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Genetics and molecular mechanisms of frontotemporal lobar degeneration: an update and future avenues

Neurobiology of aging, 2019-06, Vol.78, p.98-110

2019

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Advances in protein-protein interaction network analysis for Parkinson's disease

Neurobiology of disease, 2021-07, Vol.155, p.105395-105395, Article 105395

2021

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Synthetic amyloid-β oligomers impair long-term memory independently of cellular prion protein

Proceedings of the National Academy of Sciences - PNAS, 2010-02, Vol.107 (5), p.2295-2300

2010

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Dysfunction of the autophagy/lysosomal degradation pathway is a shared feature of the genetic synucleinopathies

The FASEB journal, 2013-09, Vol.27 (9), p.3424-3429

2013

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Creep stability of the DART/Hera mission target 65803 Didymos: II. The role of cohesion

Icarus (New York, N.Y. 1962), 2021-07, Vol.362, p.114433, Article 114433

2021

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The LRRK2-macroautophagy axis and its relevance to Parkinson's disease

Biochemical Society transactions, 2017-02, Vol.45 (1), p.155-162

Manzoni, Claudia

2017

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Genetic and phenotypic characterization of complex hereditary spastic paraplegia

Brain (London, England : 1878), 2016-07, Vol.139 (Pt 7), p.1904-1918

2016

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Inhibition of LRRK2 kinase activity stimulates macroautophagy

Biochimica et biophysica acta, 2013-12, Vol.1833 (12), p.2900-2910

2013

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LRRK2 and human disease: a complicated question or a question of complexes?

Science signaling, 2012-01, Vol.5 (207), p.pe2

2012

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Preclinical modeling of chronic inhibition of the Parkinson's disease associated kinase LRRK2 reveals altered function of the endolysosomal system in vivo

Molecular neurodegeneration, 2021-03, Vol.16 (1), p.17-17, Article 17

2021

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mTOR independent regulation of macroautophagy by Leucine Rich Repeat Kinase 2 via Beclin-1

Scientific reports, 2016-10, Vol.6 (1), p.35106-35106, Article 35106

2016

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Cytosolic sequestration of spatacsin by Protein Kinase A and 14-3-3 proteins

Neurobiology of disease, 2022-11, Vol.174, p.105858-105858, Article 105858

2022

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Comparative Protein Interaction Network Analysis Identifies Shared and Distinct Functions for the Human ROCO Proteins

Proteomics (Weinheim), 2018-05, Vol.18 (10), p.e1700444-n/a

2018

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Stratification of candidate genes for Parkinson's disease using weighted protein-protein interaction network analysis

BMC genomics, 2018-06, Vol.19 (1), p.452-452, Article 452

2018

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Tissue specific LRRK2 interactomes reveal a distinct striatal functional unit

PLoS computational biology, 2023-01, Vol.19 (1), p.e1010847-e1010847

2023

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Seventy-Two-Hour LRRK2 Kinase Activity Inhibition Increases Lysosomal GBA Expression in H4, a Human Neuroglioma Cell Line

International journal of molecular sciences, 2022-06, Vol.23 (13), p.6935

2022

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Divergent α-synuclein solubility and aggregation properties in G2019S LRRK2 Parkinson's disease brains with Lewy Body pathology compared to idiopathic cases

Neurobiology of disease, 2013-10, Vol.58, p.183-190

2013

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Pathogenic Parkinson’s disease mutations across the functional domains of LRRK2 alter the autophagic/lysosomal response to starvation

Biochemical and biophysical research communications, 2013-11, Vol.441 (4), p.862-866

2013

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PINOT: an intuitive resource for integrating protein-protein interactions

Cell communication and signaling, 2020-06, Vol.18 (1), p.92-92, Article 92

2020

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Transcriptomics and weighted protein network analyses of the LRRK2 protein interactome reveal distinct molecular signatures for sporadic and LRRK2 Parkinson’s Disease

NPJ Parkinson's Disease, 2024-08, Vol.10 (1), p.144-12, Article 144

2024

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Computational analysis of the LRRK2 interactome

PeerJ (San Francisco, CA), 2015-02, Vol.3, p.e778-e778

2015

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Cellular processes associated with LRRK2 function and dysfunction

The FEBS journal, 2015-08, Vol.282 (15), p.2806-2826

2015

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Human mutations in SLITRK3 implicated in GABAergic synapse development in mice

Frontiers in molecular neuroscience, 2024-03, Vol.17, p.1222935-1222935

2024

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Mendelian and Sporadic FTD: Disease Risk and Avenues from Genetics to Disease Pathways Through In Silico Modelling

Advances in experimental medicine and biology, 2021-01, Vol.1281, p.283-296

2021

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The molecular assembly of amyloid aβ controls its neurotoxicity and binding to cellular proteins

PloS one, 2011-09, Vol.6 (9), p.e24909

2011

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Genetics and molecular mechanisms of frontotemporal lobar degeneration: an update and future avenues

Advances in protein-protein interaction network analysis for Parkinson's disease

Synthetic amyloid-β oligomers impair long-term memory independently of cellular prion protein

Dysfunction of the autophagy/lysosomal degradation pathway is a shared feature of the genetic synucleinopathies

Creep stability of the DART/Hera mission target 65803 Didymos: II. The role of cohesion

The LRRK2-macroautophagy axis and its relevance to Parkinson's disease

Genetic and phenotypic characterization of complex hereditary spastic paraplegia

Inhibition of LRRK2 kinase activity stimulates macroautophagy

LRRK2 and human disease: a complicated question or a question of complexes?

Preclinical modeling of chronic inhibition of the Parkinson's disease associated kinase LRRK2 reveals altered function of the endolysosomal system in vivo

mTOR independent regulation of macroautophagy by Leucine Rich Repeat Kinase 2 via Beclin-1

Cytosolic sequestration of spatacsin by Protein Kinase A and 14-3-3 proteins

Comparative Protein Interaction Network Analysis Identifies Shared and Distinct Functions for the Human ROCO Proteins

Stratification of candidate genes for Parkinson's disease using weighted protein-protein interaction network analysis

Tissue specific LRRK2 interactomes reveal a distinct striatal functional unit

Seventy-Two-Hour LRRK2 Kinase Activity Inhibition Increases Lysosomal GBA Expression in H4, a Human Neuroglioma Cell Line

Divergent α-synuclein solubility and aggregation properties in G2019S LRRK2 Parkinson's disease brains with Lewy Body pathology compared to idiopathic cases

Pathogenic Parkinson’s disease mutations across the functional domains of LRRK2 alter the autophagic/lysosomal response to starvation

PINOT: an intuitive resource for integrating protein-protein interactions

Transcriptomics and weighted protein network analyses of the LRRK2 protein interactome reveal distinct molecular signatures for sporadic and LRRK2 Parkinson’s Disease

Computational analysis of the LRRK2 interactome

Cellular processes associated with LRRK2 function and dysfunction

Human mutations in SLITRK3 implicated in GABAergic synapse development in mice

Mendelian and Sporadic FTD: Disease Risk and Avenues from Genetics to Disease Pathways Through In Silico Modelling

The molecular assembly of amyloid aβ controls its neurotoxicity and binding to cellular proteins

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