Biomedical and environmental sciences, 2013-07, Vol.26 (7), p.625-628
2013
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- Autor(en) / Beteiligte
- Titel
- The Mechanism of Acute Lung Injury Induced by Nickel Carbonyl in Rats
- Ist Teil von
- Biomedical and environmental sciences, 2013-07, Vol.26 (7), p.625-628
- Ort / Verlag
- China: Elsevier B.V
- Erscheinungsjahr
- 2013
- Quelle
- MEDLINE
- Beschreibungen/Notizen
- Nickel carbonyl is a highly toxic metal compound produced from the reaction that occurs between nickel and carbon monoxide under pressure. As previously reported, nickel carbonyl can cause acute aspiration pneumonia, and animal experiments showed it was toxic to animal lung, liver, brain, and other vital organs[1]. However, few studies have investigated nickel carbonyl poisoning in humans.
- Sprache
- Englisch
- Identifikatoren
- ISSN: 0895-3988eISSN: 2214-0190DOI: 10.3967/0895-3988.2013.07.018Titel-ID: cdi_wanfang_journals_bes201307018
- Format
- –
- Schlagworte
- Acute Lung Injury - chemically induced, Acute Lung Injury - metabolism, Acute Lung Injury - pathology, Animals, CDC2 Protein Kinase - genetics, CDC2 Protein Kinase - metabolism, Cell Cycle - drug effects, Checkpoint Kinase 1, Female, Lung - metabolism, Lung - pathology, Lung - ultrastructure, Male, Malondialdehyde - metabolism, Microscopy, Electron, Transmission, Organometallic Compounds - toxicity, Oxidative Stress, Protein Kinases - genetics, Protein Kinases - metabolism, Rats, Rats, Sprague-Dawley, RNA, Messenger - metabolism, 一氧化碳, 动物实验, 大鼠, 急性, 损伤机制, 羰基镍, 肺炎, 金属化合物