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Important roles of reversible acetylation in the function of hematopoietic transcription factors

Journal of cellular and molecular medicine, 2005-01, Vol.9 (1), p.103-112

2005

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NOD-Like Receptor Protein 3 Inflammasome Priming and Activation in Barrett’s Epithelial Cells

Cellular and molecular gastroenterology and hepatology, 2016-07, Vol.2 (4), p.439-453

2016

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Reflux-Induced Inflammation and the Development of Barrett's Esophagus

Digestive diseases and sciences, 2022-05, Vol.55 (10), p.2699

2022

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528 – In Barret's Epithelial Cells, Nucleophosmin 1 Facilitates Repair of Oxidative Dna Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair Pathway

Gastroenterology (New York, N.Y. 1943), 2019-05, Vol.156 (6), p.S-107-S-107

2019

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Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H2-Receptor Antagonist

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-260-S-260

2018

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Su1054 - in Barrett's Epithelial Cells, Ursodeoxycholic Acid Hastens Repair of Oxidative DNA Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-469-S-469

2018

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Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H 2 -Receptor Antagonist

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-260

2018

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Abstract 5833: Genomic mechanisms of RET inhibitor resistance in RET-fusion positive NSCLC

Cancer research (Chicago, Ill.), 2024-03, Vol.84 (6_Supplement), p.5833-5833

2024

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166 – L-Type Calcium Channel Inhibitors (Verapamil and Diltiazem) Block Th2-Cytokine-Stimulated Eotaxin-3 Secretion in Esophageal Squamous Cells from Patients with Eosinophilic Esophagitis

Gastroenterology (New York, N.Y. 1943), 2019-05, Vol.156 (6), p.S-39-S-39

2019

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Sa1660 - Esophageal Adenocarcinoma (Eac) Exosomes Induce Features of Epithelial Mesenchymal Transition in Non-Neoplastic Barrett's Epithelial Cells: A Potential Mechanism for Carcinogenesis in the Barrett's Metaplasia Surrounding Eacs

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-345-S-345

2018

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Sa1669 - The Synthetic Flavagline Fl3 Protects Against Colitisassociated Cancer via Inhibition of Wnt/β-Catenin Signaling

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-347-S-348

2018

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Abstract 2405: Delineating molecular vulnerabilities of ATM mutant prostate cancers

Cancer research (Chicago, Ill.), 2023-04, Vol.83 (7_Supplement), p.2405-2405

2023

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332 - Reflux-Induced Epithelial Mesenchymal Transition: A Potential Mechanism for the Development of Sub-Squamous Intestinal Metaplasia in Barrett's Esophagus

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-83-S-83

2018

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Abstract 5857: Trastuzumab deruxtecan resistance can be mediated by payload resistance or secondary extracellular ERBB2 mutations but sensitivity to HER2 tyrosine kinase inhibitors is maintained

Cancer research (Chicago, Ill.), 2024-03, Vol.84 (6_Supplement), p.5857-5857

2024

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Abstract A020: Deciphering DNA damage repair in ATM mutant prostate cancers

Cancer research (Chicago, Ill.), 2024-01, Vol.84 (1_Supplement), p.A020-A020

2024

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580 - Epithelial-Mesenchymal Transition in Barrett's Esophagus: The Role of HIF-1α, microRNA-200, SP1, and MAZ

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S126-S126

2017

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104 - In Esophageal Fibroblasts, TH2 Cytokines Induce Pro-Fibrotic Periostin Expression that can be Blocked by JAK-STAT6 Inhibitors

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S29-S30

2017

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Sa1140 - Aspirin Prevents NF-kB Activation and CDX2 Expression Induced by Acid and Bile Salts in Esophageal Squamous Cells of Patients with Barrett’s Esophagus

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S233-S233

2017

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Mo1030 - In Non-Neoplastic Barrett's Epithelial Cells, Acid and Bile Salts Induce Epithelial Mesenchymal Transition (EMT) through a Hif-Mediated Reduction in Microrna-200a and -200b Expression

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S661-S661

2017

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Hypoxia-Inducible Factor-2α Plays a Role in Mediating Oesophagitis in Gastro-Oesophageal Reflux Disease

Gut, 2016-09, Vol.66 (9), p.1542-1554

2016

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In oesophageal squamous cells exposed to acidic bile salt medium, omeprazole inhibits IL-8 expression through effects on nuclear factor-[kappa]B and activator protein-1

Gut, 2014-07, Vol.63 (7), p.1042

2014

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Therapeutic and Chemopreventive Effects of Ursodeoxycholic Acid (UDCA): Potential Role in Patients with Barrett’s Esophagus

Gastro - Open Journal, 2015-10, Vol.1 (4), p.89-93

Huo, Xiaofang

2015

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Deoxycholic acid causes DNA damage while inducing apoptotic resistance through NE-[kappa]B activation in benign Barrett's epithelial cells

American journal of physiology: Gastrointestinal and liver physiology, 2011-08, Vol.301 (2), p.G278

2011

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NOD-Like Receptor Protein 3 Inflammasome Priming and Activation in Barrettâs Epithelial CellsSummary

Cellular and molecular gastroenterology and hepatology, 2016-07, Vol.2 (4), p.439-453

2016

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Acid and Bile Salt Induced CDX2 Expression Differs in Squamous Cells from Patients with and without Barrett’s Esophagus

Gastroenterology (New York, N.Y. 1943), 2010-03, Vol.139 (1), p.194-203.e1

2010

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Important roles of reversible acetylation in the function of hematopoietic transcription factors

NOD-Like Receptor Protein 3 Inflammasome Priming and Activation in Barrett’s Epithelial Cells

Reflux-Induced Inflammation and the Development of Barrett's Esophagus

528 – In Barret's Epithelial Cells, Nucleophosmin 1 Facilitates Repair of Oxidative Dna Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair Pathway

Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H2-Receptor Antagonist

Su1054 - in Barrett's Epithelial Cells, Ursodeoxycholic Acid Hastens Repair of Oxidative DNA Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair

Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H 2 -Receptor Antagonist

Abstract 5833: Genomic mechanisms of RET inhibitor resistance in RET-fusion positive NSCLC

166 – L-Type Calcium Channel Inhibitors (Verapamil and Diltiazem) Block Th2-Cytokine-Stimulated Eotaxin-3 Secretion in Esophageal Squamous Cells from Patients with Eosinophilic Esophagitis

Sa1660 - Esophageal Adenocarcinoma (Eac) Exosomes Induce Features of Epithelial Mesenchymal Transition in Non-Neoplastic Barrett's Epithelial Cells: A Potential Mechanism for Carcinogenesis in the Barrett's Metaplasia Surrounding Eacs

Sa1669 - The Synthetic Flavagline Fl3 Protects Against Colitisassociated Cancer via Inhibition of Wnt/β-Catenin Signaling

Abstract 2405: Delineating molecular vulnerabilities of ATM mutant prostate cancers

332 - Reflux-Induced Epithelial Mesenchymal Transition: A Potential Mechanism for the Development of Sub-Squamous Intestinal Metaplasia in Barrett's Esophagus

Abstract 5857: Trastuzumab deruxtecan resistance can be mediated by payload resistance or secondary extracellular ERBB2 mutations but sensitivity to HER2 tyrosine kinase inhibitors is maintained

Abstract A020: Deciphering DNA damage repair in ATM mutant prostate cancers

580 - Epithelial-Mesenchymal Transition in Barrett's Esophagus: The Role of HIF-1α, microRNA-200, SP1, and MAZ

104 - In Esophageal Fibroblasts, TH2 Cytokines Induce Pro-Fibrotic Periostin Expression that can be Blocked by JAK-STAT6 Inhibitors

Sa1140 - Aspirin Prevents NF-kB Activation and CDX2 Expression Induced by Acid and Bile Salts in Esophageal Squamous Cells of Patients with Barrett’s Esophagus

Mo1030 - In Non-Neoplastic Barrett's Epithelial Cells, Acid and Bile Salts Induce Epithelial Mesenchymal Transition (EMT) through a Hif-Mediated Reduction in Microrna-200a and -200b Expression

Hypoxia-Inducible Factor-2α Plays a Role in Mediating Oesophagitis in Gastro-Oesophageal Reflux Disease

In oesophageal squamous cells exposed to acidic bile salt medium, omeprazole inhibits IL-8 expression through effects on nuclear factor-[kappa]B and activator protein-1

Therapeutic and Chemopreventive Effects of Ursodeoxycholic Acid (UDCA): Potential Role in Patients with Barrett’s Esophagus

Deoxycholic acid causes DNA damage while inducing apoptotic resistance through NE-[kappa]B activation in benign Barrett's epithelial cells

NOD-Like Receptor Protein 3 Inflammasome Priming and Activation in Barrettâs Epithelial CellsSummary

Acid and Bile Salt Induced CDX2 Expression Differs in Squamous Cells from Patients with and without Barrett’s Esophagus

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