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Nearly 100% Internal Quantum Efficiency in Undoped Electroluminescent Devices Employing Pure Organic Emitters

Advanced materials (Weinheim), 2015-03, Vol.27 (12), p.2096-2100

2015

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Systemic sclerosis: Is the epithelium a missing piece of the pathogenic puzzle?

Journal of dermatological science, 2019-05, Vol.94 (2), p.259-265

2019

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Fibrosis, Vascular Activation, and Immune Abnormalities Resembling Systemic Sclerosis in Bleomycin‐Treated Fli‐1–Haploinsufficient Mice

Arthritis & rheumatology (Hoboken, N.J.), 2015-02, Vol.67 (2), p.517-526

2015

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Simultaneous downregulation of KLF5 and Fli1 is a key feature underlying systemic sclerosis

Nature communications, 2014-12, Vol.5 (1), p.5797-5797, Article 5797

2014

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Donor-acceptor-structured 1,4-diazatriphenylene derivatives exhibiting thermally activated delayed fluorescence: design and synthesis, photophysical properties and OLED characteristics

Science and technology of advanced materials, 2014-06, Vol.15 (3), p.034202-10

2014

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Altered Properties of Endothelial Cells and Mesenchymal Stem Cells Underlying the Development of Scleroderma‐like Vasculopathy in KLF5+/−;Fli‐1+/− Mice

Arthritis & rheumatology (Hoboken, N.J.), 2020-12, Vol.72 (12), p.2136-2146

2020

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CXCL13 produced by macrophages due to Fli1 deficiency may contribute to the development of tissue fibrosis, vasculopathy and immune activation in systemic sclerosis

Experimental dermatology, 2018-09, Vol.27 (9), p.1030-1037

2018

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Glycyrrhizin Ameliorates Fibrosis, Vasculopathy, and Inflammation in Animal Models of Systemic Sclerosis

Journal of investigative dermatology, 2017-03, Vol.137 (3), p.631-640

2017

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Amelioration of Tissue Fibrosis by Toll‐like Receptor 4 Knockout in Murine Models of Systemic Sclerosis

Arthritis & rheumatology (Hoboken, N.J.), 2015-01, Vol.67 (1), p.254-265

2015

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Fli1 deficiency induces endothelial adipsin expression, contributing to the onset of pulmonary arterial hypertension in systemic sclerosis

Rheumatology (Oxford, England), 2020-08, Vol.59 (8), p.2005-2015

2020

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Application of wide-energy-gap material 3,4-di(9H-carbazol-9-yl) benzonitrile in organic light-emitting diodes

Thin solid films, 2016-11, Vol.619, p.120-124

2016

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Endothelin Receptor Blockade Ameliorates Vascular Fragility in Endothelial Cell–Specific Fli‐1–Knockout Mice by Increasing Fli‐1 DNA Binding Ability

Arthritis & rheumatology (Hoboken, N.J.), 2015-05, Vol.67 (5), p.1335-1344

2015

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Clinical practice pattern in patients with advanced urothelial cancer who had progressed on pembrolizumab in the pre‐enfortumab vedotin era

International journal of urology, 2022-07, Vol.29 (7), p.647-655

2022

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Fli1-haploinsufficient dermal fibroblasts promote skin-localized transdifferentiation of Th2-like regulatory T cells

Arthritis research & therapy, 2018-02, Vol.20 (1), p.23-23, Article 23

2018

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Increased expression of chemerin in endothelial cells due to Fli1 deficiency may contribute to the development of digital ulcers in systemic sclerosis

Rheumatology (Oxford, England), 2015-07, Vol.54 (7), p.1308-1316

2015

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Tamibarotene Ameliorates Bleomycin-Induced Dermal Fibrosis by Modulating Phenotypes of Fibroblasts, Endothelial Cells, and Immune Cells

Journal of investigative dermatology, 2016-02, Vol.136 (2), p.387-398

2016

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A Three-Level GaN Driver for High False Turn-ON Tolerance with Minimal Reverse Conduction Loss

IEEE open journal of power electronics, 2023-01, Vol.4, p.1-10

2023

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Progranulin Overproduction Due to Fli‐1 Deficiency Contributes to the Resistance of Dermal Fibroblasts to Tumor Necrosis Factor in Systemic Sclerosis

Arthritis & rheumatology (Hoboken, N.J.), 2015-12, Vol.67 (12), p.3245-3255

2015

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Bosentan reverses the pro-fibrotic phenotype of systemic sclerosis dermal fibroblasts via increasing DNA binding ability of transcription factor Fli1

Arthritis research & therapy, 2014-04, Vol.16 (2), p.R86-R86

2014

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The impact of transcription factor Fli1 deficiency on the regulation of angiogenesis

Experimental dermatology, 2017-10, Vol.26 (10), p.912-918

2017

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Regulation of skin fibrosis by RALDH1-producing dermal dendritic cells via retinoic acid-mediated regulatory T cell induction: A role in scleroderma

Journal of dermatological science, 2020-02, Vol.97 (2), p.125-134

2020

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Increased expression of aquaporin-1 in dermal fibroblasts and dermal microvascular endothelial cells possibly contributes to skin fibrosis and edema in patients with systemic sclerosis

Journal of dermatological science, 2019-01, Vol.93 (1), p.24-32

2019

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A possible contribution of endothelial CCN1 downregulation due to Fli1 deficiency to the development of digital ulcers in systemic sclerosis

Experimental dermatology, 2015-02, Vol.24 (2), p.127-132

2015

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Progranulin overproduction due to constitutively activated c-Abl/PKC-δ/Fli1 pathway contributes to the resistance of dermal fibroblasts to the anti-fibrotic effect of tumor necrosis factor-α in localized scleroderma

Journal of dermatological science, 2018-11, Vol.92 (2), p.207-214

2018

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Nearly 100% Internal Quantum Efficiency in Undoped Electroluminescent Devices Employing Pure Organic Emitters

Systemic sclerosis: Is the epithelium a missing piece of the pathogenic puzzle?

Fibrosis, Vascular Activation, and Immune Abnormalities Resembling Systemic Sclerosis in Bleomycin‐Treated Fli‐1–Haploinsufficient Mice

Simultaneous downregulation of KLF5 and Fli1 is a key feature underlying systemic sclerosis

Donor-acceptor-structured 1,4-diazatriphenylene derivatives exhibiting thermally activated delayed fluorescence: design and synthesis, photophysical properties and OLED characteristics

Impaired Insulin Signaling in Endothelial Cells Reduces Insulin-Induced Glucose Uptake by Skeletal Muscle

Altered Properties of Endothelial Cells and Mesenchymal Stem Cells Underlying the Development of Scleroderma‐like Vasculopathy in KLF5+/−;Fli‐1+/− Mice

CXCL13 produced by macrophages due to Fli1 deficiency may contribute to the development of tissue fibrosis, vasculopathy and immune activation in systemic sclerosis

Glycyrrhizin Ameliorates Fibrosis, Vasculopathy, and Inflammation in Animal Models of Systemic Sclerosis

Amelioration of Tissue Fibrosis by Toll‐like Receptor 4 Knockout in Murine Models of Systemic Sclerosis

Fli1 deficiency induces endothelial adipsin expression, contributing to the onset of pulmonary arterial hypertension in systemic sclerosis

Application of wide-energy-gap material 3,4-di(9H-carbazol-9-yl) benzonitrile in organic light-emitting diodes

Endothelin Receptor Blockade Ameliorates Vascular Fragility in Endothelial Cell–Specific Fli‐1–Knockout Mice by Increasing Fli‐1 DNA Binding Ability

Clinical practice pattern in patients with advanced urothelial cancer who had progressed on pembrolizumab in the pre‐enfortumab vedotin era

Fli1-haploinsufficient dermal fibroblasts promote skin-localized transdifferentiation of Th2-like regulatory T cells

Increased expression of chemerin in endothelial cells due to Fli1 deficiency may contribute to the development of digital ulcers in systemic sclerosis

Tamibarotene Ameliorates Bleomycin-Induced Dermal Fibrosis by Modulating Phenotypes of Fibroblasts, Endothelial Cells, and Immune Cells

A Three-Level GaN Driver for High False Turn-ON Tolerance with Minimal Reverse Conduction Loss

Progranulin Overproduction Due to Fli‐1 Deficiency Contributes to the Resistance of Dermal Fibroblasts to Tumor Necrosis Factor in Systemic Sclerosis

Bosentan reverses the pro-fibrotic phenotype of systemic sclerosis dermal fibroblasts via increasing DNA binding ability of transcription factor Fli1

The impact of transcription factor Fli1 deficiency on the regulation of angiogenesis

Regulation of skin fibrosis by RALDH1-producing dermal dendritic cells via retinoic acid-mediated regulatory T cell induction: A role in scleroderma

Increased expression of aquaporin-1 in dermal fibroblasts and dermal microvascular endothelial cells possibly contributes to skin fibrosis and edema in patients with systemic sclerosis

A possible contribution of endothelial CCN1 downregulation due to Fli1 deficiency to the development of digital ulcers in systemic sclerosis

Progranulin overproduction due to constitutively activated c-Abl/PKC-δ/Fli1 pathway contributes to the resistance of dermal fibroblasts to the anti-fibrotic effect of tumor necrosis factor-α in localized scleroderma

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