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Abstract P6-02-09: Hyperglycaemia and fibronectin: The criminal partnership during breast cancer progression

Cancer research (Chicago, Ill.), 2017-02, Vol.77 (4_Supplement), p.P6-P6-02-09

2017

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Epithelial-to-mesenchymal transition in breast cancer: a role for insulin-like growth factor I and insulin-like growth factor-binding protein 3?

Breast cancer targets and therapy, 2015-01, Vol.7 (default), p.9-19

2015

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OP13Socio-economic inequalities in components of the neuroendocrine system (insulin-like growth factor-I and testosterone) among older adults: findings from the 1946 British Birth Cohort Study

Journal of epidemiology and community health (1979), 2014-09, Vol.68 (Suppl 1), p.A9-A10

2014

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Differential signalling mechanisms predisposing primary human skeletal muscle cells to altered proliferation and differentiation: roles of IGF-I and TNFα

Experimental cell research, 2004-03, Vol.294 (1), p.223-235

2004

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Insulin-like growth factor binding protein 3 modulates epidermal growth factor (EGF)-induced growth of breast epithelial cells by altering EGF receptor internalization

Breast cancer research : BCR, 2008-01, Vol.10 (S2), p.P40-P40, Article P40

2008

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Actions of IGF-I are differentially regulated by fatty acids in normal and breast cancer epithelial cells

Breast cancer research : BCR, 2008-01, Vol.10 (S2), p.P39-P39, Article P39

2008

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NIA0004Unsaturated fatty acids diminish the detrimental effects of saturated fatty acids on insulin signalling in human muscle by decreasing de novo ceramide production

Obesity reviews, 2006-09, Vol.7 (s2), p.37-37

2006

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NIA0004 Unsaturated fatty acids diminish the detrimental effects of saturated fatty acids on insulin signalling in human muscle by decreasing de novo ceramide production

Obesity reviews, 2006-09, Vol.7 (s2), p.37-37

2006

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Involvement of mitogen-activated protein kinase (MAPK and the insulin-like growth factor-i receptor (IGF-IR) in apoptosis

Growth hormone & IGF research, 1998-08, Vol.8 (4), p.333-333

1998

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Ceramide induces apoptosis in a human keratinocyte cell line and is accentuated by insulin-like growth factor binding protein 3

Journal of dermatological science, 1998-03, Vol.16, p.S154-S154

1998

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The insulin-like growth factor system in critical illness : pathophysiology and therapeutic potential

Clinical nutrition (Edinburgh, Scotland), 1995-12, Vol.14 (6), p.321-328

1995

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Influence of treatment with tamoxifen and change in tumor burden on the IGF-system in breast cancer patients

International journal of cancer, 1996, Vol.69 (4), p.335

1996

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Differential interactions of insulin-like growth factor binding proteins (1–6) with the ceramide-induced apoptotic signalling pathway in an IGF-independent manner

Growth hormone & IGF research, 1998-08, Vol.8 (4), p.339-339

1998

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Alterations in plasma IGF-I caused by hormone manipulation; of relevance to the mechanism of action and acquired resistance to endocrine treatment in breast cancer?

Endocrine-related cancer, 1995-03, Vol.2 (1), p.127-130

1995

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RE-ANALYSIS OF DATA IN TWO LANCET PAPERS ON THE EFFECT OF DIETARY SODIUM AND POTASSIUM ON BLOOD PRESSURE

The Lancet (British edition), 1981-12, Vol.318 (8260-8261), p.1384-1387

1981

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INACCURACIES IN TWO LANCET ARTICLES

The Lancet (British edition), 1981-08, Vol.318 (8241), p.311-311

1981

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Abstract P6-02-09: Hyperglycaemia and fibronectin: The criminal partnership during breast cancer progression

Epithelial-to-mesenchymal transition in breast cancer: a role for insulin-like growth factor I and insulin-like growth factor-binding protein 3?

OP13Socio-economic inequalities in components of the neuroendocrine system (insulin-like growth factor-I and testosterone) among older adults: findings from the 1946 British Birth Cohort Study

Differential signalling mechanisms predisposing primary human skeletal muscle cells to altered proliferation and differentiation: roles of IGF-I and TNFα

Insulin-like growth factor binding protein 3 modulates epidermal growth factor (EGF)-induced growth of breast epithelial cells by altering EGF receptor internalization

Actions of IGF-I are differentially regulated by fatty acids in normal and breast cancer epithelial cells

NIA0004Unsaturated fatty acids diminish the detrimental effects of saturated fatty acids on insulin signalling in human muscle by decreasing de novo ceramide production

NIA0004 Unsaturated fatty acids diminish the detrimental effects of saturated fatty acids on insulin signalling in human muscle by decreasing de novo ceramide production

Involvement of mitogen-activated protein kinase (MAPK and the insulin-like growth factor-i receptor (IGF-IR) in apoptosis

Ceramide induces apoptosis in a human keratinocyte cell line and is accentuated by insulin-like growth factor binding protein 3

The insulin-like growth factor system in critical illness : pathophysiology and therapeutic potential

Influence of treatment with tamoxifen and change in tumor burden on the IGF-system in breast cancer patients

Differential interactions of insulin-like growth factor binding proteins (1–6) with the ceramide-induced apoptotic signalling pathway in an IGF-independent manner

Alterations in plasma IGF-I caused by hormone manipulation; of relevance to the mechanism of action and acquired resistance to endocrine treatment in breast cancer?

RE-ANALYSIS OF DATA IN TWO LANCET PAPERS ON THE EFFECT OF DIETARY SODIUM AND POTASSIUM ON BLOOD PRESSURE

INACCURACIES IN TWO LANCET ARTICLES

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