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Ion channel networks in the control of cerebral blood flow

Journal of Cerebral Blood Flow & Metabolism, 2016-03, Vol.36 (3), p.492-512

2016

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Traumatic Brain Injury Causes Endothelial Dysfunction in the Systemic Microcirculation through Arginase-1-Dependent Uncoupling of Endothelial Nitric Oxide Synthase

Journal of neurotrauma, 2017-01, Vol.34 (1), p.192-203

2017

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Epidermal Growth Factor Receptors in Vascular Endothelial Cells Contribute to Functional Hyperemia in the Brain

International journal of molecular sciences, 2023-11, Vol.24 (22), p.16284

2023

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The yin and yang of KV channels in cerebral small vessel pathologies

Microcirculation (New York, N.Y. 1994), 2018-01, Vol.25 (1), p.n/a

2018

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Uncoupling of neurovascular communication after transient global cerebral ischemia is caused by impaired parenchymal smooth muscle Kir channel function

Journal of cerebral blood flow and metabolism, 2016-07, Vol.36 (7), p.1195-1201

2016

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Piezo1 Is a Mechanosensor Channel in Central Nervous System Capillaries

Circulation research, 2022-05, Vol.130 (10), p.1531-1546

2022

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Calcium signaling in smooth muscle

Cold Spring Harbor perspectives in biology, 2011-09, Vol.3 (9), p.a004549-a004549

2011

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Differential restoration of functional hyperemia by antihypertensive drug classes in hypertension-related cerebral small vessel disease

The Journal of clinical investigation, 2021-09, Vol.131 (18)

2021

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Lack of direct effect of adiponectin on vascular smooth muscle cell BKCa channels or Ca2+ signaling in the regulation of small artery pressure‐induced constriction

Physiological reports, 2017-08, Vol.5 (16), p.n/a

2017

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Lack of direct effect of adiponectin on vascular smooth muscle cell BK Ca channels or Ca 2+ signaling in the regulation of small artery pressure-induced constriction

Physiological reports, 2017-08, Vol.5 (16), p.e13337

2017

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Uncoupling of neurovascular communication after transient global cerebral ischemia is caused by impaired parenchymal smooth muscle K ir channel function

Journal of cerebral blood flow and metabolism, 2016-07, Vol.36 (7), p.1195-1201

2016

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Prostaglandin E2, a Postulated Astrocyte-Derived Neurovascular Coupling Agent, Constricts Rather than Dilates Parenchymal Arterioles

Journal of cerebral blood flow and metabolism, 2013-04, Vol.33 (4), p.479-482

2013

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The yin and yang of K V channels in cerebral small vessel pathologies

Microcirculation (New York, N.Y. 1994), 2018-01, Vol.25 (1)

2018

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NFAT Regulation in Smooth Muscle

Trends in Cardiovascular Medicine, 2003-02, Vol.13 (2), p.56-62

2003

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Differential restoration of functional hyperemia by antihypertensive drug classes in hypertension-related cerebral small vessel disease

The Journal of clinical investigation, 2021-09, Vol.131 (18), p.1-6

2021

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Prostaglandin E sub(2), a postulated astrocyte-derived neurovascular coupling agent, constricts rather than dilates parenchymal arterioles

Journal of cerebral blood flow and metabolism, 2013-04, Vol.33 (4), p.479-482

2013

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Prostaglandin E 2 , a Postulated Astrocyte-Derived Neurovascular Coupling Agent, Constricts Rather than Dilates Parenchymal Arterioles

Journal of cerebral blood flow and metabolism, 2013-02, Vol.33 (4), p.479-482

2013

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Local IP3 receptor-mediated Ca2+ signals compound to direct blood flow in brain capillaries

Science advances, 2021-07, Vol.7 (30)

2021

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Inhibition of KIR2.1 by Intracellular Acidification Contributes to Sour Taste Transduction

Biophysical journal, 2016-02, Vol.110 (3), p.424a-424a

2016

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Local IP 3 receptor-mediated Ca 2+ signals compound to direct blood flow in brain capillaries

Science advances, 2021-07, Vol.7 (30)

2021

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Ion channel networks in the control of cerebral blood flow

Traumatic Brain Injury Causes Endothelial Dysfunction in the Systemic Microcirculation through Arginase-1-Dependent Uncoupling of Endothelial Nitric Oxide Synthase

Epidermal Growth Factor Receptors in Vascular Endothelial Cells Contribute to Functional Hyperemia in the Brain

The yin and yang of KV channels in cerebral small vessel pathologies

Uncoupling of neurovascular communication after transient global cerebral ischemia is caused by impaired parenchymal smooth muscle Kir channel function

Piezo1 Is a Mechanosensor Channel in Central Nervous System Capillaries

Calcium signaling in smooth muscle

Differential restoration of functional hyperemia by antihypertensive drug classes in hypertension-related cerebral small vessel disease

Lack of direct effect of adiponectin on vascular smooth muscle cell BKCa channels or Ca2+ signaling in the regulation of small artery pressure‐induced constriction

Lack of direct effect of adiponectin on vascular smooth muscle cell BK Ca channels or Ca 2+ signaling in the regulation of small artery pressure-induced constriction

Uncoupling of neurovascular communication after transient global cerebral ischemia is caused by impaired parenchymal smooth muscle K ir channel function

Prostaglandin E2, a Postulated Astrocyte-Derived Neurovascular Coupling Agent, Constricts Rather than Dilates Parenchymal Arterioles

The yin and yang of K V channels in cerebral small vessel pathologies

NFAT Regulation in Smooth Muscle

Differential restoration of functional hyperemia by antihypertensive drug classes in hypertension-related cerebral small vessel disease

Prostaglandin E sub(2), a postulated astrocyte-derived neurovascular coupling agent, constricts rather than dilates parenchymal arterioles

Prostaglandin E 2 , a Postulated Astrocyte-Derived Neurovascular Coupling Agent, Constricts Rather than Dilates Parenchymal Arterioles

Local IP3 receptor-mediated Ca2+ signals compound to direct blood flow in brain capillaries

Inhibition of KIR2.1 by Intracellular Acidification Contributes to Sour Taste Transduction

Local IP 3 receptor-mediated Ca 2+ signals compound to direct blood flow in brain capillaries

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