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Hydrogen Peroxide as a Damage Signal in Tissue Injury and Inflammation: Murderer, Mediator, or Messenger?

Journal of cellular biochemistry, 2014-03, Vol.115 (3), p.427-435

2014

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Structural and functional fine mapping of cysteines in mammalian glutaredoxin reveal their differential oxidation susceptibility

Nature communications, 2023-07, Vol.14 (1), p.4550-4550, Article 4550

2023

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Animal Models of Allergic Airways Disease: Where Are We and Where to Next?

Journal of cellular biochemistry, 2014-12, Vol.115 (12), p.2055-2064

2014

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The glutathione S-transferase Gstt1 drives survival and dissemination in metastases

Nature cell biology, 2024-06, Vol.26 (6), p.975

2024

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JNK inhibition reduces lung remodeling and pulmonary fibrotic systemic markers

Clinical and translational medicine, 2016-09, Vol.5 (1), p.1-18

2016

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Asbestos, Lung Cancers, and Mesotheliomas: From Molecular Approaches to Targeting Tumor Survival Pathways

American journal of respiratory cell and molecular biology, 2010-02, Vol.42 (2), p.133-139

2010

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Glutathionylation chemistry promotes interleukin‐1 beta‐mediated glycolytic reprogramming and pro‐inflammatory signaling in lung epithelial cells

The FASEB journal, 2021-05, Vol.35 (5), p.e21525-n/a

2021

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Emerging mechanisms of glutathione-dependent chemistry in biology and disease

Journal of cellular biochemistry, 2013-09, Vol.114 (9), p.1962-1968

2013

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Macrophage-intrinsic DUOX1 contributes to type 2 inflammation and mucus metaplasia during allergic airway disease

Mucosal immunology, 2022-05, Vol.15 (5), p.977-989

2022

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Pyruvate Kinase M2 Promotes Expression of Proinflammatory Mediators in House Dust Mite-Induced Allergic Airways Disease

The Journal of immunology (1950), 2020-02, Vol.204 (4), p.763-774

2020

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Endothelial cell‐specific redox gene modulation inhibits angiogenesis but promotes B16F0 tumor growth in mice

The FASEB journal, 2019-12, Vol.33 (12), p.14147-14158

2019

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Tumor necrosis factor‐alpha inhibits myogenic differentiation through MyoD protein destabilization

The FASEB journal, 2004-02, Vol.18 (2), p.227-237

2004

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c-Jun N-Terminal Kinase 1 Is Required for the Development of Pulmonary Fibrosis

American journal of respiratory cell and molecular biology, 2009-04, Vol.40 (4), p.422-432

2009

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Epithelial NF-κB orchestrates house dust mite-induced airway inflammation, hyperresponsiveness, and fibrotic remodeling

The Journal of immunology (1950), 2013-12, Vol.191 (12), p.5811-5821

2013

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Redox amplification of apoptosis by caspase-dependent cleavage of glutaredoxin 1 and S-glutathionylation of Fas

The Journal of cell biology, 2009-01, Vol.184 (2), p.241-252

2009

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The Duffy antigen receptor for chemokines regulates asthma pathophysiology

Clinical and experimental allergy, 2017-09, Vol.47 (9), p.1214-1222

2017

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Dynamic Redox Control of NF-κB through Glutaredoxin-Regulated S-Glutathionylation of Inhibitory κB Kinase β

Proceedings of the National Academy of Sciences - PNAS, 2006-08, Vol.103 (35), p.13086-13091

2006

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Fingerprint of the oxido-reductase ERO1: A protein disulfide bond producer and supporter of cancer

Biochimica et biophysica acta. Reviews on cancer, 2024-01, Vol.1879 (1), p.189027-189027, Article 189027

2024

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Glutaredoxin-1 Attenuates S-Glutathionylation of the Death Receptor Fas and Decreases Resolution of Pseudomonas aeruginosa Pneumonia

American journal of respiratory and critical care medicine, 2014-02, Vol.189 (4), p.463-474

2014

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Muscle Wasting and Impaired Muscle Regeneration in a Murine Model of Chronic Pulmonary Inflammation

American journal of respiratory cell and molecular biology, 2006-12, Vol.35 (6), p.689-696

2006

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Inhibition of Arginase Activity Enhances Inflammation in Mice with Allergic Airway Disease, in Association with Increases in Protein S-Nitrosylation and Tyrosine Nitration

The Journal of immunology (1950), 2008-09, Vol.181 (6), p.4255-4264

2008

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Transforming growth factor-β1 suppresses airway hyperresponsiveness in allergic airway disease

American journal of respiratory and critical care medicine, 2007-11, Vol.176 (10), p.974-982

2007

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Oxidative Processing of Latent Fas in the Endoplasmic Reticulum Controls the Strength of Apoptosis

Molecular and Cellular Biology, 2012-09, Vol.32 (17), p.3464-3478

2012

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Ablation of glutaredoxin-1 attenuates lipopolysaccharide-induced lung inflammation and alveolar macrophage activation

American journal of respiratory cell and molecular biology, 2011-04, Vol.44 (4), p.491-499

2011

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Oxidation of peroxiredoxin-4 induces oligomerization and promotes interaction with proteins governing protein folding and endoplasmic reticulum stress

The Journal of biological chemistry, 2021-01, Vol.296, p.100665, Article 100665

2021

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Hydrogen Peroxide as a Damage Signal in Tissue Injury and Inflammation: Murderer, Mediator, or Messenger?

Structural and functional fine mapping of cysteines in mammalian glutaredoxin reveal their differential oxidation susceptibility

Animal Models of Allergic Airways Disease: Where Are We and Where to Next?

The glutathione S-transferase Gstt1 drives survival and dissemination in metastases

JNK inhibition reduces lung remodeling and pulmonary fibrotic systemic markers

Asbestos, Lung Cancers, and Mesotheliomas: From Molecular Approaches to Targeting Tumor Survival Pathways

Glutathionylation chemistry promotes interleukin‐1 beta‐mediated glycolytic reprogramming and pro‐inflammatory signaling in lung epithelial cells

Emerging mechanisms of glutathione-dependent chemistry in biology and disease

Macrophage-intrinsic DUOX1 contributes to type 2 inflammation and mucus metaplasia during allergic airway disease

Pyruvate Kinase M2 Promotes Expression of Proinflammatory Mediators in House Dust Mite-Induced Allergic Airways Disease

Endothelial cell‐specific redox gene modulation inhibits angiogenesis but promotes B16F0 tumor growth in mice

Tumor necrosis factor‐alpha inhibits myogenic differentiation through MyoD protein destabilization

c-Jun N-Terminal Kinase 1 Is Required for the Development of Pulmonary Fibrosis

Epithelial NF-κB orchestrates house dust mite-induced airway inflammation, hyperresponsiveness, and fibrotic remodeling

Redox amplification of apoptosis by caspase-dependent cleavage of glutaredoxin 1 and S-glutathionylation of Fas

The Duffy antigen receptor for chemokines regulates asthma pathophysiology

Dynamic Redox Control of NF-κB through Glutaredoxin-Regulated S-Glutathionylation of Inhibitory κB Kinase β

Fingerprint of the oxido-reductase ERO1: A protein disulfide bond producer and supporter of cancer

Glutaredoxin-1 Attenuates S-Glutathionylation of the Death Receptor Fas and Decreases Resolution of Pseudomonas aeruginosa Pneumonia

Muscle Wasting and Impaired Muscle Regeneration in a Murine Model of Chronic Pulmonary Inflammation

Inhibition of Arginase Activity Enhances Inflammation in Mice with Allergic Airway Disease, in Association with Increases in Protein S-Nitrosylation and Tyrosine Nitration

Transforming growth factor-β1 suppresses airway hyperresponsiveness in allergic airway disease

Oxidative Processing of Latent Fas in the Endoplasmic Reticulum Controls the Strength of Apoptosis

Ablation of glutaredoxin-1 attenuates lipopolysaccharide-induced lung inflammation and alveolar macrophage activation

Oxidation of peroxiredoxin-4 induces oligomerization and promotes interaction with proteins governing protein folding and endoplasmic reticulum stress

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