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Important roles of reversible acetylation in the function of hematopoietic transcription factors

Journal of cellular and molecular medicine, 2005-01, Vol.9 (1), p.103-112

2005

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NOD-Like Receptor Protein 3 Inflammasome Priming and Activation in Barrett’s Epithelial Cells

Cellular and molecular gastroenterology and hepatology, 2016-07, Vol.2 (4), p.439-453

2016

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Genomic mechanisms of RET inhibitor sensitivity and resistance in RET-fusion positive cancer cells

Journal of clinical oncology, 2023-06, Vol.41 (16_suppl), p.e21029-e21029

2023

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Reflux-Induced Inflammation and the Development of Barrett's Esophagus

Digestive diseases and sciences, 2022-05, Vol.55 (10), p.2699

2022

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528 – In Barret's Epithelial Cells, Nucleophosmin 1 Facilitates Repair of Oxidative Dna Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair Pathway

Gastroenterology (New York, N.Y. 1943), 2019-05, Vol.156 (6), p.S-107-S-107

2019

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Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H2-Receptor Antagonist

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-260-S-260

2018

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Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H 2 -Receptor Antagonist

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-260

2018

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Su1054 - in Barrett's Epithelial Cells, Ursodeoxycholic Acid Hastens Repair of Oxidative DNA Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-469-S-469

2018

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166 – L-Type Calcium Channel Inhibitors (Verapamil and Diltiazem) Block Th2-Cytokine-Stimulated Eotaxin-3 Secretion in Esophageal Squamous Cells from Patients with Eosinophilic Esophagitis

Gastroenterology (New York, N.Y. 1943), 2019-05, Vol.156 (6), p.S-39-S-39

2019

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Sa1669 - The Synthetic Flavagline Fl3 Protects Against Colitisassociated Cancer via Inhibition of Wnt/β-Catenin Signaling

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-347-S-348

2018

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Sa1660 - Esophageal Adenocarcinoma (Eac) Exosomes Induce Features of Epithelial Mesenchymal Transition in Non-Neoplastic Barrett's Epithelial Cells: A Potential Mechanism for Carcinogenesis in the Barrett's Metaplasia Surrounding Eacs

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-345-S-345

2018

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Abstract 2405: Delineating molecular vulnerabilities of ATM mutant prostate cancers

Cancer research (Chicago, Ill.), 2023-04, Vol.83 (7_Supplement), p.2405-2405

2023

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332 - Reflux-Induced Epithelial Mesenchymal Transition: A Potential Mechanism for the Development of Sub-Squamous Intestinal Metaplasia in Barrett's Esophagus

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-83-S-83

2018

Volltextzugriff (PDF)

580 - Epithelial-Mesenchymal Transition in Barrett's Esophagus: The Role of HIF-1α, microRNA-200, SP1, and MAZ

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S126-S126

2017

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104 - In Esophageal Fibroblasts, TH2 Cytokines Induce Pro-Fibrotic Periostin Expression that can be Blocked by JAK-STAT6 Inhibitors

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S29-S30

2017

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Mo1030 - In Non-Neoplastic Barrett's Epithelial Cells, Acid and Bile Salts Induce Epithelial Mesenchymal Transition (EMT) through a Hif-Mediated Reduction in Microrna-200a and -200b Expression

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S661-S661

2017

Volltextzugriff (PDF)

In Esophageal Fibroblasts, TH2 Cytokines Induce Pro-Fibrotic Periostin Expression that can be Blocked by JAK-STAT6 Inhibitors

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S29-S30

2017

Volltextzugriff (PDF)

Sa1140 - Aspirin Prevents NF-kB Activation and CDX2 Expression Induced by Acid and Bile Salts in Esophageal Squamous Cells of Patients with Barrett’s Esophagus

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S233-S233

2017

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Hypoxia-Inducible Factor-2α Plays a Role in Mediating Oesophagitis in Gastro-Oesophageal Reflux Disease

Gut, 2016-09, Vol.66 (9), p.1542-1554

2016

Volltextzugriff (PDF)

622 Pathological Acid Reflux Stimulates Esophageal Epithelial Production of Hypoxia Inducible Factor (HIF)-2a and Its Target Pro-Inflammatory Molecules: A Role for HIFs in GERD Pathogenesis

Gastroenterology (New York, N.Y. 1943), 2016-04, Vol.150 (4), p.S128-S128

2016

Volltextzugriff (PDF)

69 Ruxolitinib, a Selective JAK1/2 Inhibitor, Blocks IL-13/STAT6 Signaling and Eotaxin-3 Expression in Esophageal Epithelial Cells and Fibroblasts: A Potential Agent to Treat Inflammation and Fibrosis in EoE

Gastroenterology (New York, N.Y. 1943), 2016-04, Vol.150 (4), p.S18-S18

2016

Volltextzugriff (PDF)

Epithelial-Mesenchymal Transition in Barrett's Esophagus: The Role of HIF-1α, microRNA-200, SP1, and MAZ

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S126-S126

2017

Volltextzugriff (PDF)

In Non-Neoplastic Barrett's Epithelial Cells, Acid and Bile Salts Induce Epithelial Mesenchymal Transition (EMT) through a Hif-Mediated Reduction in Microrna-200a and -200b Expression

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S661-S661

2017

Volltextzugriff (PDF)

Aspirin Prevents NF-kB Activation and CDX2 Expression Induced by Acid and Bile Salts in Esophageal Squamous Cells of Patients with Barrett’s Esophagus

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S233-S233

2017

Volltextzugriff (PDF)

In oesophageal squamous cells exposed to acidic bile salt medium, omeprazole inhibits IL-8 expression through effects on nuclear factor-[kappa]B and activator protein-1

Gut, 2014-07, Vol.63 (7), p.1042

2014

Volltextzugriff (PDF)

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Important roles of reversible acetylation in the function of hematopoietic transcription factors

NOD-Like Receptor Protein 3 Inflammasome Priming and Activation in Barrett’s Epithelial Cells

Genomic mechanisms of RET inhibitor sensitivity and resistance in RET-fusion positive cancer cells

Reflux-Induced Inflammation and the Development of Barrett's Esophagus

528 – In Barret's Epithelial Cells, Nucleophosmin 1 Facilitates Repair of Oxidative Dna Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair Pathway

Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H2-Receptor Antagonist

Sa1153 - Esophageal Squamous Cells from Eosinophilic Esophagitis Patients Express a Non-Gastric Proton Pump, and their Th2 Cytokine-Stimulated Secretion of Eotaxin-3 can be Blocked by a Potassium-Competitive Acid Blocker and a Histamine H 2 -Receptor Antagonist

Su1054 - in Barrett's Epithelial Cells, Ursodeoxycholic Acid Hastens Repair of Oxidative DNA Damage Caused by Acidic Bile Salts Through P38-Dependent Effects on the Base Excision Repair

166 – L-Type Calcium Channel Inhibitors (Verapamil and Diltiazem) Block Th2-Cytokine-Stimulated Eotaxin-3 Secretion in Esophageal Squamous Cells from Patients with Eosinophilic Esophagitis

Sa1669 - The Synthetic Flavagline Fl3 Protects Against Colitisassociated Cancer via Inhibition of Wnt/β-Catenin Signaling

Sa1660 - Esophageal Adenocarcinoma (Eac) Exosomes Induce Features of Epithelial Mesenchymal Transition in Non-Neoplastic Barrett's Epithelial Cells: A Potential Mechanism for Carcinogenesis in the Barrett's Metaplasia Surrounding Eacs

Abstract 2405: Delineating molecular vulnerabilities of ATM mutant prostate cancers

332 - Reflux-Induced Epithelial Mesenchymal Transition: A Potential Mechanism for the Development of Sub-Squamous Intestinal Metaplasia in Barrett's Esophagus

580 - Epithelial-Mesenchymal Transition in Barrett's Esophagus: The Role of HIF-1α, microRNA-200, SP1, and MAZ

104 - In Esophageal Fibroblasts, TH2 Cytokines Induce Pro-Fibrotic Periostin Expression that can be Blocked by JAK-STAT6 Inhibitors

Mo1030 - In Non-Neoplastic Barrett's Epithelial Cells, Acid and Bile Salts Induce Epithelial Mesenchymal Transition (EMT) through a Hif-Mediated Reduction in Microrna-200a and -200b Expression

In Esophageal Fibroblasts, TH2 Cytokines Induce Pro-Fibrotic Periostin Expression that can be Blocked by JAK-STAT6 Inhibitors

Sa1140 - Aspirin Prevents NF-kB Activation and CDX2 Expression Induced by Acid and Bile Salts in Esophageal Squamous Cells of Patients with Barrett’s Esophagus

Hypoxia-Inducible Factor-2α Plays a Role in Mediating Oesophagitis in Gastro-Oesophageal Reflux Disease

622 Pathological Acid Reflux Stimulates Esophageal Epithelial Production of Hypoxia Inducible Factor (HIF)-2a and Its Target Pro-Inflammatory Molecules: A Role for HIFs in GERD Pathogenesis

69 Ruxolitinib, a Selective JAK1/2 Inhibitor, Blocks IL-13/STAT6 Signaling and Eotaxin-3 Expression in Esophageal Epithelial Cells and Fibroblasts: A Potential Agent to Treat Inflammation and Fibrosis in EoE

Epithelial-Mesenchymal Transition in Barrett's Esophagus: The Role of HIF-1α, microRNA-200, SP1, and MAZ

In Non-Neoplastic Barrett's Epithelial Cells, Acid and Bile Salts Induce Epithelial Mesenchymal Transition (EMT) through a Hif-Mediated Reduction in Microrna-200a and -200b Expression

Aspirin Prevents NF-kB Activation and CDX2 Expression Induced by Acid and Bile Salts in Esophageal Squamous Cells of Patients with Barrett’s Esophagus

In oesophageal squamous cells exposed to acidic bile salt medium, omeprazole inhibits IL-8 expression through effects on nuclear factor-[kappa]B and activator protein-1

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