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Loss of excitatory amino acid transporter restraint following chronic intermittent hypoxia contributes to synaptic alterations in nucleus tractus solitarii

Journal of neurophysiology, 2020-06, Vol.123 (6), p.2122-2135

2020

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Corrigendum to “Exaggerated potassium current reduction by oxytocin in visceral sensory neurons following chronic intermittent hypoxia” [Auton Neurosci. 229 2020 Sep 28 102735]

Autonomic neuroscience, 2021-01, Vol.230, p.102758-102758, Article 102758

2021

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Correction: Hypobaric Intermittent Hypoxia Attenuates Hypoxia-induced Depressor Response

PloS one, 2013-04, Vol.8 (4)

2013

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H 2 O 2 augments cytosolic calcium in nucleus tractus solitarii neurons via multiple voltage-gated calcium channels

American Journal of Physiology: Cell Physiology, 2017-05, Vol.312 (5), p.C651-C662

2017

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N-Methyl-D-aspartate receptor (NMDAR) activity in the nucleus tractus solitarii (nTS) is required for the full expression of phrenic long-term facilitation after acute intermittent hypoxia

Physiology (Bethesda, Md.), 2024-05, Vol.39 (S1)

2024

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Orexin 1 Receptors in The Paraventricular Nucleus of The Hypothalamus (PVN) Facilitate The Peripheral Chemoreflex

Physiology (Bethesda, Md.), 2024-05, Vol.39 (S1)

2024

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Chronic intermittent hypoxia (CIH) enhances nTS sensitivity to oxytocin (OT) and corticotropin-releasing hormone (CRH) via effects at the nTS soma and on presynaptic release

Physiology (Bethesda, Md.), 2024-05, Vol.39 (S1)

2024

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Chronic Vagotomy Blunts Cardiorespiratory Reflex Responses Through Pre- and Postsynaptic Effects in the Brainstem Nucleus Tractus Solitarii (nTS)

Physiology (Bethesda, Md.), 2024-05, Vol.39 (S1)

2024

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Sensory afferent and hypoxia-mediated activation of nucleus tractus solitarius neurons that project to the rostral ventrolateral medulla

Neuroscience, 2010-05, Vol.167 (2), p.510-527

2010

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Excitatory amino acid transporters (EAATs) restrain nucleus tractus solitarii (nTS) activity induced by ionotropic and metabotropic glutamate receptor activation

Revista cubana de investigaciones biomédicas, 2020-01, Vol.38 (5)

2020

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Chronic intermittent hypoxia affects integration of sensory input by neurons in the nucleus tractus solitarii: Central Cardiorespiratory Regulation: Physiology and Pathology

Respiratory physiology & neurobiology, 2010, Vol.174 (1-2), p.29-36

KLINE, David D

2010

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Depressed GABA and glutamate synaptic signaling by 5-HT 1A receptors in the nucleus tractus solitarii and their role in cardiorespiratory function

Journal of neurophysiology, 2014-06, Vol.111 (12), p.2493-2504

2014

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A Projection from the Hypothalamic Paraventricular Nucleus to the Nucleus Tractus Solitarii is Essential for Cardiorespiratory Responses to Hypoxia

Revista cubana de investigaciones biomédicas, 2020-01, Vol.38 (5)

2020

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Plasticity in glutamatergic NTS neurotransmission: Neurochemistry of Respiratory Control

Respiratory physiology & neurobiology, 2008, Vol.164 (1-2), p.105-111

KLINE, David D

2008

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Hypoxia activates nucleus tractus solitarii neurons projecting to the paraventricular nucleus of the hypothalamus

American journal of physiology. Regulatory, integrative and comparative physiology, 2012, Vol.71 (5)

2012

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Chapter 26 - Pharmacology of the neurotransmission of the baro- and chemoreflex in the Nucleus Tractus Solitarius

Primer on the Autonomic Nervous System, 2023, p.147-153

Fourth Edition, 2023

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Role of nitric oxide in short-term potentiation and long-term facilitation: involvement of NO in breathing stability

Advances in experimental medicine and biology, 2001, Vol.499, p.215

2001

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Defective carotid body function and impaired ventilatory responses to chronic hypoxia in mice partially deficient for hypoxia-indusible factor 1alpha

Proceedings of the National Academy of Sciences - PNAS, 2002-01, Vol.99 (2), p.821

2002

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The role of nitric oxide in the modulation of breathing during hypoxia; studies in transgenic mice

Kline, David D

2000

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Operative neurophysiology of the brachial plexus intraoperative electrodiagnostic studies

Practical Management of Pediatric and Adult Brachial Plexus Palsies, 2012, p.212-219

2012

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Outcomes of treatment for adult brachial plexus injuries

Practical Management of Pediatric and Adult Brachial Plexus Palsies, 2012, p.344-365

2012

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Thoracic Outlet Syndrome

Atlas of Peripheral Nerve Surgery, 2013, p.91-94

Second Edition, 2013

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External and Internal Neurolysis

Atlas of Peripheral Nerve Surgery, 2013, p.241-245

Second Edition, 2013

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Nerve Transfers

Atlas of Peripheral Nerve Surgery, 2013, p.259-265

Second Edition, 2013

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Axillary Nerve

Atlas of Peripheral Nerve Surgery, 2013, p.107-114

Second Edition, 2013

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Loss of excitatory amino acid transporter restraint following chronic intermittent hypoxia contributes to synaptic alterations in nucleus tractus solitarii

Corrigendum to “Exaggerated potassium current reduction by oxytocin in visceral sensory neurons following chronic intermittent hypoxia” [Auton Neurosci. 229 2020 Sep 28 102735]

Correction: Hypobaric Intermittent Hypoxia Attenuates Hypoxia-induced Depressor Response

H 2 O 2 augments cytosolic calcium in nucleus tractus solitarii neurons via multiple voltage-gated calcium channels

N-Methyl-D-aspartate receptor (NMDAR) activity in the nucleus tractus solitarii (nTS) is required for the full expression of phrenic long-term facilitation after acute intermittent hypoxia

Orexin 1 Receptors in The Paraventricular Nucleus of The Hypothalamus (PVN) Facilitate The Peripheral Chemoreflex

Chronic intermittent hypoxia (CIH) enhances nTS sensitivity to oxytocin (OT) and corticotropin-releasing hormone (CRH) via effects at the nTS soma and on presynaptic release

Chronic Vagotomy Blunts Cardiorespiratory Reflex Responses Through Pre- and Postsynaptic Effects in the Brainstem Nucleus Tractus Solitarii (nTS)

Sensory afferent and hypoxia-mediated activation of nucleus tractus solitarius neurons that project to the rostral ventrolateral medulla

Excitatory amino acid transporters (EAATs) restrain nucleus tractus solitarii (nTS) activity induced by ionotropic and metabotropic glutamate receptor activation

Chronic intermittent hypoxia affects integration of sensory input by neurons in the nucleus tractus solitarii: Central Cardiorespiratory Regulation: Physiology and Pathology

Depressed GABA and glutamate synaptic signaling by 5-HT 1A receptors in the nucleus tractus solitarii and their role in cardiorespiratory function

A Projection from the Hypothalamic Paraventricular Nucleus to the Nucleus Tractus Solitarii is Essential for Cardiorespiratory Responses to Hypoxia

Plasticity in glutamatergic NTS neurotransmission: Neurochemistry of Respiratory Control

Hypoxia activates nucleus tractus solitarii neurons projecting to the paraventricular nucleus of the hypothalamus

Chapter 26 - Pharmacology of the neurotransmission of the baro- and chemoreflex in the Nucleus Tractus Solitarius

Role of nitric oxide in short-term potentiation and long-term facilitation: involvement of NO in breathing stability

Defective carotid body function and impaired ventilatory responses to chronic hypoxia in mice partially deficient for hypoxia-indusible factor 1alpha

The role of nitric oxide in the modulation of breathing during hypoxia; studies in transgenic mice

Operative neurophysiology of the brachial plexus intraoperative electrodiagnostic studies

Outcomes of treatment for adult brachial plexus injuries

Thoracic Outlet Syndrome

External and Internal Neurolysis

Nerve Transfers

Axillary Nerve

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