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Oropharyngeal Candidiasis: Fungal Invasion and Epithelial Cell Responses

PLoS pathogens, 2017-01, Vol.13 (1), p.e1006056-e1006056

2017

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Candida albicans Als3, a Multifunctional Adhesin and Invasin

Eukaryotic Cell, 2011-02, Vol.10 (2), p.168-173

2011

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Fungal Extracellular Vesicles Are Involved in Intraspecies Intracellular Communication

mBio, 2022-02, Vol.13 (1), p.e0327221-e0327221

2022

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Cryptococcus gattii VGIII isolates causing infections in HIV/AIDS patients in Southern California: identification of the local environmental source as arboreal

PLoS pathogens, 2014-08, Vol.10 (8), p.e1004285

2014

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Fungal invasion of normally non-phagocytic host cells

PLoS pathogens, 2006-12, Vol.2 (12), p.e129-e129

2006

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Control of β-glucan exposure by the endo-1,3-glucanase Eng1 in Candida albicans modulates virulence

PLoS pathogens, 2022-01, Vol.18 (1), p.e1010192-e1010192

2022

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Activation and alliance of regulatory pathways in C. albicans during mammalian infection

PLoS biology, 2015-02, Vol.13 (2), p.e1002076

2015

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Combination Polyene-Caspofungin Treatment of Rhino-Orbital-Cerebral Mucormycosis

Clinical infectious diseases, 2008-08, Vol.47 (3), p.364-371

2008

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the hyphal-associated adhesin and invasin Als3 of Candida albicans mediates iron acquisition from host ferritin

PLoS pathogens, 2008-11, Vol.4 (11), p.e1000217-e1000217

2008

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Candida albicans stimulates formation of a multi-receptor complex that mediates epithelial cell invasion during oropharyngeal infection

PLoS pathogens, 2023-08, Vol.19 (8), p.e1011579-e1011579

2023

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Roles of Candida albicans Mig1 and Mig2 in glucose repression, pathogenicity traits, and SNF1 essentiality

PLoS genetics, 2020-01, Vol.16 (1), p.e1008582-e1008582

2020

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Mouse model of oropharyngeal candidiasis

Nature protocols, 2012-04, Vol.7 (4), p.637-642

2012

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Activation of EphA2-EGFR signaling in oral epithelial cells by Candida albicans virulence factors

PLoS pathogens, 2021-01, Vol.17 (1), p.e1009221-e1009221

2021

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Host cell invasion and virulence mediated by Candida albicans Ssa1

PLoS pathogens, 2010-11, Vol.6 (11), p.e1001181-e1001181

2010

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Candidalysin Is Required for Neutrophil Recruitment and Virulence During Systemic Candida albicans Infection

The Journal of infectious diseases, 2019-11, Vol.220 (9), p.1477-1488

2019

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Serum bridging molecules drive candidal invasion of human but not mouse endothelial cells

PLoS pathogens, 2022-07, Vol.18 (7), p.e1010681-e1010681

2022

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Systemic Staphylococcus aureus infection mediated by Candida albicans hyphal invasion of mucosal tissue

Microbiology (Society for General Microbiology), 2015-01, Vol.161 (Pt 1), p.168-181

2015

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Selection of Candida albicans trisomy during oropharyngeal infection results in a commensal-like phenotype

PLoS genetics, 2019-05, Vol.15 (5), p.e1008137-e1008137

2019

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Aberrant type 1 immunity drives susceptibility to mucosal fungal infections

Science (American Association for the Advancement of Science), 2021-01, Vol.371 (6526)

2021

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Role of Arf GTPases in fungal morphogenesis and virulence

PLoS pathogens, 2017-02, Vol.13 (2), p.e1006205

2017

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Als3 is a Candida albicans invasin that binds to cadherins and induces endocytosis by host cells

PLoS biology, 2007-03, Vol.5 (3), p.e64-e64

2007

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Clinical Practice Guidelines for the Management Candidiasis: 2009 Update by the Infectious Diseases Society of America

Clinical infectious diseases, 2009-03, Vol.48 (5), p.503-535

2009

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Candida albicans Oropharyngeal Infection Is an Exception to Iron-Based Nutritional Immunity

mBio, 2023-04, Vol.14 (2), p.e0009523-e0009523

2023

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The Hyr1 protein from the fungus Candida albicans is a cross kingdom immunotherapeutic target for Acinetobacter bacterial infection

PLoS pathogens, 2018-05, Vol.14 (5), p.e1007056-e1007056

2018

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The pH-responsive PacC transcription factor of Aspergillus fumigatus governs epithelial entry and tissue invasion during pulmonary aspergillosis

PLoS pathogens, 2014-10, Vol.10 (10), p.e1004413

2014

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Oropharyngeal Candidiasis: Fungal Invasion and Epithelial Cell Responses

Candida albicans Als3, a Multifunctional Adhesin and Invasin

Fungal Extracellular Vesicles Are Involved in Intraspecies Intracellular Communication

Cryptococcus gattii VGIII isolates causing infections in HIV/AIDS patients in Southern California: identification of the local environmental source as arboreal

Fungal invasion of normally non-phagocytic host cells

Control of β-glucan exposure by the endo-1,3-glucanase Eng1 in Candida albicans modulates virulence

Activation and alliance of regulatory pathways in C. albicans during mammalian infection

Combination Polyene-Caspofungin Treatment of Rhino-Orbital-Cerebral Mucormycosis

the hyphal-associated adhesin and invasin Als3 of Candida albicans mediates iron acquisition from host ferritin

Candida albicans stimulates formation of a multi-receptor complex that mediates epithelial cell invasion during oropharyngeal infection

Roles of Candida albicans Mig1 and Mig2 in glucose repression, pathogenicity traits, and SNF1 essentiality

Mouse model of oropharyngeal candidiasis

Activation of EphA2-EGFR signaling in oral epithelial cells by Candida albicans virulence factors

Host cell invasion and virulence mediated by Candida albicans Ssa1

Candidalysin Is Required for Neutrophil Recruitment and Virulence During Systemic Candida albicans Infection

Serum bridging molecules drive candidal invasion of human but not mouse endothelial cells

Systemic Staphylococcus aureus infection mediated by Candida albicans hyphal invasion of mucosal tissue

Selection of Candida albicans trisomy during oropharyngeal infection results in a commensal-like phenotype

Aberrant type 1 immunity drives susceptibility to mucosal fungal infections

Role of Arf GTPases in fungal morphogenesis and virulence

Als3 is a Candida albicans invasin that binds to cadherins and induces endocytosis by host cells

Clinical Practice Guidelines for the Management Candidiasis: 2009 Update by the Infectious Diseases Society of America

Candida albicans Oropharyngeal Infection Is an Exception to Iron-Based Nutritional Immunity

The Hyr1 protein from the fungus Candida albicans is a cross kingdom immunotherapeutic target for Acinetobacter bacterial infection

The pH-responsive PacC transcription factor of Aspergillus fumigatus governs epithelial entry and tissue invasion during pulmonary aspergillosis

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