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Wilms' tumours: about tumour suppressor genes, an oncogene and a chameleon gene

Nature reviews. Cancer, 2011-02, Vol.11 (2), p.111

Huff, Vicki

2011

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Wt1 functions in ovarian follicle development by regulating granulosa cell differentiation

Human molecular genetics, 2014-01, Vol.23 (2), p.333-341

2014

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A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28 (KAP1), a gene critical in early renal development: A Children's Oncology Group study

PloS one, 2018-12, Vol.13 (12), p.e0208936-e0208936

2018

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Reprogramming of Sertoli cells to fetal-like Leydig cells by Wt1 ablation

Proceedings of the National Academy of Sciences - PNAS, 2015-03, Vol.112 (13), p.4003-4008

2015

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Wilms tumor genetics: Mutations in WT1, WTX, and CTNNB1 account for only about one-third of tumors

Genes chromosomes & cancer, 2008-06, Vol.47 (6), p.461-470

2008

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Children's Oncology Group's 2013 blueprint for research: Renal tumors

Pediatric blood & cancer, 2013-06, Vol.60 (6), p.994-1000

2013

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The Wilms tumor gene, Wt1, is critical for mouse spermatogenesis via regulation of sertoli cell polarity and is associated with non-obstructive azoospermia in humans

PLoS genetics, 2013-08, Vol.9 (8), p.e1003645-e1003645

2013

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The Wilms Tumor Gene, Wt1, Is Required for Sox9 Expression and Maintenance of Tubular Architecture in the Developing Testis

Proceedings of the National Academy of Sciences - PNAS, 2006-08, Vol.103 (32), p.11987-11992

2006

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Loss of Heterozygosity for Chromosomes 1p and 16q Is an Adverse Prognostic Factor in Favorable-Histology Wilms Tumor: A Report From the National Wilms Tumor Study Group

Journal of clinical oncology, 2005-10, Vol.23 (29), p.7312-7321

2005

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Wilm's tumor 1 promotes memory flexibility

Nature communications, 2019-08, Vol.10 (1), p.3756-18, Article 3756

2019

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Rare variant detection using family-based sequencing analysis

Proceedings of the National Academy of Sciences - PNAS, 2013-03, Vol.110 (10), p.3985-3990

2013

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WT1 Mutation and 11P15 Loss of Heterozygosity Predict Relapse in Very Low-Risk Wilms Tumors Treated With Surgery Alone: A Children's Oncology Group Study

Journal of clinical oncology, 2011-02, Vol.29 (6), p.698-703

2011

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Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors

Translational oncology, 2014-08, Vol.7 (4), p.484-492

2014

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Loss of Heterozygosity at 2q37 in Sporadic Wilms' Tumor: Putative Role for miR-562

Clinical cancer research, 2009-10, Vol.15 (19), p.5985-5992

2009

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Wilms’ Tumor Blastemal Stem Cells Dedifferentiate to Propagate the Tumor Bulk

Stem cell reports, 2014-07, Vol.3 (1), p.24-33

2014

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Familial Wilms tumor

American journal of medical genetics. Part C, Seminars in medical genetics, 2004-08, Vol.129C (1), p.29-34

2004

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Twenty-four new cases of WT1 germline mutations and review of the literature: Genotype/phenotype correlations for Wilms tumor development

American journal of medical genetics, 2004-06, Vol.127A (3), p.249-257

2004

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Treatment With Nephrectomy Only for Small, Stage I/Favorable Histology Wilms’ Tumor: A Report From the National Wilms’ Tumor Study Group

Journal of clinical oncology, 2001-09, Vol.19 (17), p.3719-3724

2001

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Wt1 directs the lineage specification of sertoli and granulosa cells by repressing Sf1 expression

Development (Cambridge), 2017-01, Vol.144 (1), p.44-53

2017

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β-Catenin Activation in a Novel Liver Progenitor Cell Type Is Sufficient to Cause Hepatocellular Carcinoma and Hepatoblastoma

Cancer research (Chicago, Ill.), 2014-08, Vol.74 (16), p.4515-4525

2014

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Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumour

Journal of medical genetics, 2016-06, Vol.53 (6), p.385-388

2016

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Wt1 negatively regulates beta-catenin signaling during testis development

Development (Cambridge), 2008-05, Vol.135 (10), p.1875-1885

2008

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Wt1 ablation and Igf2 upregulation in mice result in Wilms tumors with elevated ERK1/2 phosphorylation

The Journal of clinical investigation, 2011-01, Vol.121 (1), p.174-183

2011

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Wilms' tumours: about tumour suppressor genes, an oncogene and a chameleon gene

Wt1 functions in ovarian follicle development by regulating granulosa cell differentiation

A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28 (KAP1), a gene critical in early renal development: A Children's Oncology Group study

Reprogramming of Sertoli cells to fetal-like Leydig cells by Wt1 ablation

Wilms tumor genetics: Mutations in WT1, WTX, and CTNNB1 account for only about one-third of tumors

Children's Oncology Group's 2013 blueprint for research: Renal tumors

The Wilms tumor gene, Wt1, is critical for mouse spermatogenesis via regulation of sertoli cell polarity and is associated with non-obstructive azoospermia in humans

The Wilms Tumor Gene, Wt1, Is Required for Sox9 Expression and Maintenance of Tubular Architecture in the Developing Testis

Loss of Heterozygosity for Chromosomes 1p and 16q Is an Adverse Prognostic Factor in Favorable-Histology Wilms Tumor: A Report From the National Wilms Tumor Study Group

Wilm's tumor 1 promotes memory flexibility

Rare variant detection using family-based sequencing analysis

WT1 Mutation and 11P15 Loss of Heterozygosity Predict Relapse in Very Low-Risk Wilms Tumors Treated With Surgery Alone: A Children's Oncology Group Study

Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors

Loss of Heterozygosity at 2q37 in Sporadic Wilms' Tumor: Putative Role for miR-562

Wilms’ Tumor Blastemal Stem Cells Dedifferentiate to Propagate the Tumor Bulk

Familial Wilms tumor

Twenty-four new cases of WT1 germline mutations and review of the literature: Genotype/phenotype correlations for Wilms tumor development

Treatment With Nephrectomy Only for Small, Stage I/Favorable Histology Wilms’ Tumor: A Report From the National Wilms’ Tumor Study Group

Wt1 directs the lineage specification of sertoli and granulosa cells by repressing Sf1 expression

A Children's Oncology Group and TARGET initiative exploring the genetic landscape of Wilms tumor

β-Catenin Activation in a Novel Liver Progenitor Cell Type Is Sufficient to Cause Hepatocellular Carcinoma and Hepatoblastoma

Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumour

Wt1 negatively regulates beta-catenin signaling during testis development

Wt1 ablation and Igf2 upregulation in mice result in Wilms tumors with elevated ERK1/2 phosphorylation

Significance of TP53 Mutation in Wilms Tumors with Diffuse Anaplasia: A Report from the Children's Oncology Group

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