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Selective inhibitory effects of zinc on cell proliferation in esophageal squamous cell carcinoma through Orai1

The FASEB journal, 2018-01, Vol.32 (1), p.404-416

2018

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Omeprazole blocks STAT6 binding to the eotaxin-3 promoter in eosinophilic esophagitis cells

PloS one, 2012-11, Vol.7 (11), p.e50037

2012

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Gastroesophageal Reflux Might Cause Esophagitis Through a Cytokine-Mediated Mechanism Rather Than Caustic Acid Injury

Gastroenterology (New York, N.Y. 1943), 2009-11, Vol.137 (5), p.1776-1784

2009

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Acid burn or cytokine sizzle in the pathogenesis of heartburn?

Journal of gastroenterology and hepatology, 2013-03, Vol.28 (3), p.385-387

2013

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Important roles of reversible acetylation in the function of hematopoietic transcription factors

Journal of cellular and molecular medicine, 2005-01, Vol.9 (1), p.103-112

2005

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NOD-Like Receptor Protein 3 Inflammasome Priming and Activation in Barrett’s Epithelial Cells

Cellular and molecular gastroenterology and hepatology, 2016-07, Vol.2 (4), p.439-453

2016

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Ablation of EWS-FLI1 by USP9X inhibition suppresses cancer cell growth in Ewing sarcoma

Cancer letters, 2023-01, Vol.552, p.215984, Article 215984

2023

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Genomic mechanisms of RET inhibitor sensitivity and resistance in RET-fusion positive cancer cells

Journal of clinical oncology, 2023-06, Vol.41 (16_suppl), p.e21029-e21029

2023

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Association of Acute Gastroesophageal Reflux Disease With Esophageal Histologic Changes

JAMA : the journal of the American Medical Association, 2016-05, Vol.315 (19), p.2104

2016

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Reflux-Induced Inflammation and the Development of Barrett's Esophagus

Digestive diseases and sciences, 2022-05, Vol.55 (10), p.2699

2022

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528 – In Barret's Epithelial Cells, Nucleophosmin 1 Facilitates Repair of Oxidative Dna Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair Pathway

Gastroenterology (New York, N.Y. 1943), 2019-05, Vol.156 (6), p.S-107-S-107

2019

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Discovery of novel cell proliferation-enhancing gene by random siRNA library based combinatorial screening

Combinatorial chemistry & high throughput screening, 2010-11, Vol.13 (9), p.798

2010

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In Barrett's epithelial cells, weakly acidic bile salt solutions cause oxidative DNA damage with response and repair mediated by p38

American journal of physiology: Gastrointestinal and liver physiology, 2020-03, Vol.318 (3), p.G464-G478

2020

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Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H2-Receptor Antagonist

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-260-S-260

2018

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Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H 2 -Receptor Antagonist

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-260

2018

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Su1054 - in Barrett's Epithelial Cells, Ursodeoxycholic Acid Hastens Repair of Oxidative DNA Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-469-S-469

2018

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Mithramycin suppresses DNA damage repair via targeting androgen receptor in prostate cancer

Cancer letters, 2020-09, Vol.488, p.40-49

2020

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166 – L-Type Calcium Channel Inhibitors (Verapamil and Diltiazem) Block Th2-Cytokine-Stimulated Eotaxin-3 Secretion in Esophageal Squamous Cells from Patients with Eosinophilic Esophagitis

Gastroenterology (New York, N.Y. 1943), 2019-05, Vol.156 (6), p.S-39-S-39

2019

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Sa1669 - The Synthetic Flavagline Fl3 Protects Against Colitisassociated Cancer via Inhibition of Wnt/β-Catenin Signaling

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-347-S-348

2018

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Sa1660 - Esophageal Adenocarcinoma (Eac) Exosomes Induce Features of Epithelial Mesenchymal Transition in Non-Neoplastic Barrett's Epithelial Cells: A Potential Mechanism for Carcinogenesis in the Barrett's Metaplasia Surrounding Eacs

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-345-S-345

2018

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Abstract 2405: Delineating molecular vulnerabilities of ATM mutant prostate cancers

Cancer research (Chicago, Ill.), 2023-04, Vol.83 (7_Supplement), p.2405-2405

2023

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332 - Reflux-Induced Epithelial Mesenchymal Transition: A Potential Mechanism for the Development of Sub-Squamous Intestinal Metaplasia in Barrett's Esophagus

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-83-S-83

2018

Volltextzugriff (PDF)

580 - Epithelial-Mesenchymal Transition in Barrett's Esophagus: The Role of HIF-1α, microRNA-200, SP1, and MAZ

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S126-S126

2017

Volltextzugriff (PDF)

104 - In Esophageal Fibroblasts, TH2 Cytokines Induce Pro-Fibrotic Periostin Expression that can be Blocked by JAK-STAT6 Inhibitors

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S29-S30

2017

Volltextzugriff (PDF)

Mo1030 - In Non-Neoplastic Barrett's Epithelial Cells, Acid and Bile Salts Induce Epithelial Mesenchymal Transition (EMT) through a Hif-Mediated Reduction in Microrna-200a and -200b Expression

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S661-S661

2017

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Selective inhibitory effects of zinc on cell proliferation in esophageal squamous cell carcinoma through Orai1

Omeprazole blocks STAT6 binding to the eotaxin-3 promoter in eosinophilic esophagitis cells

Gastroesophageal Reflux Might Cause Esophagitis Through a Cytokine-Mediated Mechanism Rather Than Caustic Acid Injury

Acid burn or cytokine sizzle in the pathogenesis of heartburn?

Important roles of reversible acetylation in the function of hematopoietic transcription factors

NOD-Like Receptor Protein 3 Inflammasome Priming and Activation in Barrett’s Epithelial Cells

Ablation of EWS-FLI1 by USP9X inhibition suppresses cancer cell growth in Ewing sarcoma

Genomic mechanisms of RET inhibitor sensitivity and resistance in RET-fusion positive cancer cells

Association of Acute Gastroesophageal Reflux Disease With Esophageal Histologic Changes

Reflux-Induced Inflammation and the Development of Barrett's Esophagus

528 – In Barret's Epithelial Cells, Nucleophosmin 1 Facilitates Repair of Oxidative Dna Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair Pathway

Discovery of novel cell proliferation-enhancing gene by random siRNA library based combinatorial screening

In Barrett's epithelial cells, weakly acidic bile salt solutions cause oxidative DNA damage with response and repair mediated by p38

Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H2-Receptor Antagonist

Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H 2 -Receptor Antagonist

Su1054 - in Barrett's Epithelial Cells, Ursodeoxycholic Acid Hastens Repair of Oxidative DNA Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair

Mithramycin suppresses DNA damage repair via targeting androgen receptor in prostate cancer

166 – L-Type Calcium Channel Inhibitors (Verapamil and Diltiazem) Block Th2-Cytokine-Stimulated Eotaxin-3 Secretion in Esophageal Squamous Cells from Patients with Eosinophilic Esophagitis

Sa1669 - The Synthetic Flavagline Fl3 Protects Against Colitisassociated Cancer via Inhibition of Wnt/β-Catenin Signaling

Sa1660 - Esophageal Adenocarcinoma (Eac) Exosomes Induce Features of Epithelial Mesenchymal Transition in Non-Neoplastic Barrett's Epithelial Cells: A Potential Mechanism for Carcinogenesis in the Barrett's Metaplasia Surrounding Eacs

Abstract 2405: Delineating molecular vulnerabilities of ATM mutant prostate cancers

332 - Reflux-Induced Epithelial Mesenchymal Transition: A Potential Mechanism for the Development of Sub-Squamous Intestinal Metaplasia in Barrett's Esophagus

580 - Epithelial-Mesenchymal Transition in Barrett's Esophagus: The Role of HIF-1α, microRNA-200, SP1, and MAZ

104 - In Esophageal Fibroblasts, TH2 Cytokines Induce Pro-Fibrotic Periostin Expression that can be Blocked by JAK-STAT6 Inhibitors

Mo1030 - In Non-Neoplastic Barrett's Epithelial Cells, Acid and Bile Salts Induce Epithelial Mesenchymal Transition (EMT) through a Hif-Mediated Reduction in Microrna-200a and -200b Expression

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