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Chromatin-Bound Cullin-Ring Ligases: Regulatory Roles in DNA Replication and Potential Targeting for Cancer Therapy

Frontiers in molecular biosciences, 2018-03, Vol.5, p.19

2018

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γH2AX and cancer

Nature reviews. Cancer, 2008-12, Vol.8 (12), p.957-967

2008

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RepID-deficient cancer cells are sensitized to a drug targeting p97/VCP segregase

Molecular & Cellular Toxicology, 2021, 17(2), , pp.141-149

2021

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Telomere-dependent and telomere-independent origins of endogenous DNA damage in tumor cells

Aging (Albany, NY.), 2009-02, Vol.1 (2), p.212-218

2009

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Switching DCAFs: Beyond substrate receptors

BioEssays, 2021-07, Vol.43 (7), p.e2100057-n/a

2021

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Histone yH2AX and Poly(ADP-Ribose) as Clinical Pharmacodynamic Biomarkers

Clinical cancer research, 2010-09, Vol.16 (18), p.4532-4542

2010

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γ-H2AX as a biomarker of DNA damage induced by ionizing radiation in human peripheral blood lymphocytes and artificial skin

Advances in space research, 2009-01, Vol.43 (8), p.1171-1178

2009

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NADPH Oxidases NOXs and DUOXs as Putative Targets for Cancer Therapy

Anti-cancer agents in medicinal chemistry, 2013-01, Vol.13 (3), p.502-514

2013

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Acetalax (Oxyphenisatin Acetate, NSC 59687) and Bisacodyl Cause Oncosis in Triple-Negative Breast Cancer Cell Lines by Poisoning the Ion Exchange Membrane Protein TRPM4

Cancer research communications, 2024-08, Vol.4 (8), p.2101-2111

2024

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Para-inflammation mediates systemic DNA damage in response to tumor growth

Communicative & integrative biology, 2011-01, Vol.4 (1), p.78-81

2011

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Preventing Excess Replication Origin Activation to Insure Genome Stability

Trends in genetics, 2021-10, Vol.38 (2), p.169-181

2021

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Histone γH2AX and Poly(ADP-Ribose) as Clinical Pharmacodynamic Biomarkers

Clinical cancer research, 2010-09, Vol.16 (18), p.4532-4542

2010

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Examining the reliability of surrogate tissue predictability of drug efficacy in reconstituted human 3D skin models containing melanomas

Journal of clinical oncology, 2012-10, Vol.30 (30_suppl), p.49-49

2012

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Abstract 4774: Histone H2AX is a novel regulator of epithelial to mesenchymal transition

Cancer research (Chicago, Ill.), 2015-08, Vol.75 (15_Supplement), p.4774-4774

2015

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Q γ −H2AX, an analysis method for partial-body radiation exposure using γ-H2AX in non-human primate lymphocytes

Radiation measurements, 2011-09, Vol.46 (9), p.877-881

2011

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Using gamma-H2AX and H2AX quantitative ELISA for monitoring DNA damage induced by chemotherapeutic agents and irradiation exposure

Journal of clinical oncology, 2015-05, Vol.33 (15_suppl), p.2559-2559

2015

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Abstract 4041: Wild-type p53-induced phosphatase 1 (Wip1) prevents cellular senescence at physiological oxygen levels by regulating DNA damage signaling during DNA replication

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.4041-4041

2013

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Use of the [gamma]-H2AX assay to monitor DNA damage and repair in translational cancer research

Cancer letters, 2012-12, Vol.327 (1-2), p.123

2012

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Abstract LB_A10: Efficacy of ATR inhibitor berzosertib plus topotecan or topotecan alone in patients with relapsed small cell lung cancer: A randomized clinical trial

Molecular cancer therapeutics, 2023-12, Vol.22 (12_Supplement), p.LB_A10-LB_A10

2023

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Systemic DNA damage accumulation underin vivotumor growth can be inhibited by the antioxidant Tempol

Cancer letters, 2014-10, Vol.353 (2), p.248

2014

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sup 90^Y-daclizumab, an anti-CD25 monoclonal antibody, provided responses in 50% of patients with relapsed Hodgkin's lymphoma

Proceedings of the National Academy of Sciences - PNAS, 2015-10, Vol.112 (42), p.13045

2015

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90 Y-daclizumab, an anti-CD25 monoclonal antibody, provided responses in 50% of patients with relapsed Hodgkin’s lymphoma

Proceedings of the National Academy of Sciences - PNAS, 2015-10, Vol.112 (42), p.13045-13050

2015

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super( 90)Y-daclizumab, an anti-CD25 monoclonal antibody, provided responses in 50% of patients with relapsed Hodgkin's lymphoma

Proceedings of the National Academy of Sciences - PNAS, 2015-10, Vol.112 (42), p.13045-13045

2015

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A Phase I Study of DMS612, a Novel Bi-functional Alkylating Agent

Clinical cancer research, 2014-12, Vol.21 (4), p.721-729

2014

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Chromatin-Bound Cullin-Ring Ligases: Regulatory Roles in DNA Replication and Potential Targeting for Cancer Therapy

γH2AX and cancer

RepID-deficient cancer cells are sensitized to a drug targeting p97/VCP segregase

Telomere-dependent and telomere-independent origins of endogenous DNA damage in tumor cells

Switching DCAFs: Beyond substrate receptors

Histone yH2AX and Poly(ADP-Ribose) as Clinical Pharmacodynamic Biomarkers

γ-H2AX as a biomarker of DNA damage induced by ionizing radiation in human peripheral blood lymphocytes and artificial skin

Targeting Replication Stress and Chemotherapy Resistance with a Combination of Sacituzumab Govitecan and Berzosertib: A Phase I Clinical Trial

NADPH Oxidases NOXs and DUOXs as Putative Targets for Cancer Therapy

Acetalax (Oxyphenisatin Acetate, NSC 59687) and Bisacodyl Cause Oncosis in Triple-Negative Breast Cancer Cell Lines by Poisoning the Ion Exchange Membrane Protein TRPM4

Para-inflammation mediates systemic DNA damage in response to tumor growth

Preventing Excess Replication Origin Activation to Insure Genome Stability

Histone γH2AX and Poly(ADP-Ribose) as Clinical Pharmacodynamic Biomarkers

Examining the reliability of surrogate tissue predictability of drug efficacy in reconstituted human 3D skin models containing melanomas

Abstract 4774: Histone H2AX is a novel regulator of epithelial to mesenchymal transition

Q γ −H2AX, an analysis method for partial-body radiation exposure using γ-H2AX in non-human primate lymphocytes

Using gamma-H2AX and H2AX quantitative ELISA for monitoring DNA damage induced by chemotherapeutic agents and irradiation exposure

Abstract 4041: Wild-type p53-induced phosphatase 1 (Wip1) prevents cellular senescence at physiological oxygen levels by regulating DNA damage signaling during DNA replication

Use of the [gamma]-H2AX assay to monitor DNA damage and repair in translational cancer research

Abstract LB_A10: Efficacy of ATR inhibitor berzosertib plus topotecan or topotecan alone in patients with relapsed small cell lung cancer: A randomized clinical trial

Systemic DNA damage accumulation underin vivotumor growth can be inhibited by the antioxidant Tempol

sup 90^Y-daclizumab, an anti-CD25 monoclonal antibody, provided responses in 50% of patients with relapsed Hodgkin's lymphoma

90 Y-daclizumab, an anti-CD25 monoclonal antibody, provided responses in 50% of patients with relapsed Hodgkin’s lymphoma

super( 90)Y-daclizumab, an anti-CD25 monoclonal antibody, provided responses in 50% of patients with relapsed Hodgkin's lymphoma

A Phase I Study of DMS612, a Novel Bi-functional Alkylating Agent

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