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Reversing tozasertib resistance in glioma through inhibition of pyruvate dehydrogenase kinases

Molecular oncology, 2022-01, Vol.16 (1), p.219-249

2022

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Targeting mitochondrial energetics reverses panobinostat- and marizomib-induced resistance in pediatric and adult high-grade gliomas

Molecular oncology, 2023-09, Vol.17 (9), p.1821-1843

2023

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Coadministration of sorafenib with rottlerin potently inhibits cell proliferation and migration in human malignant glioma cells

The Journal of pharmacology and experimental therapeutics, 2006-12, Vol.319 (3), p.1070-1080

2006

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Bortezomib-induced sensitization of malignant human glioma cells to vorinostat-induced apoptosis depends on reactive oxygen species production, mitochondrial dysfunction, Noxa upregulation, Mcl-1 cleavage, and DNA damage

Molecular carcinogenesis, 2013-02, Vol.52 (2), p.118-133

2013

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Enhanced Differentiation of Embryonic and Neural Stem Cells to Neuronal Fates on Laminin Peptides Doped Polypyrrole

Macromolecular bioscience, 2010-12, Vol.10 (12), p.1456-1464

2010

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A profile of 23 indian patients with purpura fulminans: A retrospective, descriptive study

Indian journal of dermatology, 2020-09, Vol.65 (5), p.381-387

2020

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Abrogation of mitogen-activated protein kinase and Akt signaling by vandetanib synergistically potentiates histone deacetylase inhibitor-induced apoptosis in human glioma cells

The Journal of pharmacology and experimental therapeutics, 2009-10, Vol.331 (1), p.327-337

2009

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Cucurbitacin-I inhibits Aurora kinase A, Aurora kinase B and survivin, induces defects in cell cycle progression and promotes ABT-737-induced cell death in a caspase-independent manner in malignant human glioma cells

Cancer biology & therapy, 2015-02, Vol.16 (2), p.233-243

2015

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Dinaciclib, a Cyclin-Dependent Kinase Inhibitor Promotes Proteasomal Degradation of Mcl-1 and Enhances ABT-737-Mediated Cell Death in Malignant Human Glioma Cell Lines

The Journal of pharmacology and experimental therapeutics, 2016-02, Vol.356 (2), p.354-365

2016

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Inhibition of phosphatidylinositol 3-kinase/AKT signaling by NVP-BKM120 promotes ABT-737-induced toxicity in a caspase-dependent manner through mitochondrial dysfunction and DNA damage response in established and primary cultured glioblastoma cells

The Journal of pharmacology and experimental therapeutics, 2014-07, Vol.350 (1), p.22-35

2014

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Survivin inhibitor YM155 induces mitochondrial dysfunction, autophagy, DNA damage and apoptosis in Bcl‐xL silenced glioma cell lines

Molecular carcinogenesis, 2017-04, Vol.56 (4), p.1251-1265

2017

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Targeting NAD + Biosynthesis Overcomes Panobinostat and Bortezomib-Induced Malignant Glioma Resistance

Molecular cancer research, 2020-07, Vol.18 (7), p.1004-1017

2020

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Abstract 3565: Dinaciclib, a CDK inhibitor promotes proteasomal degradation of Mcl-1 and enhances ABT-737 mediated cell death in malignant human glioma cell lines

Cancer research (Chicago, Ill.), 2016-07, Vol.76 (14_Supplement), p.3565-3565

2016

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Survivin inhibitor YM-155 sensitizes tumor necrosis factor- related apoptosis-inducing ligand-resistant glioma cells to apoptosis through Mcl-1 downregulation and by engaging the mitochondrial death pathway

The Journal of pharmacology and experimental therapeutics, 2013-08, Vol.346 (2), p.201-210

2013

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Mitochondrial dysfunction RAD51, and Ku80 proteolysis promote apoptotic effects of Dinaciclib in Bcl‐xL silenced cells

Molecular carcinogenesis, 2018-04, Vol.57 (4), p.469-482

2018

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ABT-737 synergizes with bortezomib to induce apoptosis, mediated by Bid cleavage, Bax activation, and mitochondrial dysfunction in an Akt-dependent context in malignant human glioma cell lines

The Journal of pharmacology and experimental therapeutics, 2012-06, Vol.341 (3), p.859-872

2012

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NVP-BKM120 potentiates apoptosis in tumor necrosis factor-related apoptosis-inducing ligand-resistant glioma cell lines via upregulation of Noxa and death receptor 5

International journal of oncology, 2015-08, Vol.47 (2), p.506-516

2015

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YM-155 potentiates the effect of ABT-737 in malignant human glioma cells via survivin and Mcl-1 downregulation in an EGFR-dependent context

Molecular cancer therapeutics, 2013-03, Vol.12 (3), p.326-338

2013

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Loss of MAT2A compromises methionine metabolism and represents a vulnerability in H3K27M mutant glioma by modulating the epigenome

Nature cancer, 2022-05, Vol.3 (5), p.629-648

2022

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Co-administration of ABT-737 and SAHA induces apoptosis, mediated by Noxa upregulation, Bax activation and mitochondrial dysfunction in PTEN-intact malignant human glioma cell lines

Journal of neuro-oncology, 2014-12, Vol.120 (3), p.459-472

2014

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Co-administration of NVP-AEW541 and dasatinib induces mitochondrial-mediated apoptosis through Bax activation in malignant human glioma cell lines

International journal of oncology, 2010-09, Vol.37 (3), p.633-643

2010

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Enzastaurin induces H2AX phosphorylation to regulate apoptosis via MAPK signalling in malignant glioma cells

European journal of cancer (1990), 2010-01, Vol.46 (2), p.412-419

2010

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The heat shock protein antagonist 17-AAG potentiates the activity of enzastaurin against malignant human glioma cells

Cancer letters, 2008-09, Vol.268 (1), p.46-55

2008

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Author Correction: Loss of MAT2A compromises methionine metabolism and represents a vulnerability in H3K27M mutant glioma by modulating the epigenome

Nature cancer, 2022-07, Vol.3 (7), p.899-899

2022

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AG490 influences UCN-01-induced cytotoxicity in Glioma cells in a p53-dependent fashion, correlating with effects on BAX cleavage and BAD phosphorylation

Cancer letters, 2007-11, Vol.257 (1), p.36-46

2007

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Reversing tozasertib resistance in glioma through inhibition of pyruvate dehydrogenase kinases

Targeting mitochondrial energetics reverses panobinostat- and marizomib-induced resistance in pediatric and adult high-grade gliomas

Coadministration of sorafenib with rottlerin potently inhibits cell proliferation and migration in human malignant glioma cells

Bortezomib-induced sensitization of malignant human glioma cells to vorinostat-induced apoptosis depends on reactive oxygen species production, mitochondrial dysfunction, Noxa upregulation, Mcl-1 cleavage, and DNA damage

Enhanced Differentiation of Embryonic and Neural Stem Cells to Neuronal Fates on Laminin Peptides Doped Polypyrrole

A profile of 23 indian patients with purpura fulminans: A retrospective, descriptive study

Abrogation of mitogen-activated protein kinase and Akt signaling by vandetanib synergistically potentiates histone deacetylase inhibitor-induced apoptosis in human glioma cells

Cucurbitacin-I inhibits Aurora kinase A, Aurora kinase B and survivin, induces defects in cell cycle progression and promotes ABT-737-induced cell death in a caspase-independent manner in malignant human glioma cells

Dinaciclib, a Cyclin-Dependent Kinase Inhibitor Promotes Proteasomal Degradation of Mcl-1 and Enhances ABT-737-Mediated Cell Death in Malignant Human Glioma Cell Lines

Inhibition of phosphatidylinositol 3-kinase/AKT signaling by NVP-BKM120 promotes ABT-737-induced toxicity in a caspase-dependent manner through mitochondrial dysfunction and DNA damage response in established and primary cultured glioblastoma cells

Survivin inhibitor YM155 induces mitochondrial dysfunction, autophagy, DNA damage and apoptosis in Bcl‐xL silenced glioma cell lines

Targeting NAD + Biosynthesis Overcomes Panobinostat and Bortezomib-Induced Malignant Glioma Resistance

Abstract 3565: Dinaciclib, a CDK inhibitor promotes proteasomal degradation of Mcl-1 and enhances ABT-737 mediated cell death in malignant human glioma cell lines

Survivin inhibitor YM-155 sensitizes tumor necrosis factor- related apoptosis-inducing ligand-resistant glioma cells to apoptosis through Mcl-1 downregulation and by engaging the mitochondrial death pathway

Mitochondrial dysfunction RAD51, and Ku80 proteolysis promote apoptotic effects of Dinaciclib in Bcl‐xL silenced cells

ABT-737 synergizes with bortezomib to induce apoptosis, mediated by Bid cleavage, Bax activation, and mitochondrial dysfunction in an Akt-dependent context in malignant human glioma cell lines

NVP-BKM120 potentiates apoptosis in tumor necrosis factor-related apoptosis-inducing ligand-resistant glioma cell lines via upregulation of Noxa and death receptor 5

YM-155 potentiates the effect of ABT-737 in malignant human glioma cells via survivin and Mcl-1 downregulation in an EGFR-dependent context

Loss of MAT2A compromises methionine metabolism and represents a vulnerability in H3K27M mutant glioma by modulating the epigenome

Co-administration of ABT-737 and SAHA induces apoptosis, mediated by Noxa upregulation, Bax activation and mitochondrial dysfunction in PTEN-intact malignant human glioma cell lines

Co-administration of NVP-AEW541 and dasatinib induces mitochondrial-mediated apoptosis through Bax activation in malignant human glioma cell lines

Enzastaurin induces H2AX phosphorylation to regulate apoptosis via MAPK signalling in malignant glioma cells

The heat shock protein antagonist 17-AAG potentiates the activity of enzastaurin against malignant human glioma cells

Author Correction: Loss of MAT2A compromises methionine metabolism and represents a vulnerability in H3K27M mutant glioma by modulating the epigenome

AG490 influences UCN-01-induced cytotoxicity in Glioma cells in a p53-dependent fashion, correlating with effects on BAX cleavage and BAD phosphorylation

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