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HDAC Inhibition Reverses Preexisting Diastolic Dysfunction and Blocks Covert Extracellular Matrix Remodeling

Circulation (New York, N.Y.), 2021-05, Vol.143 (19), p.1874-1890

2021

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Histone deacetylase activity governs diastolic dysfunction through a nongenomic mechanism

Science translational medicine, 2018-02, Vol.10 (427)

2018

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Structural and in Vivo Characterization of Tubastatin A, a Widely Used Histone Deacetylase 6 Inhibitor

ACS medicinal chemistry letters, 2020-05, Vol.11 (5), p.706-712

2020

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HDAC6 modulates myofibril stiffness and diastolic function of the heart

The Journal of clinical investigation, 2022-05, Vol.132 (10), p.1-13

2022

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Female and male mice have differential longterm cardiorenal outcomes following a matched degree of ischemia–reperfusion acute kidney injury

Scientific reports, 2022-01, Vol.12 (1), p.643-12, Article 643

2022

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Discovery of Novel Small Molecule Inhibitors of Cardiac Hypertrophy Using High Throughput, High Content Imaging

Journal of molecular and cellular cardiology, 2016-08, Vol.97, p.106-113

2016

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Cardiac HDAC6 catalytic activity is induced in response to chronic hypertension

Journal of molecular and cellular cardiology, 2011-07, Vol.51 (1), p.41-50

2011

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An injectable sulfonated reversible thermal gel for therapeutic angiogenesis to protect cardiac function after a myocardial infarction

Journal of biological engineering, 2019-01, Vol.13 (1), p.6-6, Article 6

2019

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Testosterone enhances early cardiac remodeling after myocardial infarction, causing rupture and degrading cardiac function

American journal of physiology. Heart and circulatory physiology, 2006-05, Vol.290 (5), p.H2043-H2050

2006

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Prolyl Oligopeptidase Is Involved in Release of the Antifibrotic Peptide Ac-SDKP

Hypertension (Dallas, Tex. 1979), 2004-05, Vol.43 (5), p.1140-1145

2004

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Decreased Endogenous Levels of Ac-SDKP Promote Organ Fibrosis

Hypertension (Dallas, Tex. 1979), 2007-07, Vol.50 (1), p.130-136

2007

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Endoplasmic Reticulum Stress Effector CCAAT/Enhancer‐binding Protein Homologous Protein (CHOP) Regulates Chronic Kidney Disease–Induced Vascular Calcification

Journal of the American Heart Association, 2014-06, Vol.3 (3), p.e000949-n/a

2014

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Dynamic Chromatin Targeting of BRD4 Stimulates Cardiac Fibroblast Activation

Circulation research, 2019-09, Vol.125 (7), p.662-677

2019

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BET acetyl-lysine binding proteins control pathological cardiac hypertrophy

Journal of molecular and cellular cardiology, 2013-10, Vol.63, p.175-179

2013

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Class I HDACs regulate angiotensin II-dependent cardiac fibrosis via fibroblasts and circulating fibrocytes

Journal of molecular and cellular cardiology, 2014-02, Vol.67, p.112-125

2014

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Inhibition of Eicosanoid Degradation Mitigates Fibrosis of the Heart

Circulation research, 2023-01, Vol.132 (1), p.10-29

2023

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HDAC6 contributes to pathological responses of heart and skeletal muscle to chronic angiotensin-II signaling

American journal of physiology. Heart and circulatory physiology, 2014-07, Vol.307 (2), p.H252-H258

2014

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Role of cardiac overexpression of ANG II in the regulation of cardiac function and remodeling postmyocardial infarction

American journal of physiology. Heart and circulatory physiology, 2007-09, Vol.293 (3), p.H1900-H1907

2007

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Tryptophan hydroxylase 1 Inhibition Impacts Pulmonary Vascular Remodeling in Two Rat Models of Pulmonary Hypertension

The Journal of pharmacology and experimental therapeutics, 2017-02, Vol.360 (2), p.267-279

2017

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Antifibrotic effect of Ac-SDKP and angiotensin-converting enzyme inhibition in hypertension

Journal of hypertension, 2004-03, Vol.22 (3), p.593-603

2004

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Role of nitric oxide in the renal hemodynamic response to unilateral nephrectomy

Journal of the American Society of Nephrology, 2004-06, Vol.15 (6), p.1413-1420

2004

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Selective Class I Histone Deacetylase Inhibition Suppresses Hypoxia-Induced Cardiopulmonary Remodeling Through an Antiproliferative Mechanism

Circulation research, 2012-03, Vol.110 (5), p.739-748

2012

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DUSP5-mediated inhibition of smooth muscle cell proliferation suppresses pulmonary hypertension and right ventricular hypertrophy

American journal of physiology. Heart and circulatory physiology, 2021-08, Vol.321 (2), p.H382-H389

2021

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HDAC11 suppresses the thermogenic program of adipose tissue via BRD2

JCI insight, 2018-08, Vol.3 (15)

2018

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Role of Inflammation in the Development of Renal Damage and Dysfunction in Angiotensin II–Induced Hypertension

Hypertension (Dallas, Tex. 1979), 2008-08, Vol.52 (2), p.256-263

2008

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HDAC Inhibition Reverses Preexisting Diastolic Dysfunction and Blocks Covert Extracellular Matrix Remodeling

Histone deacetylase activity governs diastolic dysfunction through a nongenomic mechanism

Structural and in Vivo Characterization of Tubastatin A, a Widely Used Histone Deacetylase 6 Inhibitor

HDAC6 modulates myofibril stiffness and diastolic function of the heart

Female and male mice have differential longterm cardiorenal outcomes following a matched degree of ischemia–reperfusion acute kidney injury

Discovery of Novel Small Molecule Inhibitors of Cardiac Hypertrophy Using High Throughput, High Content Imaging

Cardiac HDAC6 catalytic activity is induced in response to chronic hypertension

An injectable sulfonated reversible thermal gel for therapeutic angiogenesis to protect cardiac function after a myocardial infarction

Testosterone enhances early cardiac remodeling after myocardial infarction, causing rupture and degrading cardiac function

Prolyl Oligopeptidase Is Involved in Release of the Antifibrotic Peptide Ac-SDKP

Decreased Endogenous Levels of Ac-SDKP Promote Organ Fibrosis

Endoplasmic Reticulum Stress Effector CCAAT/Enhancer‐binding Protein Homologous Protein (CHOP) Regulates Chronic Kidney Disease–Induced Vascular Calcification

Dynamic Chromatin Targeting of BRD4 Stimulates Cardiac Fibroblast Activation

BET acetyl-lysine binding proteins control pathological cardiac hypertrophy

Class I HDACs regulate angiotensin II-dependent cardiac fibrosis via fibroblasts and circulating fibrocytes

Inhibition of Eicosanoid Degradation Mitigates Fibrosis of the Heart

HDAC6 contributes to pathological responses of heart and skeletal muscle to chronic angiotensin-II signaling

Role of cardiac overexpression of ANG II in the regulation of cardiac function and remodeling postmyocardial infarction

Tryptophan hydroxylase 1 Inhibition Impacts Pulmonary Vascular Remodeling in Two Rat Models of Pulmonary Hypertension

Antifibrotic effect of Ac-SDKP and angiotensin-converting enzyme inhibition in hypertension

Role of nitric oxide in the renal hemodynamic response to unilateral nephrectomy

Selective Class I Histone Deacetylase Inhibition Suppresses Hypoxia-Induced Cardiopulmonary Remodeling Through an Antiproliferative Mechanism

DUSP5-mediated inhibition of smooth muscle cell proliferation suppresses pulmonary hypertension and right ventricular hypertrophy

HDAC11 suppresses the thermogenic program of adipose tissue via BRD2

Role of Inflammation in the Development of Renal Damage and Dysfunction in Angiotensin II–Induced Hypertension

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