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RGS4 Regulates Parasympathetic Signaling and Heart Rate Control in the Sinoatrial Node

Circulation research, 2008-08, Vol.103 (5), p.527-535

2008

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Regulation of Proliferation and Membrane Potential by Chloride Currents in Rat Pulmonary Artery Smooth Muscle Cells

Hypertension (Dallas, Tex. 1979), 2009-08, Vol.54 (2), p.286-293

2009

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Glucagon-Like Peptide (GLP)-1(9-36)Amide-Mediated Cytoprotection Is Blocked by Exendin(9-39) Yet Does Not Require the Known GLP-1 Receptor

Endocrinology (Philadelphia), 2010-04, Vol.151 (4), p.1520-1531

2010

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Selective Inhibition of Inward Rectifier K+ Channels (Kir2.1 or Kir2.2) Abolishes Protection by Ischemic Preconditioning in Rabbit Ventricular Cardiomyocytes

Circulation research, 2004-08, Vol.95 (3), p.325-332

2004

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Iroquois Homeodomain Transcription Factors in Heart Development and Function

Circulation research, 2012-05, Vol.110 (11), p.1513-1524

2012

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Phosphodiesterase 4D Regulates Baseline Sarcoplasmic Reticulum Ca2+ Release and Cardiac Contractility, Independently of L-Type Ca2+ Current

Circulation research, 2011-10, Vol.109 (9), p.1024-1030

2011

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Nuclear Factor κB Downregulates the Transient Outward Potassium Current Ito,f Through Control of KChIP2 Expression

Circulation research, 2011-03, Vol.108 (5), p.537-543

2011

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Loss of PTEN attenuates the development of pathological hypertrophy and heart failure in response to biomechanical stress

Cardiovascular research, 2008-06, Vol.78 (3), p.505-514

2008

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Tumor Necrosis Factor-α Enhances Microvascular Tone and Reduces Blood Flow in the Cochlea via Enhanced Sphingosine-1-Phosphate Signaling

Stroke (1970), 2010-11, Vol.41 (11), p.2618-2624

2010

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Combination of Tumor Necrosis Factor-α Ablation and Matrix Metalloproteinase Inhibition Prevents Heart Failure After Pressure Overload in Tissue Inhibitor of Metalloproteinase-3 Knock-Out Mice

Circulation research, 2005-08, Vol.97 (4), p.380-390

2005

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Phosphatidylinositol 3-Kinase γ Is a Critical Mediator of Myocardial Ischemic and Adenosine-Mediated Preconditioning

Circulation research, 2008-09, Vol.103 (6), p.643-653

2008

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Loss of PI3Kγ Enhances cAMP-Dependent MMP Remodeling of the Myocardial N-Cadherin Adhesion Complexes and Extracellular Matrix in Response to Early Biomechanical Stress

Circulation research, 2010-11, Vol.107 (10), p.1275-1289

2010

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PI3Kγ Is Required for PDE4, not PDE3, Activity in Subcellular Microdomains Containing the Sarcoplasmic Reticular Calcium ATPase in Cardiomyocytes

Circulation research, 2007-08, Vol.101 (4), p.400-408

2007

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Lack of Microsomal Prostaglandin E2 Synthase-1 in Bone Marrow―Derived Myeloid Cells Impairs Left Ventricular Function and Increases Mortality After Acute Myocardial Infarction

Circulation (New York, N.Y.), 2012-06, Vol.125 (23), p.2904-2913

2012

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The ShcA Phosphotyrosine Docking Protein Uses Distinct Mechanisms to Regulate Myocyte and Global Heart Function

Circulation research, 2011-01, Vol.108 (2), p.184-193

2011

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Phosphorylation-Independent Modulation of L-Type Calcium Channels by Magnesium-Nucleotide Complexes

Science (American Association for the Advancement of Science), 1992-07, Vol.257 (5067), p.245-248

1992

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Molecular components of transient outward potassium current in cultured neonatal rat ventricular myocytes

Journal of molecular medicine (Berlin, Germany), 2002-06, Vol.80 (6), p.351-358

2002

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Altered Heart Rate and Sinoatrial Node Function in Mice Lacking the cAMP Regulator Phosphoinositide 3-Kinase-γ

Circulation research, 2007-12, Vol.101 (12), p.1274-1282

2007

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Calcium-Sensitive Inactivation in the Gating of Single Calcium Channels

Science (American Association for the Advancement of Science), 1990-12, Vol.250 (4988), p.1735-1738

1990

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Cardiac sarcoplasmic reticulum calcium release and load are enhanced by subcellular cAMP elevations in PI3Kγ-deficient mice

Circulation research, 2005-05, Vol.96 (10), p.1079-1086

2005

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Molecular Localization of an Ion-Binding Site within the Pore of Mammalian Sodium Channels

Science (American Association for the Advancement of Science), 1992-07, Vol.257 (5067), p.248-251

1992

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Background Potassium Current Active During the Plateau of the Action Potential in Guinea Pig Ventricular Myocytes

Circulation research, 1993-04, Vol.72 (4), p.890-900

1993

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Modulation of Ca2+ Release in Cardiac Myocytes by Changes in Repolarization Rate: Role of Phase-1 Action Potential Repolarization in Excitation-Contraction Coupling

Circulation research, 2002-02, Vol.90 (2), p.165-173

2002

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Insulin-Like Growth Factor-1 and PTEN Deletion Enhance Cardiac L-Type Ca2+ Currents via Increased PI3Kα/PKB Signaling

Circulation research, 2006-06, Vol.98 (11), p.1390-1397

2006

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α-Crystallin B prevents apoptosis after H2O2 exposure in mouse neonatal cardiomyocytes

American journal of physiology. Heart and circulatory physiology, 2012, Vol.72 (4)

2012

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ThemaFundamental And Applied Biological Sciences. Psychology

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RGS4 Regulates Parasympathetic Signaling and Heart Rate Control in the Sinoatrial Node

Regulation of Proliferation and Membrane Potential by Chloride Currents in Rat Pulmonary Artery Smooth Muscle Cells

Glucagon-Like Peptide (GLP)-1(9-36)Amide-Mediated Cytoprotection Is Blocked by Exendin(9-39) Yet Does Not Require the Known GLP-1 Receptor

Selective Inhibition of Inward Rectifier K+ Channels (Kir2.1 or Kir2.2) Abolishes Protection by Ischemic Preconditioning in Rabbit Ventricular Cardiomyocytes

Iroquois Homeodomain Transcription Factors in Heart Development and Function

Phosphodiesterase 4D Regulates Baseline Sarcoplasmic Reticulum Ca2+ Release and Cardiac Contractility, Independently of L-Type Ca2+ Current

Nuclear Factor κB Downregulates the Transient Outward Potassium Current Ito,f Through Control of KChIP2 Expression

Loss of PTEN attenuates the development of pathological hypertrophy and heart failure in response to biomechanical stress

Tumor Necrosis Factor-α Enhances Microvascular Tone and Reduces Blood Flow in the Cochlea via Enhanced Sphingosine-1-Phosphate Signaling

Combination of Tumor Necrosis Factor-α Ablation and Matrix Metalloproteinase Inhibition Prevents Heart Failure After Pressure Overload in Tissue Inhibitor of Metalloproteinase-3 Knock-Out Mice

Phosphatidylinositol 3-Kinase γ Is a Critical Mediator of Myocardial Ischemic and Adenosine-Mediated Preconditioning

Loss of PI3Kγ Enhances cAMP-Dependent MMP Remodeling of the Myocardial N-Cadherin Adhesion Complexes and Extracellular Matrix in Response to Early Biomechanical Stress

PI3Kγ Is Required for PDE4, not PDE3, Activity in Subcellular Microdomains Containing the Sarcoplasmic Reticular Calcium ATPase in Cardiomyocytes

Lack of Microsomal Prostaglandin E2 Synthase-1 in Bone Marrow―Derived Myeloid Cells Impairs Left Ventricular Function and Increases Mortality After Acute Myocardial Infarction

The ShcA Phosphotyrosine Docking Protein Uses Distinct Mechanisms to Regulate Myocyte and Global Heart Function

Phosphorylation-Independent Modulation of L-Type Calcium Channels by Magnesium-Nucleotide Complexes

Molecular components of transient outward potassium current in cultured neonatal rat ventricular myocytes

Altered Heart Rate and Sinoatrial Node Function in Mice Lacking the cAMP Regulator Phosphoinositide 3-Kinase-γ

Calcium-Sensitive Inactivation in the Gating of Single Calcium Channels

Cardiac sarcoplasmic reticulum calcium release and load are enhanced by subcellular cAMP elevations in PI3Kγ-deficient mice

Molecular Localization of an Ion-Binding Site within the Pore of Mammalian Sodium Channels

Background Potassium Current Active During the Plateau of the Action Potential in Guinea Pig Ventricular Myocytes

Modulation of Ca2+ Release in Cardiac Myocytes by Changes in Repolarization Rate: Role of Phase-1 Action Potential Repolarization in Excitation-Contraction Coupling

Insulin-Like Growth Factor-1 and PTEN Deletion Enhance Cardiac L-Type Ca2+ Currents via Increased PI3Kα/PKB Signaling

α-Crystallin B prevents apoptosis after H2O2 exposure in mouse neonatal cardiomyocytes

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