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Proteolytic activation of proapoptotic kinase protein kinase Cδ by tumor necrosis factor α death receptor signaling in dopaminergic neurons during neuroinflammation

Journal of neuroinflammation, 2012-04, Vol.9 (1), p.82-82, Article 82

2012

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The Peptidyl-prolyl Isomerase Pin1 Up-regulation and Proapoptotic Function in Dopaminergic Neurons

The Journal of biological chemistry, 2013-07, Vol.288 (30), p.21955-21971

2013

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Environmental Neurotoxic Pesticide Exposure Induces Gut Inflammation and Enteric Neuronal Degeneration by Impairing Enteric Glial Mitochondrial Function in Pesticide Models of Parkinson’s Disease: Potential Relevance to Gut-Brain Axis Inflammation in Disease Pathogenesis

The international journal of biochemistry & cell biology, 2022-05, Vol.147, p.106225-106225

2022

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Protein kinase Cδ upregulation in microglia drives neuroinflammatory responses and dopaminergic neurodegeneration in experimental models of Parkinson's disease

Neurobiology of disease, 2016-09, Vol.93, p.96-114

2016

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Histone Hyperacetylation Up-regulates Protein Kinase Cδ in Dopaminergic Neurons to Induce Cell Death

The Journal of biological chemistry, 2014-12, Vol.289 (50), p.34743-34767

2014

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Rapid and Refined CD11b Magnetic Isolation of Primary Microglia with Enhanced Purity and Versatility

Journal of visualized experiments, 2017-04 (122)

2017

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Tumor Necrosis Factor-Like Weak Inducer of Apoptosis Enhances Activation of STAT3/NLRC4 Inflammasome Signaling Axis through PKC[delta] in Astrocytes: Implications for Parkinson's Disease

Cells (Basel, Switzerland), 2020-08, Vol.9 (8), p.1

2020

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Lysosomal dysfunction caused by the environmental neurotoxicant manganese increases exosome-mediated cell-to-cell transfer of α-synuclein by a prion-like mechanism

Neurotoxicology and teratology, 2015-05, Vol.49, p.109-109

2015

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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1

Autophagy, 2021-02, Vol.17 (1), p.1-382

2021

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Proteolytic activation of proapoptotic kinase protein kinase C[delta] by tumor necrosis factor [alpha] death receptor signaling in dopaminergic neurons during neuroinflammation

Journal of neuroinflammation, 2012-04, Vol.9 (1)

2012

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Neurotoxicity of Vanadium

Advances in neurobiology, 2017, Vol.18, p.287-301

2017

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Transcriptional Regulation of Pro-apoptotic Protein Kinase Cδ

The Journal of biological chemistry, 2011-06, Vol.286 (22), p.19840-19859

2011

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PKC-Delta Is a Major Molecular Target for Diverse Dopaminergic Toxicants: Implications for Mechanistic and Translational Neurotoxicology

Parkinsonism and the Environment, 2022, p.103-129

2022

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Methamphetamine-induced neurotoxicity linked to UPS dysfunction and autophagy related changes that can be modulated by PKCδ in dopaminergic neuronal cells

Neuroscience, 2012-03, Vol.210, p.308-332

2012

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Tyrosine phosphorylation regulates the proteolytic activation of protein kinase Cdelta in dopaminergic neuronal cells

The Journal of biological chemistry, 2005-08, Vol.280 (31), p.28721

2005

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Tyrosine Phosphorylation Regulates the Proteolytic Activation of Protein Kinase CÎ´ in Dopaminergic Neuronal Cells

The Journal of biological chemistry, 2005-08, Vol.280 (31), p.28721

2005

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Transcriptional regulation of pro-apoptotic protein kinase Cdelta: implications for oxidative stress-induced neuronal cell death

The Journal of biological chemistry, 2011-06, Vol.286 (22), p.19840

2011

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Chapter 55 - Cell signaling mechanisms in developmental neurotoxicity

Reproductive and Developmental Toxicology, 2022, p.1117-1127

Third Edition, 2022

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Protein kinase Cdelta is a key downstream mediator of manganese-induced apoptosis in dopaminergic neuronal cells

The Journal of pharmacology and experimental therapeutics, 2005-04, Vol.313 (1), p.46

2005

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Role of proteolytic activation of protein kinase Cdelta in oxidative stress-induced apoptosis

Antioxidants & redox signaling, 2003-10, Vol.5 (5), p.609-620

2003

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Wild-type alpha-synuclein interacts with pro-apoptotic proteins PKCdelta and BAD to protect dopaminergic neuronal cells against MPP+-induced apoptotic cell death

Brain research. Molecular brain research., 2005-09, Vol.139 (1), p.137

2005

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Chapter 54 - Cell Signaling Mechanisms in Developmental Neurotoxicity

Reproductive and Developmental Toxicology, 2017, p.1037-1049

Second Edition, 2017

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Investigations into the neurochemical mechanisms underlying 3,4-methylenedioxymethamphetamine (MDMA)-induced neurotoxicity: Assessment of the role of GABA and the dopamine transporter

Kanthasamy, Arthi

2001

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ProQuest

Protein Kinase C{delta} Negatively Regulates Tyrosine Hydroxylase Activity and Dopamine Synthesis by Enhancing Protein Phosphatase-2A Activity in Dopaminergic Neurons

The Journal of neuroscience, 2007-05, Vol.27 (20), p.5349

2007

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Quercetin

Nutraceuticals - Efficacy, Safety and Toxicity, 2016, p.1-1

Gupta Ramesh C

2016

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Proteolytic activation of proapoptotic kinase protein kinase Cδ by tumor necrosis factor α death receptor signaling in dopaminergic neurons during neuroinflammation

The Peptidyl-prolyl Isomerase Pin1 Up-regulation and Proapoptotic Function in Dopaminergic Neurons

Environmental Neurotoxic Pesticide Exposure Induces Gut Inflammation and Enteric Neuronal Degeneration by Impairing Enteric Glial Mitochondrial Function in Pesticide Models of Parkinson’s Disease: Potential Relevance to Gut-Brain Axis Inflammation in Disease Pathogenesis

Protein kinase Cδ upregulation in microglia drives neuroinflammatory responses and dopaminergic neurodegeneration in experimental models of Parkinson's disease

Histone Hyperacetylation Up-regulates Protein Kinase Cδ in Dopaminergic Neurons to Induce Cell Death

Rapid and Refined CD11b Magnetic Isolation of Primary Microglia with Enhanced Purity and Versatility

Tumor Necrosis Factor-Like Weak Inducer of Apoptosis Enhances Activation of STAT3/NLRC4 Inflammasome Signaling Axis through PKC[delta] in Astrocytes: Implications for Parkinson's Disease

Lysosomal dysfunction caused by the environmental neurotoxicant manganese increases exosome-mediated cell-to-cell transfer of α-synuclein by a prion-like mechanism

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1

Proteolytic activation of proapoptotic kinase protein kinase C[delta] by tumor necrosis factor [alpha] death receptor signaling in dopaminergic neurons during neuroinflammation

Neurotoxicity of Vanadium

Transcriptional Regulation of Pro-apoptotic Protein Kinase Cδ

PKC-Delta Is a Major Molecular Target for Diverse Dopaminergic Toxicants: Implications for Mechanistic and Translational Neurotoxicology

Methamphetamine-induced neurotoxicity linked to UPS dysfunction and autophagy related changes that can be modulated by PKCδ in dopaminergic neuronal cells

Tyrosine phosphorylation regulates the proteolytic activation of protein kinase Cdelta in dopaminergic neuronal cells

Tyrosine Phosphorylation Regulates the Proteolytic Activation of Protein Kinase CÎ´ in Dopaminergic Neuronal Cells

Transcriptional regulation of pro-apoptotic protein kinase Cdelta: implications for oxidative stress-induced neuronal cell death

Chapter 55 - Cell signaling mechanisms in developmental neurotoxicity

Protein kinase Cdelta is a key downstream mediator of manganese-induced apoptosis in dopaminergic neuronal cells

Role of proteolytic activation of protein kinase Cdelta in oxidative stress-induced apoptosis

Wild-type alpha-synuclein interacts with pro-apoptotic proteins PKCdelta and BAD to protect dopaminergic neuronal cells against MPP+-induced apoptotic cell death

Chapter 54 - Cell Signaling Mechanisms in Developmental Neurotoxicity

Investigations into the neurochemical mechanisms underlying 3,4-methylenedioxymethamphetamine (MDMA)-induced neurotoxicity: Assessment of the role of GABA and the dopamine transporter

Protein Kinase C{delta} Negatively Regulates Tyrosine Hydroxylase Activity and Dopamine Synthesis by Enhancing Protein Phosphatase-2A Activity in Dopaminergic Neurons

Quercetin

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