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A Doubly Fluorescent HIV-1 Reporter Shows that the Majority of Integrated HIV-1 Is Latent Shortly after Infection

Journal of Virology, 2013-04, Vol.87 (8), p.4716-4727

2013

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An upstream YY1 binding site on the HIV-1 LTR contributes to latent infection

PloS one, 2013-10, Vol.8 (10), p.e77052-e77052

2013

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Identification of the Mismatch Repair Genes PMS2 and MLH1 as p53 Target Genes by Using Serial Analysis of Binding Elements

Proceedings of the National Academy of Sciences - PNAS, 2005-03, Vol.102 (13), p.4813-4818

2005

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HIV Provirus Stably Reproduces Parental Latent and Induced Transcription Phenotypes Regardless of the Chromosomal Integration Site

Journal of virology, 2016-06, Vol.90 (11), p.5302-5314

2016

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TFII-I Regulates Induction of Chromosomally Integrated Human Immunodeficiency Virus Type 1 Long Terminal Repeat in Cooperation with USF

Journal of Virology, 2005-04, Vol.79 (7), p.4396-4406

2005

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Identification of protein interaction antagonists using the repressed transactivator two-hybrid system

BioTechniques, 2007-05, Vol.42 (5), p.635-644

2007

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The Suv39H1 methyltransferase inhibitor chaetocin causes induction of integrated HIV-1 without producing a T cell response

FEBS letters, 2011-11, Vol.585 (22), p.3549-3554

2011

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Naturally Occurring Human Immunodeficiency Virus Type 1 Long Terminal Repeats Have a Frequently Observed Duplication That Binds RBF-2 and Represses Transcription

Journal of Virology, 1998-08, Vol.72 (8), p.6465-6474

1998

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Direct non-productive HIV-1 infection in a T-cell line is driven by cellular activation state and NFκB

Retrovirology, 2014-02, Vol.11 (1), p.17-17, Article 17

2014

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Specific interaction of TFII-I with an upstream element on the HIV-1 LTR regulates induction of latent provirus

FEBS letters, 2008-11, Vol.582 (28), p.3903-3908

2008

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Induction of chromosomally integrated HIV-1 LTR requires RBF-2 (USF/TFII-I) and RAS/MAPK signaling

Virus genes, 2007-10, Vol.35 (2), p.215-223

2007

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Identification and functional analysis of a second RBF-2 binding site within the HIV-1 promoter

Virology (New York, N.Y.), 2011-09, Vol.418 (1), p.57-66

2011

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Purification of RBF-2, a Transcription Factor with Specificity for the Most Conserved cis-Element of Naturally Occurring HIV-1 LTRs

Journal of biomedical science, 1999-10, Vol.6 (5), p.320-332

1999

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A Doubly Fluorescent HIV-1 Reporter Shows that the Majority of Integrated HIV-1 Is Latent Shortly after Infection

An upstream YY1 binding site on the HIV-1 LTR contributes to latent infection

Identification of the Mismatch Repair Genes PMS2 and MLH1 as p53 Target Genes by Using Serial Analysis of Binding Elements

HIV Provirus Stably Reproduces Parental Latent and Induced Transcription Phenotypes Regardless of the Chromosomal Integration Site

TFII-I Regulates Induction of Chromosomally Integrated Human Immunodeficiency Virus Type 1 Long Terminal Repeat in Cooperation with USF

Identification of protein interaction antagonists using the repressed transactivator two-hybrid system

The Suv39H1 methyltransferase inhibitor chaetocin causes induction of integrated HIV-1 without producing a T cell response

Naturally Occurring Human Immunodeficiency Virus Type 1 Long Terminal Repeats Have a Frequently Observed Duplication That Binds RBF-2 and Represses Transcription

Direct non-productive HIV-1 infection in a T-cell line is driven by cellular activation state and NFκB

Specific interaction of TFII-I with an upstream element on the HIV-1 LTR regulates induction of latent provirus

Induction of chromosomally integrated HIV-1 LTR requires RBF-2 (USF/TFII-I) and RAS/MAPK signaling

Identification and functional analysis of a second RBF-2 binding site within the HIV-1 promoter

Purification of RBF-2, a Transcription Factor with Specificity for the Most Conserved cis-Element of Naturally Occurring HIV-1 LTRs

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