Vol.89 (Pt B), p.326-334

2015

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CaM or cAMP: Linking β-Adrenergic Stimulation to ‘Leaky’ RyRs

Circulation research, 2007-02, Vol.100 (3), p.296-298

Sipido, Karin R

2007

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T-tubule remodelling and ryanodine receptor organization modulate sodium-calcium exchange

Advances in experimental medicine and biology, 2013, Vol.961, p.375

2013

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Blink and Youʼll See It: How to Detect Ca2+ Quarks

Circulation research, 2011-01, Vol.108 (2), p.154-156

2011

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Phospholamban ablation in hearts expressing the high affinity SERCA2b isoform normalizes global Ca²⁺ homeostasis but not Ca²⁺-dependent hypertrophic signaling

American journal of physiology. Heart and circulatory physiology, 2012-06, Vol.302 (12), p.H2574-H2582

2012

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Inhibition of the calcium-activated chloride current in cardiac ventricular myocytes by N-( p-amylcinnamoyl)anthranilic acid (ACA)

Biochemical and biophysical research communications, 2010-11, Vol.402 (3), p.531-536

2010

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Crosstalk between L-type Ca2+ channels and the sarcoplasmic reticulum: alterations during cardiac remodelling

Cardiovascular research, 2008-01, Vol.77 (2), p.315-324

2008

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Targeting calcium handling in arrhythmias

Europace (London, England), 2008-12, Vol.10 (12), p.1364-1369

2008

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Intracellular na in animal models of hypertrophy and heart failure: contractile function and arrhythmogenesis

Cardiovascular research, 2003-03, Vol.57 (4), p.887-896

2003

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Window Ca2+ current and its modulation by Ca2+ release in hypertrophied cardiac myocytes from dogs with chronic atrioventricular block

The Journal of physiology, 2007-02, Vol.579 (1), p.147-160

2007

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Progress in the understanding of cardiac early afterdepolarizations and torsades de pointes : time to revise current concepts

Cardiovascular research, 2000-06, Vol.46 (3), p.376-392

2000

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Enhanced Ca(2+) release and Na/Ca exchange activity in hypertrophied canine ventricular myocytes: potential link between contractile adaptation and arrhythmogenesis

Circulation (New York, N.Y.), 2000-10, Vol.102 (17), p.2137-2144

2000

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Impairment of trafficking by downregulation of an anchor protein: Novel insights into additional mechanisms responsible for heart failure

Heart rhythm, 2012-05, Vol.9 (5), p.821-822

2012

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Early exercise training after myocardial infarction prevents contractile but not electrical remodelling or hypertrophy

Cardiovascular research, 2010-04, Vol.86 (1), p.72-81

2010

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T-type Ca2+ current as a trigger for Ca2+ release from the sarcoplasmic reticulum in guinea-pig ventricular myocytes

The Journal of physiology, 1998-04, Vol.508 (2), p.439-451

1998

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A SERCA2 pump with an increased Ca2+ affinity can lead to severe cardiac hypertrophy, stress intolerance and reduced life span

Journal of molecular and cellular cardiology, 2006-08, Vol.41 (2), p.308-317

2006

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Selective Modulation of Coupled Ryanodine Receptors During Microdomain Activation of Calcium/Calmodulin-Dependent Kinase II in the Dyadic Cleft

Calcium/calmodulin‐dependent kinase II and nitric oxide synthase 1‐dependent modulation of ryanodine receptors during β‐adrenergic stimulation is restricted to the dyadic cleft

Microdomain [Ca2+] near ryanodine receptors as reported by L‐type Ca2+ and Na+/Ca2+ exchange currents

Pharmacological Inhibition of Na/Ca Exchange Results in Increased Cellular Ca2 Load Attributable to the Predominance of Forward Mode Block

T-tubules and ryanodine receptor microdomains: on the road to translation

Dyssynchrony of Ca2+ release from the sarcoplasmic reticulum as subcellular mechanism of cardiac contractile dysfunction

Impaired calcium homeostasis is associated with sudden cardiac death and arrhythmias in a genetic equivalent mouse model of the human HRC-Ser96Ala variant

Calcium overload, spontaneous calcium release, and ventricular arrhythmias

Targeting Sarcoplasmic Reticulum Ca2+ Uptake to Improve Heart Failure: Hit or Miss

Calcium release near l -type calcium channels promotes beat-to-beat variability in ventricular myocytes from the chronic AV block dog

CaM or cAMP: Linking β-Adrenergic Stimulation to ‘Leaky’ RyRs

T-tubule remodelling and ryanodine receptor organization modulate sodium-calcium exchange

Blink and Youʼll See It: How to Detect Ca2+ Quarks

Phospholamban ablation in hearts expressing the high affinity SERCA2b isoform normalizes global Ca²⁺ homeostasis but not Ca²⁺-dependent hypertrophic signaling

Inhibition of the calcium-activated chloride current in cardiac ventricular myocytes by N-( p-amylcinnamoyl)anthranilic acid (ACA)

Crosstalk between L-type Ca2+ channels and the sarcoplasmic reticulum: alterations during cardiac remodelling

Targeting calcium handling in arrhythmias

Intracellular na in animal models of hypertrophy and heart failure: contractile function and arrhythmogenesis

Window Ca2+ current and its modulation by Ca2+ release in hypertrophied cardiac myocytes from dogs with chronic atrioventricular block

Progress in the understanding of cardiac early afterdepolarizations and torsades de pointes : time to revise current concepts

Enhanced Ca(2+) release and Na/Ca exchange activity in hypertrophied canine ventricular myocytes: potential link between contractile adaptation and arrhythmogenesis

Impairment of trafficking by downregulation of an anchor protein: Novel insights into additional mechanisms responsible for heart failure

Early exercise training after myocardial infarction prevents contractile but not electrical remodelling or hypertrophy

T-type Ca2+ current as a trigger for Ca2+ release from the sarcoplasmic reticulum in guinea-pig ventricular myocytes

A SERCA2 pump with an increased Ca2+ affinity can lead to severe cardiac hypertrophy, stress intolerance and reduced life span

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