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Downregulation of SREBP inhibits tumor growth and initiation by altering cellular metabolism in colon cancer

Cell death & disease, 2018-02, Vol.9 (3), p.265-14, Article 265

2018

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Altered lipid metabolism in APC-driven colorectal cancer: the potential for therapeutic intervention

Frontiers in oncology, 2024-03, Vol.14, p.1343061-1343061

2024

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Spermine synthase and MYC cooperate to maintain colorectal cancer cell survival by repressing Bim expression

Nature communications, 2020-06, Vol.11 (1), p.3243-16, Article 3243

2020

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Inhibition of Fatty Acid Synthase Upregulates Expression of CD36 to Sustain Proliferation of Colorectal Cancer Cells

Frontiers in oncology, 2020-07, Vol.10, p.1185

2020

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N-glycosylation-defective splice variants of neuropilin-1 promote metastasis by activating endosomal signals

Nature communications, 2019-08, Vol.10 (1), p.3708-16, Article 3708

2019

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De Novo Fatty Acid Synthesis-Driven Sphingolipid Metabolism Promotes Metastatic Potential of Colorectal Cancer

Molecular cancer research, 2019-01, Vol.17 (1), p.140-152

2019

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Overexpression of Fatty Acid Synthase Upregulates Glutamine-Fructose-6-Phosphate Transaminase 1 and O-Linked N-Acetylglucosamine Transferase to Increase O-GlcNAc Protein Glycosylation and Promote Colorectal Cancer Growth

International journal of molecular sciences, 2024-05, Vol.25 (9), p.4883

2024

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Increased expression of fatty acid synthase provides a survival advantage to colorectal cancer cells via upregulation of cellular respiration

Oncotarget, 2015-08, Vol.6 (22), p.18891-18904

2015

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Preclinical evaluation of novel fatty acid synthase inhibitors in primary colorectal cancer cells and a patient-derived xenograft model of colorectal cancer

Oncotarget, 2018-05, Vol.9 (37), p.24787-24800

2018

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Upregulation of CD36, a Fatty Acid Translocase, Promotes Colorectal Cancer Metastasis by Increasing MMP28 and Decreasing E-Cadherin Expression

Cancers, 2022-01, Vol.14 (1), p.252

2022

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Novel chemotherapeutic agent, FND-4b, activates AMPK and inhibits colorectal cancer cell proliferation

PloS one, 2019-10, Vol.14 (10), p.e0224253

2019

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Cancer cell-associated fatty acid synthase activates endothelial cells and promotes angiogenesis in colorectal cancer

Carcinogenesis (New York), 2014-06, Vol.35 (6), p.1341-1351

2014

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Activation of c-Met and upregulation of CD44 expression are associated with the metastatic phenotype in the colorectal cancer liver metastasis model

PloS one, 2014-05, Vol.9 (5), p.e97432-e97432

2014

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Tissue-Specific Downregulation of Fatty Acid Synthase Suppresses Intestinal Adenoma Formation via Coordinated Reprograming of Transcriptome and Metabolism in the Mouse Model of Apc-Driven Colorectal Cancer

International journal of molecular sciences, 2022-06, Vol.23 (12), p.6510

2022

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The role of perfluorooctane sulfonic acid (PFOS) exposure in inflammation of intestinal tissues and intestinal carcinogenesis

Frontiers in toxicology, 2023-08, Vol.5, p.1244457-1244457

2023

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mTOR inhibitors in cancer therapy

Cancer letters, 2012-06, Vol.319 (1), p.1-7

2012

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Sorafenib enhances the therapeutic efficacy of rapamycin in colorectal cancers harboring oncogenic KRAS and PIK3CA

Carcinogenesis (New York), 2012-09, Vol.33 (9), p.1782-1790

2012

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Adipocytes activate mitochondrial fatty acid oxidation and autophagy to promote tumor growth in colon cancer

Cell death & disease, 2017-02, Vol.8 (2), p.e2593-e2593

2017

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Down-regulation of PPARgamma1 suppresses cell growth and induces apoptosis in MCF-7 breast cancer cells

Molecular cancer, 2008-12, Vol.7 (1), p.90-90, Article 90

2008

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Upregulation of CPT1A is essential for the tumor-promoting effect of adipocytes in colon cancer

Cell death & disease, 2020-09, Vol.11 (9), p.736, Article 736

2020

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Inflammation and cardiometabolic diseases induced by persistent organic pollutants and nutritional interventions: Effects of multi-organ interactions

Environmental pollution (1987), 2023-12, Vol.339, p.122756-122756, Article 122756

2023

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Colorectal cancer lung metastasis treatment with polymer–drug nanoparticles

Journal of controlled release, 2018-04, Vol.275, p.85-91

2018

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Perfluorooctanesulfonic acid exposure leads to downregulation of 3-hydroxy-3-methylglutaryl-CoA synthase 2 expression and upregulation of markers associated with intestinal carcinogenesis in mouse intestinal tissues

Chemosphere (Oxford), 2024-07, Vol.359, p.142332, Article 142332

2024

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Diaminobutoxy-substituted Isoflavonoid (DBI-1) Enhances the Therapeutic Efficacy of GLUT1 Inhibitor BAY-876 by Modulating Metabolic Pathways in Colon Cancer Cells

Molecular cancer therapeutics, 2022-05, Vol.21 (5), p.740-750

2022

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Temperature induces significant changes in both glycolytic reserve and mitochondrial spare respiratory capacity in colorectal cancer cell lines

Experimental cell research, 2017-05, Vol.354 (2), p.112-121

2017

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Downregulation of SREBP inhibits tumor growth and initiation by altering cellular metabolism in colon cancer

Altered lipid metabolism in APC-driven colorectal cancer: the potential for therapeutic intervention

Spermine synthase and MYC cooperate to maintain colorectal cancer cell survival by repressing Bim expression

Inhibition of Fatty Acid Synthase Upregulates Expression of CD36 to Sustain Proliferation of Colorectal Cancer Cells

N-glycosylation-defective splice variants of neuropilin-1 promote metastasis by activating endosomal signals

De Novo Fatty Acid Synthesis-Driven Sphingolipid Metabolism Promotes Metastatic Potential of Colorectal Cancer

Overexpression of Fatty Acid Synthase Upregulates Glutamine-Fructose-6-Phosphate Transaminase 1 and O-Linked N-Acetylglucosamine Transferase to Increase O-GlcNAc Protein Glycosylation and Promote Colorectal Cancer Growth

Increased expression of fatty acid synthase provides a survival advantage to colorectal cancer cells via upregulation of cellular respiration

Preclinical evaluation of novel fatty acid synthase inhibitors in primary colorectal cancer cells and a patient-derived xenograft model of colorectal cancer

Upregulation of CD36, a Fatty Acid Translocase, Promotes Colorectal Cancer Metastasis by Increasing MMP28 and Decreasing E-Cadherin Expression

Novel chemotherapeutic agent, FND-4b, activates AMPK and inhibits colorectal cancer cell proliferation

Cancer cell-associated fatty acid synthase activates endothelial cells and promotes angiogenesis in colorectal cancer

Activation of c-Met and upregulation of CD44 expression are associated with the metastatic phenotype in the colorectal cancer liver metastasis model

Tissue-Specific Downregulation of Fatty Acid Synthase Suppresses Intestinal Adenoma Formation via Coordinated Reprograming of Transcriptome and Metabolism in the Mouse Model of Apc-Driven Colorectal Cancer

The role of perfluorooctane sulfonic acid (PFOS) exposure in inflammation of intestinal tissues and intestinal carcinogenesis

mTOR inhibitors in cancer therapy

Sorafenib enhances the therapeutic efficacy of rapamycin in colorectal cancers harboring oncogenic KRAS and PIK3CA

Adipocytes activate mitochondrial fatty acid oxidation and autophagy to promote tumor growth in colon cancer

Down-regulation of PPARgamma1 suppresses cell growth and induces apoptosis in MCF-7 breast cancer cells

Upregulation of CPT1A is essential for the tumor-promoting effect of adipocytes in colon cancer

Inflammation and cardiometabolic diseases induced by persistent organic pollutants and nutritional interventions: Effects of multi-organ interactions

Colorectal cancer lung metastasis treatment with polymer–drug nanoparticles

Perfluorooctanesulfonic acid exposure leads to downregulation of 3-hydroxy-3-methylglutaryl-CoA synthase 2 expression and upregulation of markers associated with intestinal carcinogenesis in mouse intestinal tissues

Diaminobutoxy-substituted Isoflavonoid (DBI-1) Enhances the Therapeutic Efficacy of GLUT1 Inhibitor BAY-876 by Modulating Metabolic Pathways in Colon Cancer Cells

Temperature induces significant changes in both glycolytic reserve and mitochondrial spare respiratory capacity in colorectal cancer cell lines

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