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Fluency tests of verbal and non-verbal executive functions performance in anxiety patients with allostatic load and neuroticism treated with alprazolam

European neuropsychopharmacology, 2016-10, Vol.26, p.S602-S602

2016

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P.1.e.009 Neurocognitive and neurochemical responses in patients with anxiety and allostatic load: beyond anxiolysis, can cognition be improved by alprazolam?

European neuropsychopharmacology, 2015-09, Vol.25, p.S214-S215

2015

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Sex and age related differences in allostatic load index in patients with general anxiety disorder treated with alprazolam

European neuropsychopharmacology, 2019, Vol.29, p.S195-S195

2019

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Allostatic load and cognitive outcome after 12 weeks of alprazolam treatment in patients with anxiety disorders

European neuropsychopharmacology, 2017-10, Vol.27, p.S1005

2017

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P.4.d.005 - Allostatic load and cognitive outcome after 12 weeks of alprazolam treatment in patients with anxiety disorders

European neuropsychopharmacology, 2017-10, Vol.27, p.S1005-S1005

2017

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P.4.b.004 - Fluency tests of verbal and non-verbal executive functions performance in anxiety patients with allostatic load and neuroticism treated with alprazolam

European neuropsychopharmacology, 2016-10, Vol.26, p.S602-S602

2016

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Moclobemide and clomipramine in resistant depressions. Six months of combined use

European neuropsychopharmacology, 1994, Vol.4 (3), p.307-308

1994

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An ancestral role for the mitochondrial pyruvate carrier in glucose-stimulated insulin secretion

Molecular metabolism (Germany), 2016-08, Vol.5 (8), p.602-614

2016

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Pancreatic [beta]-cell identity in diabetes

Diabetes, obesity & metabolism, 2016-09, Vol.18 (S1), p.110

2016

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KATP channelopathies in the pancreas

Pflügers Archiv, 2010-07, Vol.460 (2), p.307-320

2010

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Biophysical and Pharmacological Characterization of Atp-Sensitive Potassium Channels in Mice Kir6.1WT/V65M Mirroring the Human Cantu′ Syndrome

Biophysical journal, 2020-02, Vol.118 (3), p.589a-590a

2020

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Beta‐cell excitability and excitability‐driven diabetes in adult Zebrafish islets

Physiological reports, 2019-06, Vol.7 (11), p.e14101-n/a

2019

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DO WE STICK TO INTERNATIONAL GUIDELINES WHEN TREATING EARLY STAGE HODGKIN LYMPHOMA?

Hematological oncology, 2021-06, Vol.39 (S2)

2021

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Hyperinsulinism in mice with heterozygous loss of KATP channels

Diabetologia, 2006-10, Vol.49 (10), p.2368-2378

2006

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Secondary Consequences of β Cell Inexcitability: Identification and Prevention in a Murine Model of KATP-Induced Neonatal Diabetes Mellitus

Cell metabolism, 2009-02, Vol.9 (2), p.140-151

2009

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Secondary Consequences of β Cell Inexcitability: Identification and Prevention in a Murine Model of K ATP-Induced Neonatal Diabetes Mellitus

Cell metabolism, 2009-02, Vol.9 (2), p.140-151

2009

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Defects in beta cell Ca2+ signalling, glucose metabolism and insulin secretion in a murine model of KATP channel-induced neonatal diabetes mellitus

Diabetologia, 2011-05, Vol.54 (5), p.1087-1097

2011

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Genetic reduction of glucose metabolism preserves functional β-cell mass in KATP-induced neonatal diabetes

Yearbook of Paediatric Endocrinology, 2022-09

2022

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K sub(ATP) channelopathies in the pancreas

Pflügers Archiv, 2010-07, Vol.460 (2), p.307-320

2010

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The Pan American Cable System

IEEE communications magazine, 1997-12, Vol.35 (12), p.134-140

1997

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Hyperinsulinism induced by targeted suppression of beta cell K ATP channels

Proceedings of the National Academy of Sciences - PNAS, 2002-12, Vol.99 (26), p.16992-16997

2002

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Remodeling of excitation-contraction coupling in transgenic mice expressing ATP-insensitive sarcolemmal K ATP channels

American journal of physiology. Heart and circulatory physiology, 2004-04, Vol.286 (4), p.H1361-H1369

2004

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Transgenic overexpression of SUR1 in the heart suppresses sarcolemmal K ATP

Journal of molecular and cellular cardiology, 2005-10, Vol.39 (4), p.647-656

2005

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Defects in beta cell Ca^sup 2+^ signalling, glucose metabolism and insulin secretion in a murine model of K^sub ATP^ channel-induced neonatal diabetes mellitus

Diabetologia, 2011-05, Vol.54 (5), p.1087

2011

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Circulation de la connaissance à travers la formation, la production et la communication scientifique au département de physiologie, biophysique et neurosciences du Cinvestav

Sens public (Revue), 2016-03, Vol.n° 19-20 (1), p.264-283

2016

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Fluency tests of verbal and non-verbal executive functions performance in anxiety patients with allostatic load and neuroticism treated with alprazolam

P.1.e.009 Neurocognitive and neurochemical responses in patients with anxiety and allostatic load: beyond anxiolysis, can cognition be improved by alprazolam?

Sex and age related differences in allostatic load index in patients with general anxiety disorder treated with alprazolam

Allostatic load and cognitive outcome after 12 weeks of alprazolam treatment in patients with anxiety disorders

P.4.d.005 - Allostatic load and cognitive outcome after 12 weeks of alprazolam treatment in patients with anxiety disorders

P.4.b.004 - Fluency tests of verbal and non-verbal executive functions performance in anxiety patients with allostatic load and neuroticism treated with alprazolam

Moclobemide and clomipramine in resistant depressions. Six months of combined use

An ancestral role for the mitochondrial pyruvate carrier in glucose-stimulated insulin secretion

Pancreatic [beta]-cell identity in diabetes

KATP channelopathies in the pancreas

Biophysical and Pharmacological Characterization of Atp-Sensitive Potassium Channels in Mice Kir6.1WT/V65M Mirroring the Human Cantu′ Syndrome

Beta‐cell excitability and excitability‐driven diabetes in adult Zebrafish islets

DO WE STICK TO INTERNATIONAL GUIDELINES WHEN TREATING EARLY STAGE HODGKIN LYMPHOMA?

Hyperinsulinism in mice with heterozygous loss of KATP channels

Secondary Consequences of β Cell Inexcitability: Identification and Prevention in a Murine Model of KATP-Induced Neonatal Diabetes Mellitus

Secondary Consequences of β Cell Inexcitability: Identification and Prevention in a Murine Model of K ATP-Induced Neonatal Diabetes Mellitus

Defects in beta cell Ca2+ signalling, glucose metabolism and insulin secretion in a murine model of KATP channel-induced neonatal diabetes mellitus

Genetic reduction of glucose metabolism preserves functional β-cell mass in KATP-induced neonatal diabetes

K sub(ATP) channelopathies in the pancreas

The Pan American Cable System

Hyperinsulinism induced by targeted suppression of beta cell K ATP channels

Remodeling of excitation-contraction coupling in transgenic mice expressing ATP-insensitive sarcolemmal K ATP channels

Transgenic overexpression of SUR1 in the heart suppresses sarcolemmal K ATP

Defects in beta cell Ca^sup 2+^ signalling, glucose metabolism and insulin secretion in a murine model of K^sub ATP^ channel-induced neonatal diabetes mellitus

Circulation de la connaissance à travers la formation, la production et la communication scientifique au département de physiologie, biophysique et neurosciences du Cinvestav

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