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Inhibition of p53 DNA binding by human papillomavirus E6 proteins

Journal of Virology, 1994-07, Vol.68 (7), p.4262-4273

1994

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The E8^E2C Protein, a Negative Regulator of Viral Transcription and Replication, Is Required for Extrachromosomal Maintenance of Human Papillomavirus Type 31 in Keratinocytes

Journal of Virology, 2000-02, Vol.74 (3), p.1178-1186

2000

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Differentiation-induced and constitutive transcription of human papillomavirus type 31b in cell lines containing viral episomes

Journal of Virology, 1992-10, Vol.66 (10), p.6070-6080

1992

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Biosynthesis of Human Papillomavirus from a Continuous Cell Line Upon Epithelial Differentiation

Science (American Association for the Advancement of Science), 1992-08, Vol.257 (5072), p.971-973

1992

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In vitro Synthesis of Oncogenic Human Papillomaviruses Requires Episomal Genomes for Differentiation-Dependent Late Expression

Proceedings of the National Academy of Sciences - PNAS, 1996-04, Vol.93 (7), p.3062-3067

1996

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Differentiation-Dependent Chromatin Rearrangement Coincides with Activation of Human Papillomavirus Type 31 Late Gene Expression

Journal of Virology, 2001-10, Vol.75 (20), p.10005-10013

2001

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Microarray Analysis Identifies Interferon-Inducible Genes and Stat-1 as Major Transcriptional Targets of Human Papillomavirus Type 31

Journal of Virology, 2000-05, Vol.74 (9), p.4174-4182

2000

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Cellular Changes Induced by Low-Risk Human Papillomavirus Type 11 in Keratinocytes That Stably Maintain Viral Episomes

Journal of Virology, 2001-08, Vol.75 (16), p.7564-7571

2001

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Human Papillomavirus Type 16 Alters Human Epithelial Cell Differentiation in vitro

Proceedings of the National Academy of Sciences - PNAS, 1988-10, Vol.85 (19), p.7169-7173

1988

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Amplification of human papillomavirus genomes in vitro is dependent on epithelial differentiation

Journal of Virology, 1991-05, Vol.65 (5), p.2254-2260

1991

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Telomerase Activation by Human Papillomavirus Type 16 E6 Protein: Induction of Human Telomerase Reverse Transcriptase Expression through Myc and GC-Rich Sp1 Binding Sites

Journal of Virology, 2001-06, Vol.75 (12), p.5559-5566

2001

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Early Polyadenylation Signals of Human Papillomavirus Type 31 Negatively Regulate Capsid Gene Expression

Journal of Virology, 2001-09, Vol.75 (17), p.8147-8157

2001

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Binding of the Human Papillomavirus E1 Origin-Recognition Protein is Regulated Through Complex Formation with the E2 Enhancer-Binding Protein

Proceedings of the National Academy of Sciences - PNAS, 1994-12, Vol.91 (26), p.12398-12402

1994

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Human papillomavirus type 18 E7 protein requires intact Cys-X-X-Cys motifs for zinc binding, dimerization, and transformation but not for Rb binding

Journal of Virology, 1993-06, Vol.67 (6), p.3142-3150

1993

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Initiation of DNA synthesis by human papillomavirus E7 oncoproteins is resistant to p21-mediated inhibition of cyclin E-cdk2 activity

Journal of Virology, 1997-07, Vol.71 (7), p.5570-5578

1997

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Identification of human papillomavirus type 18 transforming genes in immortalized and primary cells

Journal of Virology, 1989-03, Vol.63 (3), p.1247-1255

1989

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The E8E2C protein, a negative regulator of viral transcription and replication, is required for extrachromosomal maintenance of human papillomavirus type 31 in keratinocytes

Journal of virology, 2000-02, Vol.74 (3), p.1178-1186

2000

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A Keratinocyte-Specific Transcription Factor, KRF-1, Interacts with AP-1 to Activate Expression of Human Papillomavirus Type 18 in Squamous Epithelial Cells

Proceedings of the National Academy of Sciences - PNAS, 1991-10, Vol.88 (20), p.9102-9106

1991

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Induction of human papillomavirus type 18 late gene expression and genomic amplification in organotypic cultures from transfected DNA templates

Journal of Virology, 1997-09, Vol.71 (9), p.7068-7072

1997

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The E6-E7 region of human papillomavirus type 18 is sufficient for transformation of NIH 3T3 and rat-1 cells

Journal of Virology, 1987-11, Vol.61 (11), p.3635-3640

1987

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The 68-kilodalton E1 protein of bovine papillomavirus is a DNA binding phosphoprotein which associates with the E2 transcriptional activator in vitro

Journal of Virology, 1991-02, Vol.65 (2), p.649-656

1991

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Human Papillomavirus Type 31 Oncoproteins E6 and E7 Are Required for the Maintenance of Episomes during the Viral Life Cycle in Normal Human Keratinocytes

Proceedings of the National Academy of Sciences - PNAS, 1999-07, Vol.96 (15), p.8449-8454

1999

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Characterization of enhancer elements in the long terminal repeat of Moloney murine sarcoma virus

Journal of Virology, 1984-01, Vol.49 (1), p.183-189

1984

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Expression of AP1 during cellular differentiation determines human papillomavirus E6/E7 expression in stratified epithelial cells

Journal of general virology, 1997-02, Vol.78 (2), p.401-411

1997

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Human papillomavirus type 31b late gene expression is regulated through protein kinase C-mediated changes in RNA processing

Journal of Virology, 1995-06, Vol.69 (6), p.3381-3388

1995

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Inhibition of p53 DNA binding by human papillomavirus E6 proteins

The E8^E2C Protein, a Negative Regulator of Viral Transcription and Replication, Is Required for Extrachromosomal Maintenance of Human Papillomavirus Type 31 in Keratinocytes

Differentiation-induced and constitutive transcription of human papillomavirus type 31b in cell lines containing viral episomes

Biosynthesis of Human Papillomavirus from a Continuous Cell Line Upon Epithelial Differentiation

In vitro Synthesis of Oncogenic Human Papillomaviruses Requires Episomal Genomes for Differentiation-Dependent Late Expression

Differentiation-Dependent Chromatin Rearrangement Coincides with Activation of Human Papillomavirus Type 31 Late Gene Expression

Microarray Analysis Identifies Interferon-Inducible Genes and Stat-1 as Major Transcriptional Targets of Human Papillomavirus Type 31

Cellular Changes Induced by Low-Risk Human Papillomavirus Type 11 in Keratinocytes That Stably Maintain Viral Episomes

Human Papillomavirus Type 16 Alters Human Epithelial Cell Differentiation in vitro

Amplification of human papillomavirus genomes in vitro is dependent on epithelial differentiation

Telomerase Activation by Human Papillomavirus Type 16 E6 Protein: Induction of Human Telomerase Reverse Transcriptase Expression through Myc and GC-Rich Sp1 Binding Sites

Early Polyadenylation Signals of Human Papillomavirus Type 31 Negatively Regulate Capsid Gene Expression

Binding of the Human Papillomavirus E1 Origin-Recognition Protein is Regulated Through Complex Formation with the E2 Enhancer-Binding Protein

Human papillomavirus type 18 E7 protein requires intact Cys-X-X-Cys motifs for zinc binding, dimerization, and transformation but not for Rb binding

Initiation of DNA synthesis by human papillomavirus E7 oncoproteins is resistant to p21-mediated inhibition of cyclin E-cdk2 activity

Identification of human papillomavirus type 18 transforming genes in immortalized and primary cells

The E8E2C protein, a negative regulator of viral transcription and replication, is required for extrachromosomal maintenance of human papillomavirus type 31 in keratinocytes

A Keratinocyte-Specific Transcription Factor, KRF-1, Interacts with AP-1 to Activate Expression of Human Papillomavirus Type 18 in Squamous Epithelial Cells

Induction of human papillomavirus type 18 late gene expression and genomic amplification in organotypic cultures from transfected DNA templates

The E6-E7 region of human papillomavirus type 18 is sufficient for transformation of NIH 3T3 and rat-1 cells

The 68-kilodalton E1 protein of bovine papillomavirus is a DNA binding phosphoprotein which associates with the E2 transcriptional activator in vitro

Human Papillomavirus Type 31 Oncoproteins E6 and E7 Are Required for the Maintenance of Episomes during the Viral Life Cycle in Normal Human Keratinocytes

Characterization of enhancer elements in the long terminal repeat of Moloney murine sarcoma virus

Expression of AP1 during cellular differentiation determines human papillomavirus E6/E7 expression in stratified epithelial cells

Human papillomavirus type 31b late gene expression is regulated through protein kinase C-mediated changes in RNA processing

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