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Genetic, electrophysiological, and pathological studies on patients with SCN9A‐related pain disorders

Journal of the peripheral nervous system, 2023-12, Vol.28 (4), p.597-607

2023

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Sodium Channels in Normal and Pathological Pain

Annual review of neuroscience, 2010-01, Vol.33 (1), p.325-347

2010

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Resilience to Pain: A Peripheral Component Identified Using Induced Pluripotent Stem Cells and Dynamic Clamp

The Journal of neuroscience, 2019-01, Vol.39 (3), p.382-392

2019

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A Novel Gain-of-Function Nav1.9 Mutation in a Child With Episodic Pain

Frontiers in neuroscience, 2019-09, Vol.13, p.918-918

2019

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Expression of Nav1.7 in DRG Neurons Extends from Peripheral Terminals in the Skin to Central Preterminal Branches and Terminals in the Dorsal Horn

Molecular pain, 2012-11, Vol.8 (1), p.82-82

2012

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CSF Monoamine Metabolites in Relation to the Diagnostic Interview for Borderline Patients (DIB)

Neuropsychobiology, 1998-11, Vol.38 (4), p.207-212

1998

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The Na(V)1.7 sodium channel: from molecule to man

Nature reviews. Neuroscience, 2013-01, Vol.14 (1), p.49

2013

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Screening fluorescent voltage indicators with spontaneously spiking HEK cells

PloS one, 2013-12, Vol.8 (12), p.e85221

2013

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Characterization of small fiber pathology in a mouse model of Fabry disease

eLife, 2018-10, Vol.7

2018

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NaV1.7: A central role in pain

Neuron (Cambridge, Mass.), 2023-09, Vol.111 (17), p.2615-2617

2023

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A Novel Persistent Tetrodotoxin-Resistant Sodium Current In SNS-Null And Wild-Type Small Primary Sensory Neurons

The Journal of neuroscience, 1999-12, Vol.19 (24), p.43-RC43

1999

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Gain of function NaV1.7 mutations in idiopathic small fiber neuropathy

Annals of neurology, 2012-01, Vol.71 (1), p.26-39

2012

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Behavioral defects associated with amygdala and cortical dysfunction in mice with seeded α-synuclein inclusions

Neurobiology of disease, 2020-02, Vol.134, p.104708-104708, Article 104708

2020

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Nav1.3 Sodium Channels: Rapid Repriming and Slow Closed-State Inactivation Display Quantitative Differences after Expression in a Mammalian Cell Line and in Spinal Sensory Neurons

The Journal of neuroscience, 2001-08, Vol.21 (16), p.5952-5961

2001

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Human Na(v)1.8: enhanced persistent and ramp currents contribute to distinct firing properties of human DRG neurons

Journal of neurophysiology, 2015-05, Vol.113 (9), p.3172-3185

2015

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A sodium channel mutation linked to epilepsy increases ramp and persistent current of Nav1.3 and induces hyperexcitability in hippocampal neurons

Experimental neurology, 2010-08, Vol.224 (2), p.362-368

2010

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Inherited erythromelalgia due to mutations in SCN9A: natural history, clinical phenotype and somatosensory profile

Brain (London, England : 1878), 2016-04, Vol.139 (Pt 4), p.1052-1065

2016

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Familial trigeminal neuralgia – a systematic clinical study with a genomic screen of the neuronal electrogenisome

Cephalalgia, 2020-07, Vol.40 (8), p.767-777

2020

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The AMPK Activator A769662 Blocks Voltage-Gated Sodium Channels: Discovery of a Novel Pharmacophore with Potential Utility for Analgesic Development

PloS one, 2017-01, Vol.12 (1), p.e0169882

2017

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A sodium channel gene SCN9A polymorphism that increases nociceptor excitability

Annals of neurology, 2009-12, Vol.66 (6), p.862-866

2009

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Nav1.7-A1632G Mutation from a Family with Inherited Erythromelalgia: Enhanced Firing of Dorsal Root Ganglia Neurons Evoked by Thermal Stimuli

The Journal of neuroscience, 2016-07, Vol.36 (28), p.7511-7522

2016

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Inherited Neuronal Ion Channelopathies: New Windows on Complex Neurological Diseases

The Journal of neuroscience, 2008-11, Vol.28 (46), p.11768-11777

2008

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Gain-of-function mutations in sodium channel Na(v)1.9 in painful neuropathy

Brain (London, England : 1878), 2014-06, Vol.137 (Pt 6), p.1627-1642

2014

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The Domain II S4-S5 Linker in Nav1.9: A Missense Mutation Enhances Activation, Impairs Fast Inactivation, and Produces Human Painful Neuropathy

Neuromolecular medicine, 2015-06, Vol.17 (2), p.158-169

2015

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Contributions of NaV1.8 and NaV1.9 to excitability in human induced pluripotent stem-cell derived somatosensory neurons

Scientific reports, 2021-12, Vol.11 (1), p.24283-24283, Article 24283

2021

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Genetic, electrophysiological, and pathological studies on patients with SCN9A‐related pain disorders

Sodium Channels in Normal and Pathological Pain

Resilience to Pain: A Peripheral Component Identified Using Induced Pluripotent Stem Cells and Dynamic Clamp

A Novel Gain-of-Function Nav1.9 Mutation in a Child With Episodic Pain

Expression of Nav1.7 in DRG Neurons Extends from Peripheral Terminals in the Skin to Central Preterminal Branches and Terminals in the Dorsal Horn

CSF Monoamine Metabolites in Relation to the Diagnostic Interview for Borderline Patients (DIB)

The Na(V)1.7 sodium channel: from molecule to man

Screening fluorescent voltage indicators with spontaneously spiking HEK cells

Characterization of small fiber pathology in a mouse model of Fabry disease

NaV1.7: A central role in pain

A Novel Persistent Tetrodotoxin-Resistant Sodium Current In SNS-Null And Wild-Type Small Primary Sensory Neurons

Gain of function NaV1.7 mutations in idiopathic small fiber neuropathy

Behavioral defects associated with amygdala and cortical dysfunction in mice with seeded α-synuclein inclusions

Nav1.3 Sodium Channels: Rapid Repriming and Slow Closed-State Inactivation Display Quantitative Differences after Expression in a Mammalian Cell Line and in Spinal Sensory Neurons

Human Na(v)1.8: enhanced persistent and ramp currents contribute to distinct firing properties of human DRG neurons

A sodium channel mutation linked to epilepsy increases ramp and persistent current of Nav1.3 and induces hyperexcitability in hippocampal neurons

Inherited erythromelalgia due to mutations in SCN9A: natural history, clinical phenotype and somatosensory profile

Familial trigeminal neuralgia – a systematic clinical study with a genomic screen of the neuronal electrogenisome

The AMPK Activator A769662 Blocks Voltage-Gated Sodium Channels: Discovery of a Novel Pharmacophore with Potential Utility for Analgesic Development

A sodium channel gene SCN9A polymorphism that increases nociceptor excitability

Nav1.7-A1632G Mutation from a Family with Inherited Erythromelalgia: Enhanced Firing of Dorsal Root Ganglia Neurons Evoked by Thermal Stimuli

Inherited Neuronal Ion Channelopathies: New Windows on Complex Neurological Diseases

Gain-of-function mutations in sodium channel Na(v)1.9 in painful neuropathy

The Domain II S4-S5 Linker in Nav1.9: A Missense Mutation Enhances Activation, Impairs Fast Inactivation, and Produces Human Painful Neuropathy

Contributions of NaV1.8 and NaV1.9 to excitability in human induced pluripotent stem-cell derived somatosensory neurons

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