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Inhibiting NLRP3 inflammasome attenuates apoptosis in contrast-induced acute kidney injury through the upregulation of HIF1A and BNIP3-mediated mitophagy

Autophagy, 2021-10, Vol.17 (10), p.2975-2990

2021

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Dimerization of mitophagy receptor BNIP3L/NIX is essential for recruitment of autophagic machinery

Autophagy, 2021-05, Vol.17 (5), p.1232-1243

2021

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BNIP3L/NIX and FUNDC1-mediated mitophagy is required for mitochondrial network remodeling during cardiac progenitor cell differentiation

Autophagy, 2019-07, Vol.15 (7), p.1182-1198

2019

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BNIP3L/NIX-mediated mitophagy protects against ischemic brain injury independent of PARK2

Autophagy, 2017-10, Vol.13 (10), p.1754-1766

2017

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BNIP3L/NIX degradation leads to mitophagy deficiency in ischemic brains

Autophagy, 2021-08, Vol.17 (8), p.1934-1946

2021

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BNIP3L/Nix-induced mitochondrial fission, mitophagy, and impaired myocyte glucose uptake are abrogated by PRKA/PKA phosphorylation

Autophagy, 2021-09, Vol.17 (9), p.2257-2272

2021

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Hypoxia-Induced Autophagy Is Mediated through Hypoxia-Inducible Factor Induction of BNIP3 and BNIP3L via Their BH3 Domains

Molecular and Cellular Biology, 2009-05, Vol.29 (10), p.2570-2581

2009

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BNIP3-dependent mitophagy promotes cytosolic localization of LC3B and metabolic homeostasis in the liver

Autophagy, 2021-11, Vol.17 (11), p.3530-3546

2021

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BNIP3L/NIX-dependent mitophagy regulates cell differentiation via metabolic reprogramming

Autophagy, 2018-05, Vol.14 (5), p.915-917

2018

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BNIP3L-mediated mitophagy is required for mitochondrial remodeling during the differentiation of optic nerve oligodendrocytes

Autophagy, 2021-10, Vol.17 (10), p.3140-3159

2021

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Baicalin relieves hypoxia-aroused H9c2 cell apoptosis by activating Nrf2/HO-1-mediated HIF1α/BNIP3 pathway

Artificial cells, nanomedicine, and biotechnology, 2019-12, Vol.47 (1), p.3657-3663

2019

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The mitophagy receptor BNIP3 is critical for the regulation of metabolic homeostasis and mitochondrial function in the nucleus pulposus cells of the intervertebral disc

Autophagy, 2023-06, Vol.19 (6), p.1821-1843

2023

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BNip3 Regulates Mitochondrial Function and Lipid Metabolism in the Liver

Molecular and Cellular Biology, 2012-07, Vol.32 (13), p.2570-2584

2012

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Tumor suppressor functions of BNIP3 and mitophagy

Autophagy, 2015, Vol.11 (10), p.1937-1938

2015

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A plant nonenveloped double-stranded RNA virus activates and co-opts BNIP3-mediated mitophagy to promote persistent infection in its insect vector

Autophagy, 2023-02, Vol.19 (2), p.616-631

2023

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BNIP3L-dependent mitophagy accounts for mitochondrial clearance during 3 factors-induced somatic cell reprogramming

Autophagy, 2017-09, Vol.13 (9), p.1543-1555

2017

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An unexpected journey for BNIP3

Autophagy, 2024-06, Vol.20 (6), p.1447-1448

2024

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Combining biomarkers of BNIP3 L, S100B, NSE, and accessible measures to predict sepsis-associated encephalopathy: a prospective observational study

Current medical research and opinion, 2024-04, Vol.40 (4), p.575-582

2024

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Bnip3-mediated mitochondrial autophagy is independent of the mitochondrial permeability transition pore

Autophagy, 2010-10, Vol.6 (7), p.855-862

2010

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Hypoxia-induced BNIP3 facilitates the progression and metastasis of uveal melanoma by driving metabolic reprogramming

Autophagy, 2024-09, p.1-19

2024

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Mir221- and Mir222 -enriched adsc-exosomes mitigate PM exposure-exacerbated cardiac ischemia-reperfusion injury through the modulation of the BNIP3-MAP1LC3B-BBC3/PUMA pathway

Autophagy, 2024-09, p.1-20

2024

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PGAM5 interacts with and maintains BNIP3 to license cancer-associated muscle wasting

Autophagy, 2024-06, p.1-16

2024

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Excessive BNIP3- and BNIP3L-dependent mitophagy underlies the pathogenesis of FBXL4-mutated mitochondrial DNA depletion syndrome

Autophagy, 2024-02, Vol.20 (2), p.460-462

2024

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BNIP3 Is an RB/E2F Target Gene Required for Hypoxia-Induced Autophagy

Molecular and Cellular Biology, 2007-09, Vol.27 (17), p.6229-6242

2007

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Humanin inhibits lymphatic endothelial cells dysfunction to alleviate myocardial infarction-reperfusion injury via BNIP3-mediated mitophagy

Free radical research, 2024-03, Vol.58 (3), p.180-193

2024

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Inhibiting NLRP3 inflammasome attenuates apoptosis in contrast-induced acute kidney injury through the upregulation of HIF1A and BNIP3-mediated mitophagy

Dimerization of mitophagy receptor BNIP3L/NIX is essential for recruitment of autophagic machinery

BNIP3L/NIX and FUNDC1-mediated mitophagy is required for mitochondrial network remodeling during cardiac progenitor cell differentiation

BNIP3L/NIX-mediated mitophagy protects against ischemic brain injury independent of PARK2

BNIP3L/NIX degradation leads to mitophagy deficiency in ischemic brains

BNIP3L/Nix-induced mitochondrial fission, mitophagy, and impaired myocyte glucose uptake are abrogated by PRKA/PKA phosphorylation

Hypoxia-Induced Autophagy Is Mediated through Hypoxia-Inducible Factor Induction of BNIP3 and BNIP3L via Their BH3 Domains

BNIP3-dependent mitophagy promotes cytosolic localization of LC3B and metabolic homeostasis in the liver

BNIP3L/NIX-dependent mitophagy regulates cell differentiation via metabolic reprogramming

BNIP3L-mediated mitophagy is required for mitochondrial remodeling during the differentiation of optic nerve oligodendrocytes

Baicalin relieves hypoxia-aroused H9c2 cell apoptosis by activating Nrf2/HO-1-mediated HIF1α/BNIP3 pathway

The mitophagy receptor BNIP3 is critical for the regulation of metabolic homeostasis and mitochondrial function in the nucleus pulposus cells of the intervertebral disc

BNip3 Regulates Mitochondrial Function and Lipid Metabolism in the Liver

Tumor suppressor functions of BNIP3 and mitophagy

A plant nonenveloped double-stranded RNA virus activates and co-opts BNIP3-mediated mitophagy to promote persistent infection in its insect vector

BNIP3L-dependent mitophagy accounts for mitochondrial clearance during 3 factors-induced somatic cell reprogramming

An unexpected journey for BNIP3

Combining biomarkers of BNIP3 L, S100B, NSE, and accessible measures to predict sepsis-associated encephalopathy: a prospective observational study

Bnip3-mediated mitochondrial autophagy is independent of the mitochondrial permeability transition pore

Hypoxia-induced BNIP3 facilitates the progression and metastasis of uveal melanoma by driving metabolic reprogramming

Mir221- and Mir222 -enriched adsc-exosomes mitigate PM exposure-exacerbated cardiac ischemia-reperfusion injury through the modulation of the BNIP3-MAP1LC3B-BBC3/PUMA pathway

PGAM5 interacts with and maintains BNIP3 to license cancer-associated muscle wasting

Excessive BNIP3- and BNIP3L-dependent mitophagy underlies the pathogenesis of FBXL4-mutated mitochondrial DNA depletion syndrome

BNIP3 Is an RB/E2F Target Gene Required for Hypoxia-Induced Autophagy

Humanin inhibits lymphatic endothelial cells dysfunction to alleviate myocardial infarction-reperfusion injury via BNIP3-mediated mitophagy

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