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Optimized Allotopic Expression of the Human Mitochondrial ND4 Prevents Blindness in a Rat Model of Mitochondrial Dysfunction

American journal of human genetics, 2008-09, Vol.83 (3), p.373-387

2008

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Neuroglobin effectively halts vision loss in Harlequin mice at an advanced stage of optic nerve degeneration

Neurobiology of disease, 2021-11, Vol.159, p.105483-105483, Article 105483

2021

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Mitochondrial DNA mutations in human degenerative diseases and aging

Biochimica et biophysica acta, 1995-05, Vol.1271 (1), p.141-151

1995

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mRNA specific subcellular localization represents a crucial step for fine-tuning of gene expression in mammalian cells

Biochimica et biophysica acta, 2007-04, Vol.1773 (4), p.473-475

Corral-Debrinski, Marisol

2007

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Downregulation of apoptosis-inducing factor in Harlequin mice induces progressive and severe optic atrophy which is durably prevented by AAV2-AIF1 gene therapy

Brain (London, England : 1878), 2012-01, Vol.135 (1), p.35-52

2012

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Transient mitochondrial transcript level decay in oxidative stressed chondrocytes

Journal of cellular physiology, 1994-01, Vol.158 (1), p.128-132

1994

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Targeting allotopic material to the mitochondrial compartment: new tools for better understanding mitochondrial physiology and prospect for therapy

M.S. Médecine sciences, 2007-05, Vol.23 (5), p.519

2007

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Differential proto-oncogene mRNA induction from rats treated with peroxisome proliferators

Biochemical and biophysical research communications, 1990-12, Vol.173 (3), p.855-861

1990

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Optimized Allotopic Expression of the Human Mitochondrial ND4 Prevents Blindness in a Rat Model of Mitochondrial Dysfunction

Neuroglobin effectively halts vision loss in Harlequin mice at an advanced stage of optic nerve degeneration

Mitochondrial DNA mutations in human degenerative diseases and aging

mRNA specific subcellular localization represents a crucial step for fine-tuning of gene expression in mammalian cells

Downregulation of apoptosis-inducing factor in Harlequin mice induces progressive and severe optic atrophy which is durably prevented by AAV2-AIF1 gene therapy

Transient mitochondrial transcript level decay in oxidative stressed chondrocytes

Targeting allotopic material to the mitochondrial compartment: new tools for better understanding mitochondrial physiology and prospect for therapy

Differential proto-oncogene mRNA induction from rats treated with peroxisome proliferators

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