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Two Epstein-Barr virus (EBV) oncoproteins cooperate to repress expression of the proapoptotic tumour-suppressor Bim: clues to the pathogenesis of Burkitt's lymphoma

Oncogene, 2008-01, Vol.27 (4), p.421-433

2008

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A common polymorphism acts as an intragenic modifier of mutant p53 behaviour

Nature genetics, 2000-05, Vol.25 (1), p.47-54

2000

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Physical and Functional Interactions between the Corepressor CtBP and the Epstein-Barr Virus Nuclear Antigen EBNA3C

Journal of Virology, 2001-08, Vol.75 (16), p.7749-7755

2001

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Epstein-Barr virus EBNA3C can disrupt multiple cell cycle checkpoints and induce nuclear division divorced from cytokinesis

Oncogene, 2000-02, Vol.19 (5), p.700-709

2000

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Epstein-Barr virus nuclear antigen (EBNA)3C is an immortalizing oncoprotein with similar properties to adenovirus E1A and papillomavirus E7

Oncogene, 1996-12, Vol.13 (12), p.2541-2549

1996

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TGF-β induces apoptosis in human B cells by transcriptional regulation of BIK and BCL-XL

Cell death and differentiation, 2009-04, Vol.16 (4), p.593-602

2009

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Activators of the Epstein-Barr Virus Lytic Program Concomitantly Induce Apoptosis, but Lytic Gene Expression Protects from Cell Death

Journal of Virology, 2001-03, Vol.75 (5), p.2400-2410

2001

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Novel p53 mutants selected in BRCA-associated tumours which dissociate transformation suppression from other wild-type p53 functions

Oncogene, 1999-04, Vol.18 (15), p.2451-2459

1999

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Epstein-Barr virus nuclear antigen EBNA3C/6 expression maintains the level of latent membrane protein 1 in G1-arrested cells

Journal of Virology, 1994-06, Vol.68 (6), p.3491-3498

1994

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Epstein-Barr virus EBNA3C represses Cp, the major promoter for EBNA expression, but has no effect on the promoter of the cell gene CD21

Journal of Virology, 1997-11, Vol.71 (11), p.8552-8562

1997

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High level expression of ΔN-p63 : a mechanism for the inactivation of p53 in undifferentiated nasopharyngeal carcinoma (NPC)?

Oncogene, 2000-07, Vol.19 (30), p.3439-3444

2000

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Apoptosis induced by TGF-beta 1 in Burkitt's lymphoma cells is caspase 8 dependent but is death receptor independent

The Journal of immunology (1950), 2000-09, Vol.165 (5), p.2500-2510

2000

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Epstein-Barr Virus Nuclear Antigen 3C Interacts with Histone Deacetylase To Repress Transcription

Journal of Virology, 1999-07, Vol.73 (7), p.5688-5697

1999

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Epstein-Barr virus nuclear antigen 3C is a powerful repressor of transcription when tethered to DNA

Journal of Virology, 1996-04, Vol.70 (4), p.2481-2489

1996

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EBV gene expression in an NPC‐related tumour

The EMBO journal, 1989-09, Vol.8 (9), p.2639-2651

1989

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Epstein--Barr virus (EBV) nuclear antigen 6 induces expression of the EBV latent membrane protein and an activated phenotype in Raji cells

Journal of general virology, 1993-03, Vol.74 (3), p.361-369

1993

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Epstein-Barr Virus Latent Gene Expression during the Initiation of B Cell Immortalization

Journal of general virology, 1989-07, Vol.70 (7), p.1755-1764

1989

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A transforming p53 mutant, which binds DNA, transactivates and induces apoptosis reveals a nuclear:cytoplasmic shuttling defect

Oncogene, 1998-03, Vol.16 (11), p.1429-1441

1998

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Epstein-Barr virus suppresses a G(2)/M checkpoint activated by genotoxins

Molecular and cellular biology, 2000-02, Vol.20 (4), p.1344

2000

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p53 mutation with frequent novel codons but not a mutator phenotype in BRCA1- and BRCA2-associated breast tumours

Oncogene, 1998-10, Vol.17 (13), p.1681-1689

1998

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DNA damage in human B cells can induce apoptosis, proceeding from G1/S when p53 is transactivation competent and G2/M when it is transactivation defective

The EMBO journal, 1995-10, Vol.14 (20), p.4994-5005

1995

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Role of epithelium in EBV persistence and pathogenesis of B-cell tumours

The Lancet (British edition), 1988-04, Vol.1 (8590), p.855-857

1988

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Epstein-Barr Virus-Associated Lymphoproliferative Disorders in Immunocompromised Individuals

Advances in Cancer Research, 1991, Vol.57, p.329-380

1991

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Epstein‐Barr virus efficiently immortalizes human B cells without neutralizing the function of p53

The EMBO journal, 1995-04, Vol.14 (7), p.1382-1391

1995

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CpG methylation of viral DNA in EBV‐associated tumours

International journal of cancer, 1990-06, Vol.45 (6), p.1125-1130

1990

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Two Epstein-Barr virus (EBV) oncoproteins cooperate to repress expression of the proapoptotic tumour-suppressor Bim: clues to the pathogenesis of Burkitt's lymphoma

A common polymorphism acts as an intragenic modifier of mutant p53 behaviour

Physical and Functional Interactions between the Corepressor CtBP and the Epstein-Barr Virus Nuclear Antigen EBNA3C

Epstein-Barr virus EBNA3C can disrupt multiple cell cycle checkpoints and induce nuclear division divorced from cytokinesis

Epstein-Barr virus nuclear antigen (EBNA)3C is an immortalizing oncoprotein with similar properties to adenovirus E1A and papillomavirus E7

TGF-β induces apoptosis in human B cells by transcriptional regulation of BIK and BCL-XL

Activators of the Epstein-Barr Virus Lytic Program Concomitantly Induce Apoptosis, but Lytic Gene Expression Protects from Cell Death

Novel p53 mutants selected in BRCA-associated tumours which dissociate transformation suppression from other wild-type p53 functions

Epstein-Barr virus nuclear antigen EBNA3C/6 expression maintains the level of latent membrane protein 1 in G1-arrested cells

Epstein-Barr virus EBNA3C represses Cp, the major promoter for EBNA expression, but has no effect on the promoter of the cell gene CD21

High level expression of ΔN-p63 : a mechanism for the inactivation of p53 in undifferentiated nasopharyngeal carcinoma (NPC)?

Apoptosis induced by TGF-beta 1 in Burkitt's lymphoma cells is caspase 8 dependent but is death receptor independent

Epstein-Barr Virus Nuclear Antigen 3C Interacts with Histone Deacetylase To Repress Transcription

Epstein-Barr virus nuclear antigen 3C is a powerful repressor of transcription when tethered to DNA

EBV gene expression in an NPC‐related tumour

Epstein--Barr virus (EBV) nuclear antigen 6 induces expression of the EBV latent membrane protein and an activated phenotype in Raji cells

Epstein-Barr Virus Latent Gene Expression during the Initiation of B Cell Immortalization

A transforming p53 mutant, which binds DNA, transactivates and induces apoptosis reveals a nuclear:cytoplasmic shuttling defect

Epstein-Barr virus suppresses a G(2)/M checkpoint activated by genotoxins

p53 mutation with frequent novel codons but not a mutator phenotype in BRCA1- and BRCA2-associated breast tumours

DNA damage in human B cells can induce apoptosis, proceeding from G1/S when p53 is transactivation competent and G2/M when it is transactivation defective

Role of epithelium in EBV persistence and pathogenesis of B-cell tumours

Epstein-Barr Virus-Associated Lymphoproliferative Disorders in Immunocompromised Individuals

Epstein‐Barr virus efficiently immortalizes human B cells without neutralizing the function of p53

CpG methylation of viral DNA in EBV‐associated tumours

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