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Contributions of inflammation and tumor microenvironment to neurofibroma tumorigenesis

The Journal of clinical investigation, 2018-07, Vol.128 (7), p.2848-2861

2018

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NF1 heterozygosity fosters de novo tumorigenesis but impairs malignant transformation

Nature communications, 2018-11, Vol.9 (1), p.5014-11, Article 5014

2018

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Translating current basic research into future therapies for neurofibromatosis type 1

British journal of cancer, 2020-07, Vol.123 (2), p.178-186

2020

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An interconnected hierarchical model of cell death regulation by the BCL-2 family

Nature cell biology, 2015-10, Vol.17 (10), p.1270-1281

2015

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Cells of Origin in the Embryonic Nerve Roots for NF1-Associated Plexiform Neurofibroma

Cancer cell, 2014-11, Vol.26 (5), p.695-706

2014

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Spatiotemporal Loss of NF1 in Schwann Cell Lineage Leads to Different Types of Cutaneous Neurofibroma Susceptible to Modification by the Hippo Pathway

Cancer discovery, 2019-01, Vol.9 (1), p.114-129

2019

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BET Bromodomain Inhibition Triggers Apoptosis of NF1-Associated Malignant Peripheral Nerve Sheath Tumors through Bim Induction

Cell reports (Cambridge), 2014-01, Vol.6 (1), p.81-92

2014

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Epithelial stem cell homeostasis in Meibomian gland development, dysfunction, and dry eye disease

JCI insight, 2021-10, Vol.6 (20)

2021

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Chemometric-Guided Exploration of Marine Anti-Neurofibroma Leads

Frontiers in Marine Science, 2022-07, Vol.9

2022

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Identification of hair shaft progenitors that create a niche for hair pigmentation

Genes & development, 2017-04, Vol.31 (8), p.744-756

2017

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Double-Stranded RNA Sensing Determines Epithelial Cell Identity

Journal of investigative dermatology, 2019-01, Vol.139 (1), p.17-19

2019

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Overcoming BET Inhibitor Resistance in Malignant Peripheral Nerve Sheath Tumors

Clinical cancer research, 2019-06, Vol.25 (11), p.3404-3416

2019

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Toll-Like Receptor 2 Mediates Alveolar Macrophage Response to Pneumocystis murina

Infection and Immunity, 2006-03, Vol.74 (3), p.1857-1864

2006

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Downregulation of PU.1 Leads to Decreased Expression of Dectin-1 in Alveolar Macrophages during Pneumocystis Pneumonia

Infection and Immunity, 2010-03, Vol.78 (3), p.1058-1065

2010

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Pneumocystis Mediates Overexpression of Antizyme Inhibitor Resulting in Increased Polyamine Levels and Apoptosis in Alveolar Macrophages

The Journal of biological chemistry, 2009-03, Vol.284 (12), p.8174-8184

2009

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The role of nerve microenvironment for neurofibroma development

Oncotarget, 2016-09, Vol.7 (38), p.61500-61508

2016

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Microarray studies on effects of Pneumocystis carinii infection on global gene expression in alveolar macrophages

BMC microbiology, 2010-04, Vol.10 (1), p.103-103, Article 103

2010

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Effects of Decreased Calmodulin Protein on the Survival Mechanisms of Alveolar Macrophages during Pneumocystis Pneumonia

Infection and Immunity, 2009-08, Vol.77 (8), p.3344-3354

2009

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Polyamine Transport as a Target for Treatment of Pneumocystis Pneumonia

Antimicrobial Agents and Chemotherapy, 2009-12, Vol.53 (12), p.5259-5264

2009

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Defective nitric oxide production by alveolar macrophages during Pneumocystis pneumonia

American journal of respiratory cell and molecular biology, 2011-04, Vol.44 (4), p.540-547

2011

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Toll-like Receptor 2 Knockout Reduces Lung Inflammation During Pneumocystis Pneumonia but has No Effect on Phagocytosis of Pneumocystis Organisms by Alveolar Macrophages

The Journal of eukaryotic microbiology, 2006-11, Vol.53 (s1), p.S132-S133

2006

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Inflammatory Cells are Sources of Polyamines that Induce Alveolar Macrophage to Undergo Apoptosis During Pneumocystis Pneumonia

The Journal of eukaryotic microbiology, 2006-11, Vol.53 (s1), p.S134-S135

2006

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Syndecan-1 Expression in the Lung during Pneumocystis Infection

The Journal of eukaryotic microbiology, 2006-11, Vol.53 (s1), p.S122-S123

2006

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Expression and Activation of Complement Protein and Alveolar Damage during Pneumocystis Pneumonia

The Journal of eukaryotic microbiology, 2006-11, Vol.53 (s1), p.S136-S137

2006

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Caspase-9 as a Target for Pneumocystis Pneumonia Therapy

The Journal of eukaryotic microbiology, 2006-11, Vol.53 (s1), p.S138-S139

2006

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Contributions of inflammation and tumor microenvironment to neurofibroma tumorigenesis

NF1 heterozygosity fosters de novo tumorigenesis but impairs malignant transformation

Translating current basic research into future therapies for neurofibromatosis type 1

An interconnected hierarchical model of cell death regulation by the BCL-2 family

Cells of Origin in the Embryonic Nerve Roots for NF1-Associated Plexiform Neurofibroma

Spatiotemporal Loss of NF1 in Schwann Cell Lineage Leads to Different Types of Cutaneous Neurofibroma Susceptible to Modification by the Hippo Pathway

BET Bromodomain Inhibition Triggers Apoptosis of NF1-Associated Malignant Peripheral Nerve Sheath Tumors through Bim Induction

Epithelial stem cell homeostasis in Meibomian gland development, dysfunction, and dry eye disease

Chemometric-Guided Exploration of Marine Anti-Neurofibroma Leads

Identification of hair shaft progenitors that create a niche for hair pigmentation

Double-Stranded RNA Sensing Determines Epithelial Cell Identity

Overcoming BET Inhibitor Resistance in Malignant Peripheral Nerve Sheath Tumors

Toll-Like Receptor 2 Mediates Alveolar Macrophage Response to Pneumocystis murina

Downregulation of PU.1 Leads to Decreased Expression of Dectin-1 in Alveolar Macrophages during Pneumocystis Pneumonia

Pneumocystis Mediates Overexpression of Antizyme Inhibitor Resulting in Increased Polyamine Levels and Apoptosis in Alveolar Macrophages

The role of nerve microenvironment for neurofibroma development

Microarray studies on effects of Pneumocystis carinii infection on global gene expression in alveolar macrophages

Effects of Decreased Calmodulin Protein on the Survival Mechanisms of Alveolar Macrophages during Pneumocystis Pneumonia

Polyamine Transport as a Target for Treatment of Pneumocystis Pneumonia

Defective nitric oxide production by alveolar macrophages during Pneumocystis pneumonia

Toll-like Receptor 2 Knockout Reduces Lung Inflammation During Pneumocystis Pneumonia but has No Effect on Phagocytosis of Pneumocystis Organisms by Alveolar Macrophages

Inflammatory Cells are Sources of Polyamines that Induce Alveolar Macrophage to Undergo Apoptosis During Pneumocystis Pneumonia

Syndecan-1 Expression in the Lung during Pneumocystis Infection

Expression and Activation of Complement Protein and Alveolar Damage during Pneumocystis Pneumonia

Caspase-9 as a Target for Pneumocystis Pneumonia Therapy

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