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PyMT-Maclow: A novel, inducible, murine model for determining the role of CD68 positive cells in breast tumor development

PloS one, 2017-12, Vol.12 (12), p.e0188591-e0188591

2017

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Nanospan, an alternatively spliced isoform of sarcospan, localizes to the sarcoplasmic reticulum in skeletal muscle and is absent in limb girdle muscular dystrophy 2F

Skeletal muscle, 2017-06, Vol.7 (1), p.11-17, Article 11

2017

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Identifying Metrics for Measuring Research Culture at the University of Leeds

Exchanges (Coventry), 2024-08, Vol.11 (3), p.363-382

2024

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A targeted deletion of the C-terminal end of titin, including the titin kinase domain, impairs myofibrillogenesis

Journal of cell science, 2003-12, Vol.116 (Pt 23), p.4811-4819

2003

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ENU mutagenesis reveals a novel phenotype of reduced limb strength in mice lacking fibrillin 2

PloS one, 2010-02, Vol.5 (2), p.e9137

2010

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Altered Macrophage Polarization Induces Experimental Pulmonary Hypertension and Is Observed in Patients With Pulmonary Arterial Hypertension

Arteriosclerosis, thrombosis, and vascular biology, 2021-01, Vol.41 (1), p.430-445

2021

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Specific and Potent RNA Interference in Terminally Differentiated Myotubes

The Journal of biological chemistry, 2003-01, Vol.278 (2), p.934-939

2003

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In Vivo Fiber Optic Raman Spectroscopy of Muscle in Preclinical Models of Amyotrophic Lateral Sclerosis and Duchenne Muscular Dystrophy

ACS chemical neuroscience, 2021-05, Vol.12 (10), p.1768-1776

2021

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Preventing phosphorylation of dystroglycan ameliorates the dystrophic phenotype in mdx mouse

Human molecular genetics, 2012-10, Vol.21 (20), p.4508-4520

2012

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Disrupted mechanical stability of the dystrophin-glycoprotein complex causes severe muscular dystrophy in sarcospan transgenic mice

Journal of cell science, 2007-03, Vol.120 (Pt 6), p.996-1008

2007

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Wandering Myths: Transcultural Uses of Myth in the Ancient World

1st edition., 2018

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Generation of a novel mouse model for the inducible depletion of macrophages in vivo

Genesis (New York, N.Y. : 2000), 2013-01, Vol.51 (1), p.41-49

2013

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Nanospan, an alternatively spliced isoform of sarcospan, localizes to the sarcoplasmic reticulum in skeletal muscle and is absent in limb girdle muscular dystrophy 2F

Skeletal muscle, 2017, Vol.7, p.11

2017

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Structural and functional analysis of the sarcoglycan–sarcospan subcomplex

Experimental cell research, 2007-02, Vol.313 (4), p.639-651

2007

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Specific changes to the mechanism of cell locomotion induced by overexpression of beta-actin

Journal of cell science, 2001-04, Vol.114 (Pt 7), p.1367-1377

2001

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Abstract 15729: Reduction of CD68+ Cells Causes Spontaneous Pulmonary Arterial Hypertension in Mice

Circulation (New York, N.Y.), 2016-11, Vol.134 (Suppl_1 Suppl 1), p.A15729-A15729

2016

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Appearances can be deceiving: phenotypes of knockout mice

Briefings in functional genomics & proteomics, 2007-06, Vol.6 (2), p.91-103

2007

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Over-expression of Microspan, a novel component of the sarcoplasmic reticulum, causes severe muscle pathology with triad abnormalities

Journal of muscle research and cell motility, 2006-11, Vol.27 (8), p.545-558

2006

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Heterologous expression of wild-type and mutant beta-cardiac myosin changes the contractile kinetics of cultured mouse myotubes

The Journal of physiology, 2003-04, Vol.548 (Pt 1), p.167-174

2003

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Heterologous expression of wild‐type and mutant β‐cardiac myosin changes the contractile kinetics of cultured mouse myotubes

The Journal of physiology, 2003-04, Vol.548 (1), p.167-174

2003

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Heterologous expression of wild-type and mutant Î²-cardiac myosin changes the contractile kinetics of cultured mouse myotubes

The Journal of physiology, 2003-04, Vol.548 (1), p.167

2003

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N232S, G741R and D778G β-cardiac myosin mutants, implicated in familial hypertrophic cardiomyopathy, do not disrupt myofibrillar organisation in cultured myotubes

FEBS letters, 2000-12, Vol.486 (3), p.325-327

2000

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Modelling gene therapy for haemophilia

Miller, Gaynor

1995

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Introduction. Travelling Myths, Travelling Heroes

Wandering Myths, 2018, p.XXXIII-LIV

2018

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Preface. Wandering Myths: Transcultural Uses of Myth in the Ancient World

Wandering Myths, 2018, p.VII-XXXII

2018

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PyMT-Maclow: A novel, inducible, murine model for determining the role of CD68 positive cells in breast tumor development

Nanospan, an alternatively spliced isoform of sarcospan, localizes to the sarcoplasmic reticulum in skeletal muscle and is absent in limb girdle muscular dystrophy 2F

Identifying Metrics for Measuring Research Culture at the University of Leeds

A targeted deletion of the C-terminal end of titin, including the titin kinase domain, impairs myofibrillogenesis

ENU mutagenesis reveals a novel phenotype of reduced limb strength in mice lacking fibrillin 2

Altered Macrophage Polarization Induces Experimental Pulmonary Hypertension and Is Observed in Patients With Pulmonary Arterial Hypertension

Specific and Potent RNA Interference in Terminally Differentiated Myotubes

In Vivo Fiber Optic Raman Spectroscopy of Muscle in Preclinical Models of Amyotrophic Lateral Sclerosis and Duchenne Muscular Dystrophy

Preventing phosphorylation of dystroglycan ameliorates the dystrophic phenotype in mdx mouse

Disrupted mechanical stability of the dystrophin-glycoprotein complex causes severe muscular dystrophy in sarcospan transgenic mice

Wandering Myths: Transcultural Uses of Myth in the Ancient World

Generation of a novel mouse model for the inducible depletion of macrophages in vivo

Nanospan, an alternatively spliced isoform of sarcospan, localizes to the sarcoplasmic reticulum in skeletal muscle and is absent in limb girdle muscular dystrophy 2F

Structural and functional analysis of the sarcoglycan–sarcospan subcomplex

Specific changes to the mechanism of cell locomotion induced by overexpression of beta-actin

Abstract 15729: Reduction of CD68+ Cells Causes Spontaneous Pulmonary Arterial Hypertension in Mice

Appearances can be deceiving: phenotypes of knockout mice

Over-expression of Microspan, a novel component of the sarcoplasmic reticulum, causes severe muscle pathology with triad abnormalities

Heterologous expression of wild-type and mutant beta-cardiac myosin changes the contractile kinetics of cultured mouse myotubes

Heterologous expression of wild‐type and mutant β‐cardiac myosin changes the contractile kinetics of cultured mouse myotubes

Heterologous expression of wild-type and mutant Î²-cardiac myosin changes the contractile kinetics of cultured mouse myotubes

N232S, G741R and D778G β-cardiac myosin mutants, implicated in familial hypertrophic cardiomyopathy, do not disrupt myofibrillar organisation in cultured myotubes

Modelling gene therapy for haemophilia

Introduction. Travelling Myths, Travelling Heroes

Preface. Wandering Myths: Transcultural Uses of Myth in the Ancient World

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