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Immune checkpoints in T cells during oncogenic γ‐herpesvirus infections

Journal of medical virology, 2023-01, Vol.95 (1), p.e27840-n/a

Münz, Christian

2023

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Epstein-Barr virus as a leading cause of multiple sclerosis: mechanisms and implications

Nature reviews. Neurology, 2023-03, Vol.19 (3), p.160

2023

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Vaccination against the Epstein–Barr virus

Cellular and molecular life sciences : CMLS, 2020-11, Vol.77 (21), p.4315-4324

2020

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Latency and lytic replication in Epstein–Barr virus-associated oncogenesis

Nature reviews. Microbiology, 2019-11, Vol.17 (11), p.691-700

Münz, Christian

2019

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MicroRNAs of Epstein-Barr Virus Attenuate T-Cell-Mediated Immune Control In Vivo

mBio, 2019-01, Vol.10 (1)

2019

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Role of human natural killer cells during Epstein-Barr virus infection

Critical reviews in immunology, 2014, Vol.34 (6), p.501-507

Muenz, Christian

2014

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A distinct subpopulation of human NK cells restricts B cell transformation by EBV

The Journal of immunology (1950), 2013-11, Vol.191 (10), p.4989-4995

2013

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Immunodeficiencies that predispose to pathologies by human oncogenic y-herpesviruses

FEMS microbiology reviews, 2019-03, Vol.43 (2), p.181

2019

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Attenuated immune control of Epstein–Barr virus in humanized mice is associated with the multiple sclerosis risk factor HLA‐DR15

European journal of immunology, 2021-01, Vol.51 (1), p.64-75

2021

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Patients with Epstein Barr virus‐positive lymphomas have decreased CD4+ T‐cell responses to the viral nuclear antigen 1

International journal of cancer, 2008-12, Vol.123 (12), p.2824-2831

2008

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Heterologous prime-boost vaccination protects against EBV antigen-expressing lymphomas

The Journal of clinical investigation, 2019-05, Vol.129 (5), p.2071-2087

2019

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The autophagy machinery interacts with EBV capsids during viral envelope release

Proceedings of the National Academy of Sciences - PNAS, 2023-08, Vol.120 (34), p.e2211281120-e2211281120

2023

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Elevated Epstein-Barr virus-encoded nuclear antigen-1 immune responses predict conversion to multiple sclerosis

Annals of neurology, 2010-02, Vol.67 (2), p.159-169

2010

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MHC class II‐deficient mice allow functional human CD4+ T‐cell development

European journal of immunology, 2023-07, Vol.53 (7), p.e2250313-n/a

2023

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Two alternate strategies for innate immunity to Epstein-Barr virus: One using NK cells and the other NK cells and γδ T cells

The Journal of experimental medicine, 2017-06, Vol.214 (6), p.1827-1841

2017

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Cognate HLA absence in trans diminishes human NK cell education

The Journal of clinical investigation, 2016-10, Vol.126 (10), p.3772-3782

2016

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Infectious diseases in humanized mice

European journal of immunology, 2013-09, Vol.43 (9), p.2246-2254

2013

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Kaposi Sarcoma-Associated Herpesvirus Infection and Endemic Burkitt Lymphoma

The Journal of infectious diseases, 2020-06, Vol.222 (1), p.111-120

2020

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EBNA3B-deficient EBV promotes B cell lymphomagenesis in humanized mice and is found in human tumors

The Journal of clinical investigation, 2012-04, Vol.122 (4), p.1487-1502

2012

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Innate immune responses against Epstein Barr virus infection

Journal of leukocyte biology, 2013-12, Vol.94 (6), p.1185-1190

2013

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KSHV infection of B cells primes protective T cell responses in humanized mice

Nature communications, 2024-06, Vol.15 (1), p.4841-13

2024

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Two alternate strategies for innate immunity to Epstein-Barr virus: One using NK cells and the other NK cells and gamma delta T cells

The Journal of experimental medicine, 2017-06, Vol.214 (6), p.1827-1841

2017

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Role of the 2B4 Receptor in CD8 super(+) T-Cell-Dependent Immune Control of Epstein-Barr Virus Infection in Mice With Reconstituted Human Immune System Components

The Journal of infectious diseases, 2015-09, Vol.212 (5), p.803-807

2015

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Intrathymic epstein-barr virus infection is not a prominent feature of myasthenia gravis

Annals of neurology, 2011-09, Vol.70 (3), p.508-514

2011

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Epstein-Barr virus: Environmental trigger of multiple sclerosis?

Journal of virology, 2007-07, Vol.81 (13), p.6777-6784

2007

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Immune checkpoints in T cells during oncogenic γ‐herpesvirus infections

Epstein-Barr virus as a leading cause of multiple sclerosis: mechanisms and implications

Vaccination against the Epstein–Barr virus

Latency and lytic replication in Epstein–Barr virus-associated oncogenesis

MicroRNAs of Epstein-Barr Virus Attenuate T-Cell-Mediated Immune Control In Vivo

Role of human natural killer cells during Epstein-Barr virus infection

A distinct subpopulation of human NK cells restricts B cell transformation by EBV

Immunodeficiencies that predispose to pathologies by human oncogenic y-herpesviruses

Attenuated immune control of Epstein–Barr virus in humanized mice is associated with the multiple sclerosis risk factor HLA‐DR15

Patients with Epstein Barr virus‐positive lymphomas have decreased CD4+ T‐cell responses to the viral nuclear antigen 1

Heterologous prime-boost vaccination protects against EBV antigen-expressing lymphomas

The autophagy machinery interacts with EBV capsids during viral envelope release

Elevated Epstein-Barr virus-encoded nuclear antigen-1 immune responses predict conversion to multiple sclerosis

MHC class II‐deficient mice allow functional human CD4+ T‐cell development

Two alternate strategies for innate immunity to Epstein-Barr virus: One using NK cells and the other NK cells and γδ T cells

Cognate HLA absence in trans diminishes human NK cell education

Infectious diseases in humanized mice

Kaposi Sarcoma-Associated Herpesvirus Infection and Endemic Burkitt Lymphoma

EBNA3B-deficient EBV promotes B cell lymphomagenesis in humanized mice and is found in human tumors

Innate immune responses against Epstein Barr virus infection

KSHV infection of B cells primes protective T cell responses in humanized mice

Two alternate strategies for innate immunity to Epstein-Barr virus: One using NK cells and the other NK cells and gamma delta T cells

Role of the 2B4 Receptor in CD8 super(+) T-Cell-Dependent Immune Control of Epstein-Barr Virus Infection in Mice With Reconstituted Human Immune System Components

Intrathymic epstein-barr virus infection is not a prominent feature of myasthenia gravis

Epstein-Barr virus: Environmental trigger of multiple sclerosis?

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