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Mechanism of ER Stress-Induced Brain Damage by IP3 Receptor

Neuron (Cambridge, Mass.), 2010-12, Vol.68 (5), p.865-878

2010

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Subtype-Specific and ER Lumenal Environment-Dependent Regulation of Inositol 1,4,5-Trisphosphate Receptor Type 1 by ERp44

Cell, 2005-01, Vol.120 (1), p.85-98

2005

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Distributed and causal influence of frontal operculum in task control

Proceedings of the National Academy of Sciences - PNAS, 2011-03, Vol.108 (10), p.4230-4235

2011

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Aberrant calcium signaling by transglutaminase-mediated posttranslational modification of inositol 1,4,5-trisphosphate receptors

Proceedings of the National Academy of Sciences - PNAS, 2014-09, Vol.111 (38), p.E3966-E3975

2014

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Distinct Roles of Inositol 1,4,5-Trisphosphate Receptor Types 1 and 3 in Ca2+ Signaling

The Journal of biological chemistry, 2004-03, Vol.279 (12), p.11967-11975

2004

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Protein 4.1N Is Required for Translocation of Inositol 1,4,5-Trisphosphate Receptor Type 1 to the Basolateral Membrane Domain in Polarized Madin-Darby Canine Kidney Cells

The Journal of biological chemistry, 2003-02, Vol.278 (6), p.4048-4056

2003

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Aberrant calcium signaling by transglutaminase-mediated posttranslational modification of inositol 1,4,5-trisphosphate receptors

Proceedings of the National Academy of Sciences - PNAS, 2014-09, Vol.111 (38), p.13688-13688

2014

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Mechanism of ER Stress-Induced Brain Damage by IP sub(3) Receptor

Neuron (Cambridge, Mass.), 2010-12, Vol.68 (5), p.865-878

2010

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Activation of the maternally preset program of apoptosis by microinjection of 5‐aza‐2′‐deoxycytidine and 5‐methyl‐2′‐deoxycytidine‐5′‐triphosphate in Xenopus laevis embryos

Development, growth & differentiation, 2001-08, Vol.43 (4), p.383-390

2001

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Mechanism of ER Stress-Induced Brain Damage by IP 3 Receptor

Neuron (Cambridge, Mass.), 2010, Vol.68 (5), p.865-878

2010

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Mechanism of ER stress-induced brain damage by IP(3) receptor

Neuron (Cambridge, Mass.), 2010-12, Vol.68 (5), p.865-878

2010

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Involvement of caspase-9 in execution of the maternal program of apoptosis in Xenopus late blastulae overexpressed with S-adenosylmethionine decarboxylase

Biochemical and biophysical research communications, 2004-12, Vol.325 (4), p.1367-1375

2004

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Occurrence of pre-MBT synthesis of caspase-8 mRNA and activation of caspase-8 prior to execution of SAMDC ( S-adenosylmethionine decarboxylase)-induced, but not p53-induced, apoptosis in Xenopus late blastulae

Biochemical and biophysical research communications, 2005-10, Vol.336 (2), p.682-691

2005

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Protein 4.1N Is Required for Translocation of Inositol 1,4,5-Trisphosphate Receptor Type 1 to the Basolateral Membrane Domain in Polarized Madin-Darby Canine Kidney Cells210

The Journal of biological chemistry, 2003-02, Vol.278 (6), p.4048-4056

2003

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Overexpression of -adenosylmethionine decarboxylase (SAMDC) in Xeno-pus embryos activates maternal program of apoptosis as a "fail-safe" mechanism of early embryogenesis

Cell research, 2003-06, Vol.13 (3), p.147-158

2003

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Maternal program of apoptosis activated shortly after midblastula transition by overexpression of S-adenosylmethionine decarboxylase in Xenopus early embryos

Comparative Biochemistry and Physiology Part B: Biochemistry and Molecular Biology, 2000-06, Vol.126 (2), p.149-155

2000

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Subtype-specific and ER lumenal environment-dependent regulation of IP3 receptor type 1 by ERp44

Tanpakushitsu kakusan koso. Protein, nucleic acid, enzyme, 2005-08, Vol.50 (10 Suppl), p.1292

2005

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Mechanism of ER Stress-Induced Brain Damage by IP3 Receptor

Subtype-Specific and ER Lumenal Environment-Dependent Regulation of Inositol 1,4,5-Trisphosphate Receptor Type 1 by ERp44

Distributed and causal influence of frontal operculum in task control

Aberrant calcium signaling by transglutaminase-mediated posttranslational modification of inositol 1,4,5-trisphosphate receptors

Distinct Roles of Inositol 1,4,5-Trisphosphate Receptor Types 1 and 3 in Ca2+ Signaling

Protein 4.1N Is Required for Translocation of Inositol 1,4,5-Trisphosphate Receptor Type 1 to the Basolateral Membrane Domain in Polarized Madin-Darby Canine Kidney Cells

Aberrant calcium signaling by transglutaminase-mediated posttranslational modification of inositol 1,4,5-trisphosphate receptors

Mechanism of ER Stress-Induced Brain Damage by IP sub(3) Receptor

Activation of the maternally preset program of apoptosis by microinjection of 5‐aza‐2′‐deoxycytidine and 5‐methyl‐2′‐deoxycytidine‐5′‐triphosphate in Xenopus laevis embryos

Mechanism of ER Stress-Induced Brain Damage by IP 3 Receptor

Mechanism of ER stress-induced brain damage by IP(3) receptor

Involvement of caspase-9 in execution of the maternal program of apoptosis in Xenopus late blastulae overexpressed with S-adenosylmethionine decarboxylase

Occurrence of pre-MBT synthesis of caspase-8 mRNA and activation of caspase-8 prior to execution of SAMDC ( S-adenosylmethionine decarboxylase)-induced, but not p53-induced, apoptosis in Xenopus late blastulae

Protein 4.1N Is Required for Translocation of Inositol 1,4,5-Trisphosphate Receptor Type 1 to the Basolateral Membrane Domain in Polarized Madin-Darby Canine Kidney Cells210

Overexpression of -adenosylmethionine decarboxylase (SAMDC) in Xeno-pus embryos activates maternal program of apoptosis as a "fail-safe" mechanism of early embryogenesis

Maternal program of apoptosis activated shortly after midblastula transition by overexpression of S-adenosylmethionine decarboxylase in Xenopus early embryos

Subtype-specific and ER lumenal environment-dependent regulation of IP3 receptor type 1 by ERp44

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