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Transcriptional Control by the SMADs

Cold Spring Harbor perspectives in biology, 2016-10, Vol.8 (10), p.a022079

Hill, Caroline S

2016

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A new class of small molecule inhibitor of BMP signaling

PloS one, 2013-04, Vol.8 (4), p.e62721

2013

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Spatial and temporal control of NODAL signaling

Current opinion in cell biology, 2018-04, Vol.51, p.50-57

Hill, Caroline S

2018

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Beyond TGFβ: roles of other TGFβ superfamily members in cancer

Nature reviews. Cancer, 2013-05, Vol.13 (5), p.328-341

2013

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TGF-β signaling to chromatin: How Smads regulate transcription during self-renewal and differentiation

Seminars in cell & developmental biology, 2014-08, Vol.32, p.107-118

2014

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Mesenchymal Cancer Cell-Stroma Crosstalk Promotes Niche Activation, Epithelial Reversion, and Metastatic Colonization

Cell reports (Cambridge), 2015-12, Vol.13 (11), p.2456-2469

2015

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TGF-β Superfamily Signaling in Embryonic Development and Homeostasis

Developmental cell, 2009-03, Vol.16 (3), p.329-343

2009

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How the Smads regulate transcription

The international journal of biochemistry & cell biology, 2008, Vol.40 (3), p.383-408

2008

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Recruitment of TIF1γ to Chromatin via Its PHD Finger-Bromodomain Activates Its Ubiquitin Ligase and Transcriptional Repressor Activities

Molecular cell, 2011-07, Vol.43 (1), p.85-96

2011

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Context-dependent TGFβ family signalling in cell fate regulation

Nature reviews. Molecular cell biology, 2023-12, Vol.24 (12), p.876-894

2023

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Older adults' lived experiences of physical rehabilitation for acquired brain injury and their perceptions of well-being: A qualitative phenomenological study

Journal of clinical nursing, 2024-03, Vol.33 (3), p.1134-1149

2024

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A systematic review of the qualitative literature on older individuals’ experiences of care and well‐being during physical rehabilitation for acquired brain injury

Journal of advanced nursing, 2022-02, Vol.78 (2), p.377-394

2022

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Transforming Growth Factor β-Induced Smad1/5 Phosphorylation in Epithelial Cells Is Mediated by Novel Receptor Complexes and Is Essential for Anchorage-Independent Growth

Molecular and Cellular Biology, 2008-11, Vol.28 (22), p.6889-6902

2008

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Alterations in components of the TGF-β superfamily signaling pathways in human cancer

Cytokine & growth factor reviews, 2006-02, Vol.17 (1), p.41-58

2006

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TGF-β uses a novel mode of receptor activation to phosphorylate SMAD1/5 and induce epithelial-to-mesenchymal transition

eLife, 2018-01, Vol.7

2018

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New insights into TGF-β–Smad signalling

Trends in biochemical sciences (Amsterdam. Regular ed.), 2004-05, Vol.29 (5), p.265-273

2004

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TGF-β family ligands exhibit distinct signalling dynamics that are driven by receptor localisation

Journal of cell science, 2019-07, Vol.132 (14)

2019

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Spatial regulation of BMP activity

FEBS letters, 2012-07, Vol.586 (14), p.1929-1941

2012

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A BMP regulatory network controls ectodermal cell fate decisions at the neural plate border

Development (Cambridge), 2013-11, Vol.140 (21), p.4435-4444

2013

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EpCAM ectodomain EpEX is a ligand of EGFR that counteracts EGF-mediated epithelial-mesenchymal transition through modulation of phospho-ERK1/2 in head and neck cancers

PLoS biology, 2018-09, Vol.16 (9), p.e2006624

2018

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Kinetic Analysis of Smad Nucleocytoplasmic Shuttling Reveals a Mechanism for Transforming Growth Factor β-Dependent Nuclear Accumulation of Smads

Molecular and Cellular Biology, 2005-11, Vol.25 (22), p.9845-9858

2005

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Mathematical modeling identifies Smad nucleocytoplasmic shuttling as a dynamic signal-interpreting system

Proceedings of the National Academy of Sciences - PNAS, 2008-05, Vol.105 (18), p.6608-6613

2008

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How do people live life successfully with Parkinson's disease?

Journal of clinical nursing, 2015-08, Vol.24 (15-16), p.2314-2322

2015

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Transforming Growth Factor β Inhibits Bone Morphogenetic Protein-Induced Transcription through Novel Phosphorylated Smad1/5-Smad3 Complexes

Molecular and Cellular Biology, 2012-07, Vol.32 (14), p.2904-2916

2012

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A network of transcription factors governs the dynamics of NODAL/Activin transcriptional responses

Journal of cell science, 2022-04, Vol.135 (8)

2022

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Transcriptional Control by the SMADs

A new class of small molecule inhibitor of BMP signaling

Spatial and temporal control of NODAL signaling

Beyond TGFβ: roles of other TGFβ superfamily members in cancer

TGF-β signaling to chromatin: How Smads regulate transcription during self-renewal and differentiation

Mesenchymal Cancer Cell-Stroma Crosstalk Promotes Niche Activation, Epithelial Reversion, and Metastatic Colonization

TGF-β Superfamily Signaling in Embryonic Development and Homeostasis

How the Smads regulate transcription

Recruitment of TIF1γ to Chromatin via Its PHD Finger-Bromodomain Activates Its Ubiquitin Ligase and Transcriptional Repressor Activities

Context-dependent TGFβ family signalling in cell fate regulation

Older adults' lived experiences of physical rehabilitation for acquired brain injury and their perceptions of well-being: A qualitative phenomenological study

A systematic review of the qualitative literature on older individuals’ experiences of care and well‐being during physical rehabilitation for acquired brain injury

Transforming Growth Factor β-Induced Smad1/5 Phosphorylation in Epithelial Cells Is Mediated by Novel Receptor Complexes and Is Essential for Anchorage-Independent Growth

Alterations in components of the TGF-β superfamily signaling pathways in human cancer

TGF-β uses a novel mode of receptor activation to phosphorylate SMAD1/5 and induce epithelial-to-mesenchymal transition

New insights into TGF-β–Smad signalling

TGF-β family ligands exhibit distinct signalling dynamics that are driven by receptor localisation

Spatial regulation of BMP activity

A BMP regulatory network controls ectodermal cell fate decisions at the neural plate border

EpCAM ectodomain EpEX is a ligand of EGFR that counteracts EGF-mediated epithelial-mesenchymal transition through modulation of phospho-ERK1/2 in head and neck cancers

Kinetic Analysis of Smad Nucleocytoplasmic Shuttling Reveals a Mechanism for Transforming Growth Factor β-Dependent Nuclear Accumulation of Smads

Mathematical modeling identifies Smad nucleocytoplasmic shuttling as a dynamic signal-interpreting system

How do people live life successfully with Parkinson's disease?

Transforming Growth Factor β Inhibits Bone Morphogenetic Protein-Induced Transcription through Novel Phosphorylated Smad1/5-Smad3 Complexes

A network of transcription factors governs the dynamics of NODAL/Activin transcriptional responses

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