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MCT1 Inhibitor AZD3965 Increases Mitochondrial Metabolism, Facilitating Combination Therapy and Noninvasive Magnetic Resonance Spectroscopy

Cancer research (Chicago, Ill.), 2017-11, Vol.77 (21), p.5913-5924

2017

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Monocarboxylate transporter 1 blockade with AZD3965 inhibits lipid biosynthesis and increases tumour immune cell infiltration

British journal of cancer, 2020-03, Vol.122 (6), p.895-903

2020

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Increased inflammatory lipid metabolism and anaplerotic mitochondrial activation follow acquired resistance to vemurafenib in BRAF-mutant melanoma cells

British journal of cancer, 2020-01, Vol.122 (1), p.72-81

2020

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Detecting human melanoma cell re-differentiation following BRAF or heat shock protein 90 inhibition using photoacoustic and magnetic resonance imaging

Scientific reports, 2017-08, Vol.7 (1), p.8215-9, Article 8215

2017

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Association of cystic fibrosis genetic modifiers with congenital bilateral absence of the vas deferens

Fertility and sterility, 2010-11, Vol.94 (6), p.2122-2127

2010

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Abstract 444: Monocarboxylate transporter 1 inhibition with AZD3965 increases cancer cell dependence on bioenergetic metabolism predicating combination therapy with mitochondrial inhibitors

Cancer research (Chicago, Ill.), 2017-07, Vol.77 (13_Supplement), p.444-444

2017

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Best practice guidelines for molecular genetic diagnosis of cystic fibrosis and CFTR-related disorders - updated European recommendations

European journal of human genetics : EJHG, 2009-01, Vol.17 (1), p.51-65

2009

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Best practice guidelines for molecular genetic diagnosis of cystic fibrosis and CFTR-related disorders – updated European recommendations

European journal of human genetics : EJHG, 2008-08, Vol.17 (1), p.51-65

2008

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Assessing the residual CFTR gene expression in human nasal epithelium cells bearing CFTR splicing mutations causing cystic fibrosis

European journal of human genetics : EJHG, 2014-06, Vol.22 (6), p.784-791

2014

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GUSB and ATP2B4 are suitable reference genes for CFTR gene expression data normalization in nasal epithelium cells

Journal of cystic fibrosis, 2012-09, Vol.11 (5), p.398-404

2012

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Abstract 4108: Longitudinal diffusion-weighted MRI assessment of NRAS mutant melanoma response to dual RAF-MEK inhibition reveals differences associated with collagen deposition

Cancer research (Chicago, Ill.), 2018-07, Vol.78 (13_Supplement), p.4108-4108

2018

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Optimizing strategies for CFTR molecular testing

Journal of cystic fibrosis, 2011-03, Vol.10 (2), p.143-144

2011

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MCT1 inhibitor AZD3965 increases mitochondrial metabolism, facilitating combination therapy and non-invasive magnetic resonance spectroscopy

Cancer research (Chicago, Ill.), 2017-09, Vol.77 (21), p.5913-5924

2017

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Can a Place of Origin of the Main Cystic Fibrosis Mutations Be Identified?

American journal of human genetics, 2002-01, Vol.70 (1), p.257-264

2002

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Common mutations in cuban cystic fibrosis patients

Journal of cystic fibrosis, 2009-01, Vol.8 (1), p.47-49

2009

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Spectrum of mutations in CFTR in Finland: 18 years follow-up study and identification of two novel mutations

Journal of cystic fibrosis, 2005-12, Vol.4 (4), p.233-237

2005

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CA/GT microsatellite alleles within the cystic fibrosis transmembrane conductance regulator (CFTR) gene are not generated by unequal crossingover

Genomics (San Diego, Calif.), 1991-07, Vol.10 (3), p.692-698

1991

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CFTR and asthma in the French EGEA study

European journal of human genetics : EJHG, 2001-01, Vol.9 (1), p.67-69

2001

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ATB0/SLC1A5 gene. Fine localisation and exclusion of association with the intestinal phenotype of cystic fibrosis

European journal of human genetics : EJHG, 2001-12, Vol.9 (11), p.860-866

2001

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ATB super(0)/SLC1A5 gene. Fine localisation and exclusion of association with the intestinal phenotype of cystic fibrosis

European journal of human genetics : EJHG, 2001-11, Vol.9 (11), p.860-866

2001

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Reproductive pathology. Heterogeneity for mutations in the CFTR gene and clinical correlations in patients with congenital absence of the vas deferens

Human reproduction (Oxford), 2000-07, Vol.15 (7), p.1476-1476

2000

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Independent origins of cystic fibrosis mutations R334W, R347P, R1162X, and 3849 + 10kbC-->T provide evidence of mutation recurrence in the CFTR gene

American journal of human genetics, 1994-11, Vol.55 (5), p.890-898

1994

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La política de lectorats de català en el període 1988-2002

Coneixement i societat, 2003 (2)

Casals i Rubio, Teresa

2003

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Allele Frequencies in a Worldwide Survey of a CA Repeat in the First Intron of the CFTR Gene

Human heredity, 1999-01, Vol.49 (1), p.15-20

1999

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CAGT Microsatellite alleles within the cystic fibrosis transmembrane conductance regulator (CFTR) gene are not generated by unequal crossingover

Genomics (San Diego, Calif.), 1991-07, Vol.10 (3), p.692-698

1991

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MCT1 Inhibitor AZD3965 Increases Mitochondrial Metabolism, Facilitating Combination Therapy and Noninvasive Magnetic Resonance Spectroscopy

Monocarboxylate transporter 1 blockade with AZD3965 inhibits lipid biosynthesis and increases tumour immune cell infiltration

Increased inflammatory lipid metabolism and anaplerotic mitochondrial activation follow acquired resistance to vemurafenib in BRAF-mutant melanoma cells

Detecting human melanoma cell re-differentiation following BRAF or heat shock protein 90 inhibition using photoacoustic and magnetic resonance imaging

Association of cystic fibrosis genetic modifiers with congenital bilateral absence of the vas deferens

Abstract 444: Monocarboxylate transporter 1 inhibition with AZD3965 increases cancer cell dependence on bioenergetic metabolism predicating combination therapy with mitochondrial inhibitors

Best practice guidelines for molecular genetic diagnosis of cystic fibrosis and CFTR-related disorders - updated European recommendations

Best practice guidelines for molecular genetic diagnosis of cystic fibrosis and CFTR-related disorders – updated European recommendations

Assessing the residual CFTR gene expression in human nasal epithelium cells bearing CFTR splicing mutations causing cystic fibrosis

GUSB and ATP2B4 are suitable reference genes for CFTR gene expression data normalization in nasal epithelium cells

Abstract 4108: Longitudinal diffusion-weighted MRI assessment of NRAS mutant melanoma response to dual RAF-MEK inhibition reveals differences associated with collagen deposition

Optimizing strategies for CFTR molecular testing

MCT1 inhibitor AZD3965 increases mitochondrial metabolism, facilitating combination therapy and non-invasive magnetic resonance spectroscopy

Can a Place of Origin of the Main Cystic Fibrosis Mutations Be Identified?

Common mutations in cuban cystic fibrosis patients

Spectrum of mutations in CFTR in Finland: 18 years follow-up study and identification of two novel mutations

CA/GT microsatellite alleles within the cystic fibrosis transmembrane conductance regulator (CFTR) gene are not generated by unequal crossingover

CFTR and asthma in the French EGEA study

ATB0/SLC1A5 gene. Fine localisation and exclusion of association with the intestinal phenotype of cystic fibrosis

ATB super(0)/SLC1A5 gene. Fine localisation and exclusion of association with the intestinal phenotype of cystic fibrosis

Reproductive pathology. Heterogeneity for mutations in the CFTR gene and clinical correlations in patients with congenital absence of the vas deferens

Independent origins of cystic fibrosis mutations R334W, R347P, R1162X, and 3849 + 10kbC-->T provide evidence of mutation recurrence in the CFTR gene

La política de lectorats de català en el període 1988-2002

Allele Frequencies in a Worldwide Survey of a CA Repeat in the First Intron of the CFTR Gene

CAGT Microsatellite alleles within the cystic fibrosis transmembrane conductance regulator (CFTR) gene are not generated by unequal crossingover

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