Details

Autor(en) / Beteiligte

• Longhitano, Y
• Candelli, M
• Muir, H A
• Audo, A
• Meda, S
• Dennet Lumb, P
• Marshall Berger, J
• Pignataro, G
• Cicchinelli, S
• Zanza, C
• Franceschi, F

Titel

Non-infarctual ST elevation and acute cardiopulmonary failure in carbon monoxide poisoning: a case report

Ist Teil von

• European review for medical and pharmacological sciences, 2019-08, Vol.23 (16), p.7128

Ort / Verlag

Italy

Erscheinungsjahr

2019

Quelle

MEDLINE

Beschreibungen/Notizen

• Carbon monoxide is produced by the incomplete combustion of organic fuel. In the United States, it is responsible for about 500 deaths annually. Increased carboxyhemoglobin concentration and hypoxia disrupt cardiac myocyte integrity and cause dysrhythmias, acute cardiac failure and coronary artery disease. We described a case of a patient with CO-poisoning and ST elevation at ECG precordial leads who developed severe transient heart failure. A 57-year-old man was admitted to the emergency department for acute carbon monoxide poisoning that led to respiratory and cardiac failure. The electrocardiogram showed ST elevation in precordial leads, but the coronary angiography was normal. The patient was successfully treated and discharged. Three days later he was readmitted for similar symptoms and subsequently died. We hypothesize that the ECG findings were related to transient coronary vasospasm due to CO poisoning and that acute respiratory and cardiac failure related to carbon monoxide toxicity caused death. The management of patients poisoned by carbon monoxide requires early identification and intensive treatment and a careful evaluation of the home environment prior to discharge. ST elevation in such patients may be related to coronary vasospasm.