UB Paderborn / Katalog / Suche / in CDI

1 – 25 von 144

Sortiert nach

Targeting Cellular Prion Protein Reverses Early Cognitive Deficits and Neurophysiological Dysfunction in Prion-Infected Mice

Neuron (Cambridge, Mass.), 2007-02, Vol.53 (3), p.325-335

2007

Link zum Volltext

Verfügbarkeit prüfen

Amyloid beta and tau pathology compared within different Alzheimer’s disease mouse models after inoculation with Alzheimer’s disease brain tissue

Alzheimer's & dementia, 2023-12, Vol.19 (S13)

2023

Link zum Volltext

Verfügbarkeit prüfen

The effect of prion protein expression on amyloid‐beta pathology

Alzheimer's & dementia, 2023-12, Vol.19 (S13)

2023

Link zum Volltext

Verfügbarkeit prüfen

Review. Lessons of kuru research: background to recent studies with some personal reflections

Philosophical transactions of the Royal Society of London. Series B. Biological sciences, 2008-11, Vol.363 (1510), p.3689

Collinge, John

2008

Link zum Volltext

Verfügbarkeit prüfen

Direct Observation of Prion Protein Fibril Elongation Kinetics

Biophysical journal, 2020-02, Vol.118 (3), p.484a-484a

2020

Link zum Volltext

Verfügbarkeit prüfen

Homozygous prion protein genotype predisposes to sporadic Creutzfeldt-Jakob disease

Nature (London), 1991-07, Vol.352 (6333), p.340-342

1991

Link zum Volltext

Verfügbarkeit prüfen

Population structure and migration in the Eastern Highlands of Papua New Guinea, a region impacted by the kuru epidemic

American journal of human genetics, 2024-04, Vol.111 (4), p.668-679

2024

Link zum Volltext

Verfügbarkeit prüfen

Prion protein monoclonal antibody (PRN100) therapy for Creutzfeldt–Jakob disease: evaluation of a first-in-human treatment programme

Lancet neurology, 2022-04, Vol.21 (4), p.342-354

2022

Link zum Volltext

Verfügbarkeit prüfen

Two mouse models of Alzheimer's disease accumulate amyloid at different rates and have distinct A[beta] oligomer profiles unaltered by ablation of cellular prion protein

PloS one, 2023-11, Vol.18 (11), p.e0294465

2023

Link zum Volltext

Verfügbarkeit prüfen

Loss of Residues 119–136, Including the First β-strand of Human Prion Protein, Generates an Aggregation-competent Partially “Open” Form

Journal of molecular biology, 2023-08, Vol.435 (15), p.168158-168158, Article 168158

2023

Link zum Volltext

Verfügbarkeit prüfen

Collinge et al. reply

Nature (London), 2016-07, Vol.535 (7611), p.E2-E3

2016

Link zum Volltext

Verfügbarkeit prüfen

Neuronal antibodies in patients with suspected or confirmed sporadic Creutzfeldt-Jakob disease: Table 1

Journal of neurology, neurosurgery and psychiatry, 2015-06, Vol.86 (6), p.692-694

2015

Link zum Volltext

Verfügbarkeit prüfen

Structural variation in amyloid- fibrils from Alzheimers disease clinical subtypes

Nature (London), 2017-01, Vol.541 (7636), p.217

2017

Link zum Volltext

Verfügbarkeit prüfen

Cell biology. The risk of prion zoonoses

Science (American Association for the Advancement of Science), 2012-01, Vol.335 (6067), p.411-413

Collinge, John

2012

Link zum Volltext

Verfügbarkeit prüfen

ESTIMATING CAUSALITY OF THE NOVEL PRION PROTEIN GENE VARIANT T201S

Journal of neurology, neurosurgery and psychiatry, 2016-12, Vol.87 (12), p.e1-e1

2016

Link zum Volltext

Verfügbarkeit prüfen

PROBING FTD GENETICS WITH NEXT-GENERATION SEQUENCING

Journal of neurology, neurosurgery and psychiatry, 2016-12, Vol.87 (12), p.e1-e1

2016

Link zum Volltext

Verfügbarkeit prüfen

J9 Probing huntington’s disease phenocopy syndromes with next-generation sequencing

Journal of neurology, neurosurgery and psychiatry, 2016-09, Vol.87 (Suppl 1), p.A78-A78

2016

Link zum Volltext

Verfügbarkeit prüfen

Amyloid-[Beta] transmission or unexamined bias?/Collinge et al. reply

Nature (London), 2016-09, Vol.537 (7620), p.E7

2016

Link zum Volltext

Verfügbarkeit prüfen

RARE STRUCTURAL GENETIC VARIATION IN HUMAN PRION DISEASES

Journal of neurology, neurosurgery and psychiatry, 2016-12, Vol.87 (12), p.e1-e1

2016

Link zum Volltext

Verfügbarkeit prüfen

Medicine. Prion strain mutation and selection

Science (American Association for the Advancement of Science), 2010-05, Vol.328 (5982), p.1111-1112

Collinge, John

2010

Link zum Volltext

Verfügbarkeit prüfen

S4-03-01 Symposia session

Neurobiology of aging, 2004-07, Vol.25, p.S70-S70

Collinge, John

2004

Link zum Volltext

Verfügbarkeit prüfen

Creutzfeld-Jakob disease--reply

JAMA neurology, 2013-12, Vol.70 (12), p.1589

2013

Link zum Volltext

Verfügbarkeit prüfen

Novel mutation of the PRNPgene of a clinical CJD case

BMC infectious diseases, 2006-11, Vol.6 (1), Article 169

2006

Link zum Volltext

Verfügbarkeit prüfen

The Residue 129 Polymorphism in Human Prion Protein Does Not Confer Susceptibility to Creutzfeldt-Jakob Disease by Altering the Structure or Global Stability of PrPC

The Journal of biological chemistry, 2004-07, Vol.279 (27), p.28515-28521

2004

Link zum Volltext

Verfügbarkeit prüfen

Transmission Properties of Human PrP 102L Prions Challenge the Relevance of Mouse Models of GSS: e1004953

PLoS pathogens, 2015-07, Vol.11 (7)

2015

Link zum Volltext

Verfügbarkeit prüfen

1 – 25 von 144

Suche als RSS-Feed

Aktive Filter

KollektionIngentaConnect Journals

KollektionNexis Uni

ThemaScience & Technology

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(116)
Mit Volltextzugriff
(117)

Publikationsform

1 weiteren anzeigen...

Weniger anzeigen...

Sprache

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 90 Tagen
(1)

Menü

Weitere Dienste

Einstellungen

Targeting Cellular Prion Protein Reverses Early Cognitive Deficits and Neurophysiological Dysfunction in Prion-Infected Mice

Amyloid beta and tau pathology compared within different Alzheimer’s disease mouse models after inoculation with Alzheimer’s disease brain tissue

The effect of prion protein expression on amyloid‐beta pathology

Review. Lessons of kuru research: background to recent studies with some personal reflections

Direct Observation of Prion Protein Fibril Elongation Kinetics

Homozygous prion protein genotype predisposes to sporadic Creutzfeldt-Jakob disease

Population structure and migration in the Eastern Highlands of Papua New Guinea, a region impacted by the kuru epidemic

Prion protein monoclonal antibody (PRN100) therapy for Creutzfeldt–Jakob disease: evaluation of a first-in-human treatment programme

Two mouse models of Alzheimer's disease accumulate amyloid at different rates and have distinct A[beta] oligomer profiles unaltered by ablation of cellular prion protein

Loss of Residues 119–136, Including the First β-strand of Human Prion Protein, Generates an Aggregation-competent Partially “Open” Form

Collinge et al. reply

Neuronal antibodies in patients with suspected or confirmed sporadic Creutzfeldt-Jakob disease: Table 1

Structural variation in amyloid- fibrils from Alzheimers disease clinical subtypes

Cell biology. The risk of prion zoonoses

ESTIMATING CAUSALITY OF THE NOVEL PRION PROTEIN GENE VARIANT T201S

PROBING FTD GENETICS WITH NEXT-GENERATION SEQUENCING

J9 Probing huntington’s disease phenocopy syndromes with next-generation sequencing

Amyloid-[Beta] transmission or unexamined bias?/Collinge et al. reply

RARE STRUCTURAL GENETIC VARIATION IN HUMAN PRION DISEASES

Medicine. Prion strain mutation and selection

S4-03-01 Symposia session

Creutzfeld-Jakob disease--reply

Novel mutation of the PRNPgene of a clinical CJD case

The Residue 129 Polymorphism in Human Prion Protein Does Not Confer Susceptibility to Creutzfeldt-Jakob Disease by Altering the Structure or Global Stability of PrPC

Transmission Properties of Human PrP 102L Prions Challenge the Relevance of Mouse Models of GSS: e1004953

Aktive Filter

Suchergebnisse filtern