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Intercellular Adhesion Molecule 1 Regulates Left Ventricular Leukocyte Infiltration, Cardiac Remodeling, and Function in Pressure Overload–Induced Heart Failure

Journal of the American Heart Association, 2016-03, Vol.5 (3), p.e003126-n/a

2016

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CD99 Is a Key Mediator of the Transendothelial Migration of Neutrophils

Journal of Immunology, 2007-01, Vol.178 (2), p.1136-1143

2007

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Neutrophil Recruitment under Shear Flow: It's All about Endothelial Cell Rings and Gaps

Microcirculation (New York, N.Y. 1994), 2009, Vol.16 (1), p.43-57

2009

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Endothelial mineralocorticoid receptor contributes to systolic dysfunction induced by pressure overload without modulating cardiac hypertrophy or inflammation

Physiological reports, 2017-06, Vol.5 (12), p.e13313-n/a

2017

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Deletion of MyD88 in T‐Cells Worsens Cardiac Pathology Through Enhanced T‐Cell Survival and Induction of Cardiac Fibrosis

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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Self‐Antigen‐Specific T Cell Receptor Activation of CD4+ T Cells in the heart is required for Maladaptive Cardiac Remodeling in Pressure Overload Induced Heart Failure

The FASEB journal, 2019-04, Vol.33 (S1), p.374.1-374.1

2019

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Gut Dysbiosis and Barrier Disruption Are Associated with Diastolic Dysfunction in a Novel Mouse Model of Heart Failure with Preserved Ejection Fraction

The FASEB journal, 2021-05, Vol.35 (S1), p.n/a, Article fasebj.2021.35.S1.03672

2021

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Combined Risk Factors Induce T Cell‐Mediated Diastolic Dysfunction in a Novel Mouse Model of Heart Failure with Preserved Ejection Fraction (HFpEF)

The FASEB journal, 2021-05, Vol.35 (S1), p.n/a, Article fasebj.2021.35.S1.01600

2021

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The Mineralocorticoid Receptor in Myeloid Cells Promotes Vascular Inflammation and Atherosclerosis via Transcriptional Regulation of Selplg

The FASEB journal, 2021-05, Vol.35 (S1), p.n/a, Article fasebj.2021.35.S1.01618

2021

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Proinflammatory T Cells with Downregulated Unfolded Protein Response Genes Contribute to Experimental Heart Failure with Preserved Ejection Fraction (HFpEF)

The FASEB journal, 2022-05, Vol.36 (S1)

2022

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GUT MICROBIOTA DEPLETION PRESERVES HEART FUNCTION, SUPPRESSES CARDIAC FIBROSIS AND HYPERTROPHY IN A NON‐ISCHEMIC HEART FAILURE MOUSE MODEL

The FASEB journal, 2018-04, Vol.32 (S1), p.287.3-287.3

2018

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Stimulator of Interferon Genes (STING) Regulates Endothelial Healing and Reendothelialization Following Angioplasty‐Mediated Vascular Injury

The FASEB journal, 2020-04, Vol.34 (S1), p.1-1

2020

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Gut Dysbiosis Induced by Cardiac Pressure Overload Promotes T Cell Activation and Adverse Cardiac Remodeling Through Alterations in the Aryl Hydrocarbon Receptor Expression

The FASEB journal, 2020-04, Vol.34 (S1), p.1-1

2020

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The CXCR3‐LFA1‐ICAM1 Axis Regulates T Cell Cardiotropism and Maladaptive Cardiac Remodeling in Heart Failure

The FASEB journal, 2017-04, Vol.31 (S1)

2017

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GUT BACTERIA MODULATE T CELL RESPONSES AND ADVERSE CARDIAC REMODELING IN PRESSURE OVERLOAD INDUCED HEART FAILURE

The FASEB journal, 2019-04, Vol.33 (S1), p.374.2-374.2

2019

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Stimulator of Interferon Genes Deletion in Primary Heart Endothelial Cells Results in Decreased T Cell Transmigration

The FASEB journal, 2019-04, Vol.33 (S1), p.374.5-374.5

2019

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Stimulator of Interferon Genes (STING) regulates Re‐endothelialization Following Vascular Injury

The FASEB journal, 2021-05, Vol.35 (S1), p.n/a, Article fasebj.2021.35.S1.01616

2021

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CD43 contributes to mouse Th17 cell adhesion to intercellular adhesion molecule‐1

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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CXCR3 Regulates CD4+ T Cell Cardiotropism and Maladaptive Cardiac Remodeling Through Mechanisms Involving ICAM1‐Mediated Adhesion

The FASEB journal, 2018-04, Vol.32 (S1), p.280.3-280.3

2018

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CD43 syalomucin contributes to cardiac inflammation and fibrosis in non‐ischemic heart failure

The FASEB journal, 2017-04, Vol.31 (S1)

2017

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Sialomucin CD 43 regulates T helper type 17 cell intercellular adhesion molecule 1 dependent adhesion, apical migration and transendothelial migration

Immunology, 2019-05, Vol.157 (1), p.52-69

2019

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Sialomucin CD43 regulates T helper type 17 cell intercellular adhesion molecule 1 dependent adhesion, apical migration and transendothelial migration

Immunology, 2019-05, Vol.157 (1), p.52-69

2019

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Th1 Effector T cells Induce Cardiac Fibroblasts Transition to Myofibroblasts and Contribute to Pressure Overload Induced Cardiac Fibrosis

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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Intercellular Adhesion Molecule 1 Regulates Left Ventricular Leukocyte Recruitment, Cardiac Remodeling and Function in Pressure Overload Induced Heart Failure

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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Intercellular Adhesion Molecule 1 (ICAM‐1) Regulates Cardiac Remodeling and Function in Pressure Overload Induced Heart Failure

The FASEB journal, 2015-04, Vol.29 (S1)

2015

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Intercellular Adhesion Molecule 1 Regulates Left Ventricular Leukocyte Infiltration, Cardiac Remodeling, and Function in Pressure Overload–Induced Heart Failure

CD99 Is a Key Mediator of the Transendothelial Migration of Neutrophils

Neutrophil Recruitment under Shear Flow: It's All about Endothelial Cell Rings and Gaps

Endothelial mineralocorticoid receptor contributes to systolic dysfunction induced by pressure overload without modulating cardiac hypertrophy or inflammation

Deletion of MyD88 in T‐Cells Worsens Cardiac Pathology Through Enhanced T‐Cell Survival and Induction of Cardiac Fibrosis

Self‐Antigen‐Specific T Cell Receptor Activation of CD4+ T Cells in the heart is required for Maladaptive Cardiac Remodeling in Pressure Overload Induced Heart Failure

Gut Dysbiosis and Barrier Disruption Are Associated with Diastolic Dysfunction in a Novel Mouse Model of Heart Failure with Preserved Ejection Fraction

Combined Risk Factors Induce T Cell‐Mediated Diastolic Dysfunction in a Novel Mouse Model of Heart Failure with Preserved Ejection Fraction (HFpEF)

The Mineralocorticoid Receptor in Myeloid Cells Promotes Vascular Inflammation and Atherosclerosis via Transcriptional Regulation of Selplg

Proinflammatory T Cells with Downregulated Unfolded Protein Response Genes Contribute to Experimental Heart Failure with Preserved Ejection Fraction (HFpEF)

GUT MICROBIOTA DEPLETION PRESERVES HEART FUNCTION, SUPPRESSES CARDIAC FIBROSIS AND HYPERTROPHY IN A NON‐ISCHEMIC HEART FAILURE MOUSE MODEL

Stimulator of Interferon Genes (STING) Regulates Endothelial Healing and Reendothelialization Following Angioplasty‐Mediated Vascular Injury

Gut Dysbiosis Induced by Cardiac Pressure Overload Promotes T Cell Activation and Adverse Cardiac Remodeling Through Alterations in the Aryl Hydrocarbon Receptor Expression

The CXCR3‐LFA1‐ICAM1 Axis Regulates T Cell Cardiotropism and Maladaptive Cardiac Remodeling in Heart Failure

GUT BACTERIA MODULATE T CELL RESPONSES AND ADVERSE CARDIAC REMODELING IN PRESSURE OVERLOAD INDUCED HEART FAILURE

Stimulator of Interferon Genes Deletion in Primary Heart Endothelial Cells Results in Decreased T Cell Transmigration

Stimulator of Interferon Genes (STING) regulates Re‐endothelialization Following Vascular Injury

CD43 contributes to mouse Th17 cell adhesion to intercellular adhesion molecule‐1

CXCR3 Regulates CD4+ T Cell Cardiotropism and Maladaptive Cardiac Remodeling Through Mechanisms Involving ICAM1‐Mediated Adhesion

CD43 syalomucin contributes to cardiac inflammation and fibrosis in non‐ischemic heart failure

Sialomucin CD 43 regulates T helper type 17 cell intercellular adhesion molecule 1 dependent adhesion, apical migration and transendothelial migration

Sialomucin CD43 regulates T helper type 17 cell intercellular adhesion molecule 1 dependent adhesion, apical migration and transendothelial migration

Th1 Effector T cells Induce Cardiac Fibroblasts Transition to Myofibroblasts and Contribute to Pressure Overload Induced Cardiac Fibrosis

Intercellular Adhesion Molecule 1 Regulates Left Ventricular Leukocyte Recruitment, Cardiac Remodeling and Function in Pressure Overload Induced Heart Failure

Intercellular Adhesion Molecule 1 (ICAM‐1) Regulates Cardiac Remodeling and Function in Pressure Overload Induced Heart Failure

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