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  • BibTeX
Long non-coding RNA LOC389641 promotes progression of pancreatic ductal adenocarcinoma and increases cell invasion by regulating E-cadherin in a TNFRSF10A-related manner
Cancer letters, 2016-02, Vol.371 (2), p.354-365
2016

Details

Autor(en) / Beteiligte
Titel
Long non-coding RNA LOC389641 promotes progression of pancreatic ductal adenocarcinoma and increases cell invasion by regulating E-cadherin in a TNFRSF10A-related manner
Ist Teil von
  • Cancer letters, 2016-02, Vol.371 (2), p.354-365
Ort / Verlag
Ireland: Elsevier B.V
Erscheinungsjahr
2016
Link zum Volltext
Quelle
MEDLINE
Beschreibungen/Notizen
  • Highlights • LOC389641 is upregulated in PDAC and can be used as an independent prognosis factor for PDAC patients. • LOC389641 promotes the progression of PDAC both in vitro and in vivo. • LOC389641 increases tumor invasion by regulating E-cadherin expression in PDAC cells. • The genomic neighboring gene TNFRSF10A of LOC389641 might be a connection between LOC389641 and E-cadherin.
Sprache
Englisch
Identifikatoren
ISSN: 0304-3835
eISSN: 1872-7980
DOI: 10.1016/j.canlet.2015.12.010
Titel-ID: cdi_proquest_miscellaneous_1773824171
Format
–
Schlagworte
Animals, Apoptosis, Cadherins - genetics, Cadherins - metabolism, Carcinoma, Pancreatic Ductal - genetics, Carcinoma, Pancreatic Ductal - metabolism, Carcinoma, Pancreatic Ductal - mortality, Carcinoma, Pancreatic Ductal - pathology, Cell cycle, Cell Line, Tumor, Cell Movement, Cell Proliferation, Confidence intervals, E-cadherin, Female, Gene Expression Regulation, Neoplastic, Gene Knockdown Techniques, Hematology, Oncology and Palliative Medicine, Humans, Invasion, Kaplan-Meier Estimate, LncRNAs, Lymphatic Metastasis, Male, Metastasis, Methods, Mice, Inbred BALB C, Mice, Nude, Middle Aged, Mortality, Multivariate analysis, Neoplasm Invasiveness, Neoplasm Staging, Pancreatic cancer, Pancreatic ductal adenocarcinoma, Pancreatic Neoplasms - genetics, Pancreatic Neoplasms - metabolism, Pancreatic Neoplasms - mortality, Pancreatic Neoplasms - pathology, Proportional Hazards Models, Receptors, TNF-Related Apoptosis-Inducing Ligand - genetics, Receptors, TNF-Related Apoptosis-Inducing Ligand - metabolism, RNA Interference, RNA, Long Noncoding - genetics, RNA, Long Noncoding - metabolism, Signal Transduction, Surgery, Time Factors, TNFRSF10A, Transfection

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