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Proteolytic Processing of Interleukin-1 Family Cytokines: Variations on a Common Theme

Immunity (Cambridge, Mass.), 2015-06, Vol.42 (6), p.991-1004

2015

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Cell death and inflammation: the case for IL‐1 family cytokines as the canonical DAMPs of the immune system

The FEBS journal, 2016-07, Vol.283 (14), p.2599-2615

Martin, Seamus J.

2016

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Oncogenic Ras-Induced Expression of Noxa and Beclin-1 Promotes Autophagic Cell Death and Limits Clonogenic Survival

Molecular cell, 2011-04, Vol.42 (1), p.23-35

2011

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Caspase-8 Acts in a Non-enzymatic Role as a Scaffold for Assembly of a Pro-inflammatory “FADDosome” Complex upon TRAIL Stimulation

Molecular cell, 2017-02, Vol.65 (4), p.715-729.e5

2017

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Bcl-2 Family Proteins Participate in Mitochondrial Quality Control by Regulating Parkin/PINK1-Dependent Mitophagy

Molecular cell, 2014-08, Vol.55 (3), p.451-466

2014

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Neutrophil-Derived Proteases Escalate Inflammation through Activation of IL-36 Family Cytokines

Cell reports (Cambridge), 2016-02, Vol.14 (4), p.708-722

2016

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Parkin Sensitizes toward Apoptosis Induced by Mitochondrial Depolarization through Promoting Degradation of Mcl-1

Cell reports (Cambridge), 2014-11, Vol.9 (4), p.1538-1553

2014

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Fas/CD95-Induced Chemokines Can Serve as “Find-Me” Signals for Apoptotic Cells

Molecular cell, 2013-03, Vol.49 (6), p.1034-1048

2013

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A Perspective on Mammalian Caspases as Positive and Negative Regulators of Inflammation

Molecular cell, 2012-05, Vol.46 (4), p.387-397

2012

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Diverse Activators of the NLRP3 Inflammasome Promote IL-1β Secretion by Triggering Necrosis

Cell reports (Cambridge), 2015-06, Vol.11 (10), p.1535-1548

2015

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Inflammatory outcomes of apoptosis, necrosis and necroptosis

Biological chemistry, 2014-10, Vol.395 (10), p.1163-1171

2014

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Professor Dame Janet Thornton to receive ‘The FEBS Journal Open Science Award’

The FEBS journal, 2022-08, Vol.289 (16), p.4678-4679

2022

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Emerging Role for Members of the Bcl-2 Family in Mitochondrial Morphogenesis

Molecular cell, 2009-11, Vol.36 (3), p.355-363

2009

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Neutrophil extracellular traps can serve as platforms for processing and activation of IL‐1 family cytokines

The FEBS journal, 2017-06, Vol.284 (11), p.1712-1725

2017

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Autophagy in malignant transformation and cancer progression

The EMBO journal, 2015-04, Vol.34 (7), p.856-880

2015

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Mitochondrial fission/fusion dynamics and apoptosis

Mitochondrion, 2010-11, Vol.10 (6), p.640-648

2010

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Fas and TRAIL ‘death receptors’ as initiators of inflammation: Implications for cancer

Seminars in cell & developmental biology, 2015-03, Vol.39, p.26-34

2015

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Cytotoxic and non-cytotoxic roles of the CTL/NK protease granzyme B

Immunological reviews, 2010-05, Vol.235 (1), p.105-116

2010

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Extracellular Neutrophil Proteases Are Efficient Regulators of IL-1, IL-33, and IL-36 Cytokine Activity but Poor Effectors of Microbial Killing

Cell reports (Cambridge), 2018-03, Vol.22 (11), p.2937-2950

2018

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Granzyme B-Dependent Proteolysis Acts as a Switch to Enhance the Proinflammatory Activity of IL-1α

Molecular cell, 2011-10, Vol.44 (2), p.265-278

2011

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Caspase activation cascades in apoptosis

Biochemical Society transactions, 2008-02, Vol.36 (Pt 1), p.1-9

2008

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Executioner caspase-3 and caspase-7 are functionally distinct proteases

Proceedings of the National Academy of Sciences - PNAS, 2008-09, Vol.105 (35), p.12815-12819

2008

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Expression, purification and use of recombinant annexin V for the detection of apoptotic cells

Nature protocols, 2009-09, Vol.4 (9), p.1383-1395

2009

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Suppression of Interleukin-33 Bioactivity through Proteolysis by Apoptotic Caspases

Immunity (Cambridge, Mass.), 2009-07, Vol.31 (1), p.84-98

2009

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RIPK1 can function as an inhibitor rather than an initiator of RIPK3‐dependent necroptosis

The FEBS journal, 2014-11, Vol.281 (21), p.4921-4934

2014

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Proteolytic Processing of Interleukin-1 Family Cytokines: Variations on a Common Theme

Cell death and inflammation: the case for IL‐1 family cytokines as the canonical DAMPs of the immune system

Oncogenic Ras-Induced Expression of Noxa and Beclin-1 Promotes Autophagic Cell Death and Limits Clonogenic Survival

Caspase-8 Acts in a Non-enzymatic Role as a Scaffold for Assembly of a Pro-inflammatory “FADDosome” Complex upon TRAIL Stimulation

Bcl-2 Family Proteins Participate in Mitochondrial Quality Control by Regulating Parkin/PINK1-Dependent Mitophagy

Neutrophil-Derived Proteases Escalate Inflammation through Activation of IL-36 Family Cytokines

Parkin Sensitizes toward Apoptosis Induced by Mitochondrial Depolarization through Promoting Degradation of Mcl-1

Fas/CD95-Induced Chemokines Can Serve as “Find-Me” Signals for Apoptotic Cells

A Perspective on Mammalian Caspases as Positive and Negative Regulators of Inflammation

Diverse Activators of the NLRP3 Inflammasome Promote IL-1β Secretion by Triggering Necrosis

Inflammatory outcomes of apoptosis, necrosis and necroptosis

Professor Dame Janet Thornton to receive ‘The FEBS Journal Open Science Award’

Emerging Role for Members of the Bcl-2 Family in Mitochondrial Morphogenesis

Neutrophil extracellular traps can serve as platforms for processing and activation of IL‐1 family cytokines

Autophagy in malignant transformation and cancer progression

Mitochondrial fission/fusion dynamics and apoptosis

Fas and TRAIL ‘death receptors’ as initiators of inflammation: Implications for cancer

Cytotoxic and non-cytotoxic roles of the CTL/NK protease granzyme B

Extracellular Neutrophil Proteases Are Efficient Regulators of IL-1, IL-33, and IL-36 Cytokine Activity but Poor Effectors of Microbial Killing

Granzyme B-Dependent Proteolysis Acts as a Switch to Enhance the Proinflammatory Activity of IL-1α

Caspase activation cascades in apoptosis

Executioner caspase-3 and caspase-7 are functionally distinct proteases

Expression, purification and use of recombinant annexin V for the detection of apoptotic cells

Suppression of Interleukin-33 Bioactivity through Proteolysis by Apoptotic Caspases

RIPK1 can function as an inhibitor rather than an initiator of RIPK3‐dependent necroptosis

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