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Deacetylation of S6 kinase promotes high glucose–induced glomerular mesangial cell hypertrophy and matrix protein accumulation

The Journal of biological chemistry, 2019-06, Vol.294 (24), p.9440-9460

2019

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Bone Morphogenetic Protein-2 (BMP-2) Activates NFATc1 Transcription Factor via an Autoregulatory Loop Involving Smad/Akt/Ca2+ Signaling

The Journal of biological chemistry, 2016-01, Vol.291 (3), p.1148-1161

2016

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MicroRNA-214 Reduces Insulin-like Growth Factor-1 (IGF-1) Receptor Expression and Downstream mTORC1 Signaling in Renal Carcinoma Cells

The Journal of biological chemistry, 2016-07, Vol.291 (28), p.14662-14676

2016

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Mesangial Cell Hypertrophy by High Glucose Is Mediated by Downregulation of the Tumor Suppressor PTEN

Diabetes (New York, N.Y.), 2006-07, Vol.55 (7), p.2115-2125

2006

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Akt2 causes TGFβ-induced deptor downregulation facilitating mTOR to drive podocyte hypertrophy and matrix protein expression

PloS one, 2018-11, Vol.13 (11), p.e0207285-e0207285

2018

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microRNA-21 governs TORC1 activation in renal cancer cell proliferation and invasion

PloS one, 2012-06, Vol.7 (6), p.e37366

2012

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TGFβ-induced deptor suppression recruits mTORC1 and not mTORC2 to enhance collagen I (α2) gene expression

PloS one, 2014-10, Vol.9 (10), p.e109608

2014

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A positive feedback loop involving Erk5 and Akt turns on mesangial cell proliferation in response to PDGF

American Journal of Physiology: Cell Physiology, 2014-06, Vol.306 (11), p.C1089-C1100

2014

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Downregulation of catalase by reactive oxygen species via PI 3 kinase/Akt signaling in mesangial cells

Journal of cellular physiology, 2007-05, Vol.211 (2), p.457-467

2007

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TGFβ acts through PDGFRβ to activate mTORC1 via the Akt/PRAS40 axis and causes glomerular mesangial cell hypertrophy and matrix protein expression

The Journal of biological chemistry, 2020-10, Vol.295 (42), p.14262-14278

2020

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Oncoprotein DJ-1 interacts with mTOR complexes to effect transcription factor Hif1α-dependent expression of collagen I (α2) during renal fibrosis

The Journal of biological chemistry, 2022-09, Vol.298 (9), p.102246-102246, Article 102246

2022

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High Glucose-Induced Pyruvate Kinase M2 (PKM2) Tyrosine Phosphorylation Drives a Feedback Loop by PDGFRβ to Force mTORC1 Activation for Mesangial Cell (MC) Injury in Diabetic Nephropathy (DN): FR-PO343

Journal of the American Society of Nephrology, 2023-11, Vol.34 (11S), p.499-499

2023

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PTEN-Long Suppresses Proximal Tubular Epithelial Cell (PTEC) Injury in Diabetic Nephropathy (DN): FR-PO344

Journal of the American Society of Nephrology, 2023-11, Vol.34 (11S), p.499-499

2023

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A Signaling Module Linking DJ-1, PTEN, and PDGF Receptor β (PDGFR) Governs Activation of mTORC1 to Induce Proximal Tubular Epithelial Cell (PTEC) Injury in Diabetic Nephropathy (DN): TH-PO197

Journal of the American Society of Nephrology, 2022-11, Vol.33 (11S), p.103-103

2022

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MicroRNA-21 Orchestrates High Glucose-induced Signals to TOR Complex 1, Resulting in Renal Cell Pathology in Diabetes

The Journal of biological chemistry, 2011-07, Vol.286 (29), p.25586-25603

2011

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Reciprocal regulation of miR-214 and PTEN by high glucose regulates renal glomerular mesangial and proximal tubular epithelial cell hypertrophy and matrix expansion

American Journal of Physiology: Cell Physiology, 2017-10, Vol.313 (4), p.C430-C447

2017

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TGFβ instructs mTORC2 to activate PKCβII for increased TWIST1 expression in proximal tubular epithelial cell injury

FEBS letters, 2023-05, Vol.597 (9), p.1300-1316

2023

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High glucose increases miR‐214 to power a feedback loop involving PTEN and the Akt/mTORC1 signaling axis

FEBS letters, 2019-08, Vol.593 (16), p.2261-2272

2019

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PDGF receptor-β uses Akt/mTORC1 signaling node to promote high glucose-induced renal proximal tubular cell collagen I (α2) expression

American journal of physiology. Renal physiology, 2017-08, Vol.313 (2), p.F291-F307

2017

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High Glucose Forces a Positive Feedback Loop Connecting Akt Kinase and FoxO1 Transcription Factor to Activate mTORC1 Kinase for Mesangial Cell Hypertrophy and Matrix Protein Expression

The Journal of biological chemistry, 2014-11, Vol.289 (47), p.32703-32716

2014

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Hydrophobic motif site-phosphorylated protein kinase CβII between mTORC2 and Akt regulates high glucose-induced mesangial cell hypertrophy

American Journal of Physiology: Cell Physiology, 2016-04, Vol.310 (7), p.C583-C596

2016

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β 2 -Adrenergic receptor agonist induced hepatic steatosis in mice: modeling nonalcoholic fatty liver disease in hyperadrenergic states

American journal of physiology: endocrinology and metabolism, 2021-07, Vol.321 (1), p.E90-E104

2021

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β2-Adrenergic receptor agonist induced hepatic steatosis in mice: modeling nonalcoholic fatty liver disease in hyperadrenergic states

American journal of physiology: endocrinology and metabolism, 2021-07, Vol.321 (1), p.E90-E104

2021

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TGF[beta]-induced PI 3 kinase-dependent Mnk-1 activation is necessary for Ser-209 phosphorylation of eIF4E and mesangial cell hypertrophy

Journal of cellular physiology, 2013-07, Vol.228 (7), p.1617

2013

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c-Abl-dependent Molecular Circuitry Involving Smad5 and Phosphatidylinositol 3-Kinase Regulates Bone Morphogenetic Protein-2-induced Osteogenesis

The Journal of biological chemistry, 2013-08, Vol.288 (34), p.24503-24517

2013

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Deacetylation of S6 kinase promotes high glucose–induced glomerular mesangial cell hypertrophy and matrix protein accumulation

Bone Morphogenetic Protein-2 (BMP-2) Activates NFATc1 Transcription Factor via an Autoregulatory Loop Involving Smad/Akt/Ca2+ Signaling

MicroRNA-214 Reduces Insulin-like Growth Factor-1 (IGF-1) Receptor Expression and Downstream mTORC1 Signaling in Renal Carcinoma Cells

Mesangial Cell Hypertrophy by High Glucose Is Mediated by Downregulation of the Tumor Suppressor PTEN

Akt2 causes TGFβ-induced deptor downregulation facilitating mTOR to drive podocyte hypertrophy and matrix protein expression

microRNA-21 governs TORC1 activation in renal cancer cell proliferation and invasion

TGFβ-induced deptor suppression recruits mTORC1 and not mTORC2 to enhance collagen I (α2) gene expression

A positive feedback loop involving Erk5 and Akt turns on mesangial cell proliferation in response to PDGF

Downregulation of catalase by reactive oxygen species via PI 3 kinase/Akt signaling in mesangial cells

TGFβ acts through PDGFRβ to activate mTORC1 via the Akt/PRAS40 axis and causes glomerular mesangial cell hypertrophy and matrix protein expression

Oncoprotein DJ-1 interacts with mTOR complexes to effect transcription factor Hif1α-dependent expression of collagen I (α2) during renal fibrosis

High Glucose-Induced Pyruvate Kinase M2 (PKM2) Tyrosine Phosphorylation Drives a Feedback Loop by PDGFRβ to Force mTORC1 Activation for Mesangial Cell (MC) Injury in Diabetic Nephropathy (DN): FR-PO343

PTEN-Long Suppresses Proximal Tubular Epithelial Cell (PTEC) Injury in Diabetic Nephropathy (DN): FR-PO344

A Signaling Module Linking DJ-1, PTEN, and PDGF Receptor β (PDGFR) Governs Activation of mTORC1 to Induce Proximal Tubular Epithelial Cell (PTEC) Injury in Diabetic Nephropathy (DN): TH-PO197

MicroRNA-21 Orchestrates High Glucose-induced Signals to TOR Complex 1, Resulting in Renal Cell Pathology in Diabetes

Reciprocal regulation of miR-214 and PTEN by high glucose regulates renal glomerular mesangial and proximal tubular epithelial cell hypertrophy and matrix expansion

TGFβ instructs mTORC2 to activate PKCβII for increased TWIST1 expression in proximal tubular epithelial cell injury

High glucose increases miR‐214 to power a feedback loop involving PTEN and the Akt/mTORC1 signaling axis

PDGF receptor-β uses Akt/mTORC1 signaling node to promote high glucose-induced renal proximal tubular cell collagen I (α2) expression

High Glucose Forces a Positive Feedback Loop Connecting Akt Kinase and FoxO1 Transcription Factor to Activate mTORC1 Kinase for Mesangial Cell Hypertrophy and Matrix Protein Expression

Hydrophobic motif site-phosphorylated protein kinase CβII between mTORC2 and Akt regulates high glucose-induced mesangial cell hypertrophy

β 2 -Adrenergic receptor agonist induced hepatic steatosis in mice: modeling nonalcoholic fatty liver disease in hyperadrenergic states

β2-Adrenergic receptor agonist induced hepatic steatosis in mice: modeling nonalcoholic fatty liver disease in hyperadrenergic states

TGF[beta]-induced PI 3 kinase-dependent Mnk-1 activation is necessary for Ser-209 phosphorylation of eIF4E and mesangial cell hypertrophy

c-Abl-dependent Molecular Circuitry Involving Smad5 and Phosphatidylinositol 3-Kinase Regulates Bone Morphogenetic Protein-2-induced Osteogenesis

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